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163 related items for PubMed ID: 22610183
1. Relationship between systemic glucose and cerebral glucose is preserved in patients with severe traumatic brain injury, but glucose delivery to the brain may become limited when oxidative metabolism is impaired: implications for glycemic control. Magnoni S, Tedesco C, Carbonara M, Pluderi M, Colombo A, Stocchetti N. Crit Care Med; 2012 Jun; 40(6):1785-91. PubMed ID: 22610183 [Abstract] [Full Text] [Related]
2. Impact of tight glycemic control on cerebral glucose metabolism after severe brain injury: a microdialysis study. Oddo M, Schmidt JM, Carrera E, Badjatia N, Connolly ES, Presciutti M, Ostapkovich ND, Levine JM, Le Roux P, Mayer SA. Crit Care Med; 2008 Dec; 36(12):3233-8. PubMed ID: 18936695 [Abstract] [Full Text] [Related]
3. Pericontusional brain tissue exhibits persistent elevation of lactate/pyruvate ratio independent of cerebral perfusion pressure. Vespa PM, O'Phelan K, McArthur D, Miller C, Eliseo M, Hirt D, Glenn T, Hovda DA. Crit Care Med; 2007 Apr; 35(4):1153-60. PubMed ID: 17334254 [Abstract] [Full Text] [Related]
4. Intensive insulin therapy reduces microdialysis glucose values without altering glucose utilization or improving the lactate/pyruvate ratio after traumatic brain injury. Vespa P, Boonyaputthikul R, McArthur DL, Miller C, Etchepare M, Bergsneider M, Glenn T, Martin N, Hovda D. Crit Care Med; 2006 Mar; 34(3):850-6. PubMed ID: 16505665 [Abstract] [Full Text] [Related]