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Journal Abstract Search

227 related items for PubMed ID: 22521362

  • 1. The relationship between Bayesian motor unit number estimation and histological measurements of motor neurons in wild-type and SOD1(G93A) mice.
    Ngo ST, Baumann F, Ridall PG, Pettitt AN, Henderson RD, Bellingham MC, McCombe PA.
    Clin Neurophysiol; 2012 Oct; 123(10):2080-91. PubMed ID: 22521362
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  • 2. Functional over-load saves motor units in the SOD1-G93A transgenic mouse model of amyotrophic lateral sclerosis.
    Gordon T, Tyreman N, Li S, Putman CT, Hegedus J.
    Neurobiol Dis; 2010 Feb; 37(2):412-22. PubMed ID: 19879358
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  • 7. Time course of preferential motor unit loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis.
    Hegedus J, Putman CT, Gordon T.
    Neurobiol Dis; 2007 Nov; 28(2):154-64. PubMed ID: 17766128
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  • 8. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.
    Neurobiol Dis; 2014 Apr; 64():48-59. PubMed ID: 24361555
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  • 10. GAB(A) receptors present higher affinity and modified subunit composition in spinal motor neurons from a genetic model of amyotrophic lateral sclerosis.
    Carunchio I, Mollinari C, Pieri M, Merlo D, Zona C.
    Eur J Neurosci; 2008 Oct; 28(7):1275-85. PubMed ID: 18973555
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  • 12. Neurovascular unit pathology is observed very early in disease progression in the mutant SOD1G93A mouse model of amyotrophic lateral sclerosis.
    Yoshikawa M, Aizawa S, Oppenheim RW, Milligan C.
    Exp Neurol; 2022 Jul; 353():114084. PubMed ID: 35439439
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  • 14. A method comparison in monitoring disease progression of G93A mouse model of ALS.
    Zhou C, Zhao CP, Zhang C, Wu GY, Xiong F, Zhang C.
    Amyotroph Lateral Scler; 2007 Dec; 8(6):366-72. PubMed ID: 17852023
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  • 15. Spinal inhibitory interneuron pathology follows motor neuron degeneration independent of glial mutant superoxide dismutase 1 expression in SOD1-ALS mice.
    Hossaini M, Cardona Cano S, van Dis V, Haasdijk ED, Hoogenraad CC, Holstege JC, Jaarsma D.
    J Neuropathol Exp Neurol; 2011 Aug; 70(8):662-77. PubMed ID: 21760539
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  • 17. Relationship of oxygen radical-induced lipid peroxidative damage to disease onset and progression in a transgenic model of familial ALS.
    Hall ED, Andrus PK, Oostveen JA, Fleck TJ, Gurney ME.
    J Neurosci Res; 1998 Jul 01; 53(1):66-77. PubMed ID: 9670993
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  • 18. Electrophysiological analysis of a murine model of motoneuron disease.
    Mancuso R, Santos-Nogueira E, Osta R, Navarro X.
    Clin Neurophysiol; 2011 Aug 01; 122(8):1660-70. PubMed ID: 21354365
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  • 19. Treatment with an antibody directed against Nogo-A delays disease progression in the SOD1G93A mouse model of Amyotrophic lateral sclerosis.
    Bros-Facer V, Krull D, Taylor A, Dick JR, Bates SA, Cleveland MS, Prinjha RK, Greensmith L.
    Hum Mol Genet; 2014 Aug 15; 23(16):4187-200. PubMed ID: 24667415
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