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Journal Abstract Search

145 related items for PubMed ID: 22227005

  • 1. Age-dependent disruption in hippocampal θ oscillation in amyloid-β overproducing transgenic mice.
    Scott L, Feng J, Kiss T, Needle E, Atchison K, Kawabe TT, Milici AJ, Hajós-Korcsok E, Riddell D, Hajós M.
    Neurobiol Aging; 2012 Jul; 33(7):1481.e13-23. PubMed ID: 22227005
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  • 2. Soluble Aβ levels correlate with cognitive deficits in the 12-month-old APPswe/PS1dE9 mouse model of Alzheimer's disease.
    Zhang W, Hao J, Liu R, Zhang Z, Lei G, Su C, Miao J, Li Z.
    Behav Brain Res; 2011 Sep 23; 222(2):342-50. PubMed ID: 21513747
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  • 3. Chronic treatment with the α2-adrenoceptor antagonist fluparoxan prevents age-related deficits in spatial working memory in APP×PS1 transgenic mice without altering β-amyloid plaque load or astrocytosis.
    Scullion GA, Kendall DA, Marsden CA, Sunter D, Pardon MC.
    Neuropharmacology; 2011 Sep 23; 60(2-3):223-34. PubMed ID: 20850464
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  • 4. Progressive age-related development of Alzheimer-like pathology in APP/PS1 mice.
    Trinchese F, Liu S, Battaglia F, Walter S, Mathews PM, Arancio O.
    Ann Neurol; 2004 Jun 23; 55(6):801-14. PubMed ID: 15174014
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  • 5. Cholinergic degeneration is associated with increased plaque deposition and cognitive impairment in APPswe/PS1dE9 mice.
    Laursen B, Mørk A, Plath N, Kristiansen U, Bastlund JF.
    Behav Brain Res; 2013 Mar 01; 240():146-52. PubMed ID: 23178660
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  • 7. Episodic memory deficits are not related to altered glutamatergic synaptic transmission and plasticity in the CA1 hippocampus of the APPswe/PS1δE9-deleted transgenic mice model of ß-amyloidosis.
    Volianskis A, Køstner R, Mølgaard M, Hass S, Jensen MS.
    Neurobiol Aging; 2010 Jul 01; 31(7):1173-87. PubMed ID: 18790549
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  • 11. Amyloid beta deposition is related to decreased glucose transporter-1 levels and hippocampal atrophy in brains of aged APP/PS1 mice.
    Hooijmans CR, Graven C, Dederen PJ, Tanila H, van Groen T, Kiliaan AJ.
    Brain Res; 2007 Nov 21; 1181():93-103. PubMed ID: 17916337
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  • 13. Synaptic transmission is impaired prior to plaque formation in amyloid precursor protein-overexpressing mice without altering behaviorally-correlated sharp wave-ripple complexes.
    Hermann D, Both M, Ebert U, Gross G, Schoemaker H, Draguhn A, Wicke K, Nimmrich V.
    Neuroscience; 2009 Sep 15; 162(4):1081-90. PubMed ID: 19477243
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  • 17. Altered levels and distribution of IGF-II/M6P receptor and lysosomal enzymes in mutant APP and APP + PS1 transgenic mouse brains.
    Amritraj A, Hawkes C, Phinney AL, Mount HT, Scott CD, Westaway D, Kar S.
    Neurobiol Aging; 2009 Jan 15; 30(1):54-70. PubMed ID: 17561313
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  • 18. IFN-γ Production by amyloid β-specific Th1 cells promotes microglial activation and increases plaque burden in a mouse model of Alzheimer's disease.
    Browne TC, McQuillan K, McManus RM, O'Reilly JA, Mills KH, Lynch MA.
    J Immunol; 2013 Mar 01; 190(5):2241-51. PubMed ID: 23365075
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  • 19. Deposition of mouse amyloid beta in human APP/PS1 double and single AD model transgenic mice.
    van Groen T, Kiliaan AJ, Kadish I.
    Neurobiol Dis; 2006 Sep 01; 23(3):653-62. PubMed ID: 16829076
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