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Journal Abstract Search

127 related items for PubMed ID: 22521321

  • 21.
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  • 22. Increased asymmetric dimethylarginine (ADMA) dimethylaminohydrolase (DDAH) activity in childhood hypercholesterolemia type II.
    Chobanyan-Jürgens K, Fuchs AJ, Tsikas D, Kanzelmeyer N, Das AM, Illsinger S, Vaske B, Jordan J, Lücke T.
    Amino Acids; 2012 Aug; 43(2):805-11. PubMed ID: 22075968
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  • 26. Dimethylarginine Dimethylaminohydrolase 1 Polymorphisms and Venous Intimal Hyperplasia in Hemodialysis Patients.
    Wu CC, Hsieh MY, Lee CK, Chuang SY, Chung MY, Lin CC.
    Am J Nephrol; 2019 Aug; 50(6):454-464. PubMed ID: 31639806
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  • 27. Inhibitory effect of resveratrol derivative BTM-0512 on high glucose-induced cell senescence involves dimethylaminohydrolase/asymmetric dimethylarginine pathway.
    Yuan Q, Peng J, Liu SY, Wang CJ, Xiang DX, Xiong XM, Hu CP, Li YJ.
    Clin Exp Pharmacol Physiol; 2010 May; 37(5-6):630-5. PubMed ID: 20132235
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  • 28. Dimethylarginine dimethylaminohydrolase-2 overexpression improves impaired nitric oxide synthesis of endothelial cells induced by glycated protein.
    Lu CW, Xiong Y, He P.
    Nitric Oxide; 2007 Feb; 16(1):94-103. PubMed ID: 16979918
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  • 29. Asymmetric dimethylarginine contributes to retinal neovascularization of diabetic retinopathy through EphrinB2 pathway.
    Du MR, Yan L, Li NS, Wang YJ, Zhou T, Jiang JL.
    Vascul Pharmacol; 2018 Sep; 108():46-56. PubMed ID: 29777874
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  • 30. Serum asymmetric dimethylarginine and arginine levels predict microvascular and macrovascular complications in type 2 diabetes mellitus.
    Ganz T, Wainstein J, Gilad S, Limor R, Boaz M, Stern N.
    Diabetes Metab Res Rev; 2017 Feb; 33(2):. PubMed ID: 27393712
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  • 31. Dimethylarginine dimethylaminohydrolase prevents progression of renal dysfunction by inhibiting loss of peritubular capillaries and tubulointerstitial fibrosis in a rat model of chronic kidney disease.
    Matsumoto Y, Ueda S, Yamagishi S, Matsuguma K, Shibata R, Fukami K, Matsuoka H, Imaizumi T, Okuda S.
    J Am Soc Nephrol; 2007 May; 18(5):1525-33. PubMed ID: 17409314
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  • 32. Pilot study of the association of the DDAH2 -449G polymorphism with asymmetric dimethylarginine and hemodynamic shock in pediatric sepsis.
    Weiss SL, Yu M, Jennings L, Haymond S, Zhang G, Wainwright MS.
    PLoS One; 2012 May; 7(3):e33355. PubMed ID: 22428028
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  • 34. ADMA elevation does not exacerbate development of diabetic nephropathy in mice with streptozotocin-induced diabetes mellitus.
    Rodionov RN, Jarzebska N, Schneider A, Rexin A, Sradnick J, Brilloff S, Martens-Lobenhoffer J, Bode-Böger SM, Todorov V, Hugo C, Weiss N, Hohenstein B.
    Atheroscler Suppl; 2019 Dec; 40():100-105. PubMed ID: 31818438
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  • 35. Molecular mechanism for elevation of asymmetric dimethylarginine and its role for hypertension in chronic kidney disease.
    Matsuguma K, Ueda S, Yamagishi S, Matsumoto Y, Kaneyuki U, Shibata R, Fujimura T, Matsuoka H, Kimoto M, Kato S, Imaizumi T, Okuda S.
    J Am Soc Nephrol; 2006 Aug; 17(8):2176-83. PubMed ID: 16807406
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  • 36. GC-MS assay for hepatic DDAH activity in diabetic and non-diabetic rats by measuring dimethylamine (DMA) formed from asymmetric dimethylarginine (ADMA): evaluation of the importance of S-nitrosothiols as inhibitors of DDAH activity in vitro and in vivo in humans.
    Chobanyan K, Thum T, Suchy MT, Zhu B, Mitschke A, Gutzki FM, Beckmann B, Stichtenoth DO, Tsikas D.
    J Chromatogr B Analyt Technol Biomed Life Sci; 2007 Oct 15; 858(1-2):32-41. PubMed ID: 17825631
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  • 37. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia.
    Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD.
    Acta Paediatr; 2016 Apr 15; 105(4):e170-5. PubMed ID: 26663142
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  • 38. Redefining the biological and pathophysiological role of dimethylarginine dimethylaminohydrolase 2.
    Nair PC, Mangoni AA, Rodionov RN.
    Trends Mol Med; 2024 Jun 15; 30(6):552-561. PubMed ID: 38553332
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  • 39. A possible involvement of crosstalk between advanced glycation end products (AGEs) and asymmetric dimethylarginine (ADMA), an endogenous nitric oxide synthase inhibitor in accelerated atherosclerosis in diabetes.
    Yamagishi S, Ueda S, Okuda S.
    Med Hypotheses; 2007 Jun 15; 69(4):922-4. PubMed ID: 17368960
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