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210 related items for PubMed ID: 22351778

  • 1. Familial focal segmental glomerulosclerosis (FSGS)-linked α-actinin 4 (ACTN4) protein mutants lose ability to activate transcription by nuclear hormone receptors.
    Khurana S, Chakraborty S, Lam M, Liu Y, Su YT, Zhao X, Saleem MA, Mathieson PW, Bruggeman LA, Kao HY.
    J Biol Chem; 2012 Apr 06; 287(15):12027-35. PubMed ID: 22351778
    [Abstract] [Full Text] [Related]

  • 2. α-Actinin 4 potentiates nuclear factor κ-light-chain-enhancer of activated B-cell (NF-κB) activity in podocytes independent of its cytoplasmic actin binding function.
    Zhao X, Hsu KS, Lim JH, Bruggeman LA, Kao HY.
    J Biol Chem; 2015 Jan 02; 290(1):338-49. PubMed ID: 25411248
    [Abstract] [Full Text] [Related]

  • 3. α Actinin 4 (ACTN4) Regulates Glucocorticoid Receptor-mediated Transactivation and Transrepression in Podocytes.
    Zhao X, Khurana S, Charkraborty S, Tian Y, Sedor JR, Bruggman LA, Kao HY.
    J Biol Chem; 2017 Feb 03; 292(5):1637-1647. PubMed ID: 27998979
    [Abstract] [Full Text] [Related]

  • 4. Focal and segmental glomerulosclerosis in mice with podocyte-specific expression of mutant alpha-actinin-4.
    Michaud JL, Lemieux LI, Dubé M, Vanderhyden BC, Robertson SJ, Kennedy CR.
    J Am Soc Nephrol; 2003 May 03; 14(5):1200-11. PubMed ID: 12707390
    [Abstract] [Full Text] [Related]

  • 5. Focal segmental glomerulosclerosis ACTN4 mutants binding to actin: regulation by phosphomimetic mutations.
    Shao H, Wingert B, Weins A, Pollak MR, Camacho C, Wells A.
    Sci Rep; 2019 Oct 29; 9(1):15517. PubMed ID: 31664084
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  • 7. FSGS-associated alpha-actinin-4 (K256E) impairs cytoskeletal dynamics in podocytes.
    Michaud JL, Chaisson KM, Parks RJ, Kennedy CR.
    Kidney Int; 2006 Sep 29; 70(6):1054-61. PubMed ID: 16837921
    [Abstract] [Full Text] [Related]

  • 8. Ubiquitin C-terminal hydrolase L1 deletion ameliorates glomerular injury in mice with ACTN4-associated focal segmental glomerulosclerosis.
    Read NC, Gutsol A, Holterman CE, Carter A, Coulombe J, Gray DA, Kennedy CR.
    Biochim Biophys Acta; 2014 Jul 29; 1842(7):1028-40. PubMed ID: 24662305
    [Abstract] [Full Text] [Related]

  • 9. Phosphorylation of ACTN4 Leads to Podocyte Vulnerability and Proteinuric Glomerulosclerosis.
    Feng D, Kumar M, Muntel J, Gurley SB, Birrane G, Stillman IE, Ding L, Wang M, Ahmed S, Schlondorff J, Alper SL, Ferrante T, Marquez SL, Ng CF, Novak R, Ingber DE, Steen H, Pollak MR.
    J Am Soc Nephrol; 2020 Jul 29; 31(7):1479-1495. PubMed ID: 32540856
    [Abstract] [Full Text] [Related]

  • 10. Screening of ACTN4 and TRPC6 mutations in a Chinese cohort of patients with adult-onset familial focal segmental glomerulosclerosis.
    Zhang Q, Ma J, Xie J, Wang Z, Zhu B, Hao X, Yang L, Ren H, Chen N.
    Contrib Nephrol; 2013 Jul 29; 181():91-100. PubMed ID: 23689571
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  • 12. Patients with ACTN4 mutations demonstrate distinctive features of glomerular injury.
    Henderson JM, Alexander MP, Pollak MR.
    J Am Soc Nephrol; 2009 May 29; 20(5):961-8. PubMed ID: 19357256
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  • 14. Functional analysis of promoter mutations in the ACTN4 and SYNPO genes in focal segmental glomerulosclerosis.
    Dai S, Wang Z, Pan X, Wang W, Chen X, Ren H, Hao C, Han B, Chen N.
    Nephrol Dial Transplant; 2010 Mar 29; 25(3):824-35. PubMed ID: 19666657
    [Abstract] [Full Text] [Related]

  • 15. Disease-causing mutation in α-actinin-4 promotes podocyte detachment through maladaptation to periodic stretch.
    Feng D, Notbohm J, Benjamin A, He S, Wang M, Ang LH, Bantawa M, Bouzid M, Del Gado E, Krishnan R, Pollak MR.
    Proc Natl Acad Sci U S A; 2018 Feb 13; 115(7):1517-1522. PubMed ID: 29378953
    [Abstract] [Full Text] [Related]

  • 16. Three-layered proteomic characterization of a novel ACTN4 mutation unravels its pathogenic potential in FSGS.
    Bartram MP, Habbig S, Pahmeyer C, Höhne M, Weber LT, Thiele H, Altmüller J, Kottoor N, Wenzel A, Krueger M, Schermer B, Benzing T, Rinschen MM, Beck BB.
    Hum Mol Genet; 2016 Mar 15; 25(6):1152-64. PubMed ID: 26740551
    [Abstract] [Full Text] [Related]

  • 17. Alpha-actinin-4-mediated FSGS: an inherited kidney disease caused by an aggregated and rapidly degraded cytoskeletal protein.
    Yao J, Le TC, Kos CH, Henderson JM, Allen PG, Denker BM, Pollak MR.
    PLoS Biol; 2004 Jun 15; 2(6):e167. PubMed ID: 15208719
    [Abstract] [Full Text] [Related]

  • 18. Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis.
    Kaplan JM, Kim SH, North KN, Rennke H, Correia LA, Tong HQ, Mathis BJ, Rodríguez-Pérez JC, Allen PG, Beggs AH, Pollak MR.
    Nat Genet; 2000 Mar 15; 24(3):251-6. PubMed ID: 10700177
    [Abstract] [Full Text] [Related]

  • 19. The actin-binding protein, actinin alpha 4 (ACTN4), is a nuclear receptor coactivator that promotes proliferation of MCF-7 breast cancer cells.
    Khurana S, Chakraborty S, Cheng X, Su YT, Kao HY.
    J Biol Chem; 2011 Jan 21; 286(3):1850-9. PubMed ID: 21078666
    [Abstract] [Full Text] [Related]

  • 20. Functional Validation of an Alpha-Actinin-4 Mutation as a Potential Cause of an Aggressive Presentation of Adolescent Focal Segmental Glomerulosclerosis: Implications for Genetic Testing.
    Feng D, Steinke JM, Krishnan R, Birrane G, Pollak MR.
    PLoS One; 2016 Jan 21; 11(12):e0167467. PubMed ID: 27977723
    [Abstract] [Full Text] [Related]

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