These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

536 related items for PubMed ID: 22795219

  • 1. The progression of calcific aortic valve disease through injury, cell dysfunction, and disruptive biologic and physical force feedback loops.
    Li C, Xu S, Gotlieb AI.
    Cardiovasc Pathol; 2013; 22(1):1-8. PubMed ID: 22795219
    [Abstract] [Full Text] [Related]

  • 2. Calcific Aortic Valve Disease: Part 1--Molecular Pathogenetic Aspects, Hemodynamics, and Adaptive Feedbacks.
    Pasipoularides A.
    J Cardiovasc Transl Res; 2016 Apr; 9(2):102-18. PubMed ID: 26891845
    [Abstract] [Full Text] [Related]

  • 3. Basic mechanisms of calcific aortic valve disease.
    Mathieu P, Boulanger MC.
    Can J Cardiol; 2014 Sep; 30(9):982-93. PubMed ID: 25085215
    [Abstract] [Full Text] [Related]

  • 4. Inflammatory and metabolic mechanisms underlying the calcific aortic valve disease.
    Cho KI, Sakuma I, Sohn IS, Jo SH, Koh KK.
    Atherosclerosis; 2018 Oct; 277():60-65. PubMed ID: 30173080
    [Abstract] [Full Text] [Related]

  • 5.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 6.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 7. Cell-matrix interactions in the pathobiology of calcific aortic valve disease: critical roles for matricellular, matricrine, and matrix mechanics cues.
    Chen JH, Simmons CA.
    Circ Res; 2011 Jun 10; 108(12):1510-24. PubMed ID: 21659654
    [Abstract] [Full Text] [Related]

  • 8. Osteogenesis in calcified aortic valve disease: From histopathological observation towards molecular understanding.
    Liu X, Xu Z.
    Prog Biophys Mol Biol; 2016 Nov 10; 122(2):156-161. PubMed ID: 26971958
    [Abstract] [Full Text] [Related]

  • 9.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 10.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 11. Simulation of early calcific aortic valve disease in a 3D platform: A role for myofibroblast differentiation.
    Hjortnaes J, Goettsch C, Hutcheson JD, Camci-Unal G, Lax L, Scherer K, Body S, Schoen FJ, Kluin J, Khademhosseini A, Aikawa E.
    J Mol Cell Cardiol; 2016 May 10; 94():13-20. PubMed ID: 26996755
    [Abstract] [Full Text] [Related]

  • 12. Cell-matrix mechanics and pattern formation in inflammatory cardiovascular calcification.
    Hsu JJ, Lim J, Tintut Y, Demer LL.
    Heart; 2016 Nov 01; 102(21):1710-1715. PubMed ID: 27406839
    [Abstract] [Full Text] [Related]

  • 13. Cadherin-11 Overexpression Induces Extracellular Matrix Remodeling and Calcification in Mature Aortic Valves.
    Sung DC, Bowen CJ, Vaidya KA, Zhou J, Chapurin N, Recknagel A, Zhou B, Chen J, Kotlikoff M, Butcher JT.
    Arterioscler Thromb Vasc Biol; 2016 Aug 01; 36(8):1627-37. PubMed ID: 27312222
    [Abstract] [Full Text] [Related]

  • 14. The response to valve injury. A paradigm to understand the pathogenesis of heart valve disease.
    Li C, Xu S, Gotlieb AI.
    Cardiovasc Pathol; 2011 Aug 01; 20(3):183-90. PubMed ID: 21075649
    [Abstract] [Full Text] [Related]

  • 15. Valve Interstitial Cells: The Key to Understanding the Pathophysiology of Heart Valve Calcification.
    Rutkovskiy A, Malashicheva A, Sullivan G, Bogdanova M, Kostareva A, Stensløkken KO, Fiane A, Vaage J.
    J Am Heart Assoc; 2017 Sep 14; 6(9):. PubMed ID: 28912209
    [No Abstract] [Full Text] [Related]

  • 16. Biophysical analysis of dystrophic and osteogenic models of valvular calcification.
    Chen J, Peacock JR, Branch J, David Merryman W.
    J Biomech Eng; 2015 Feb 01; 137(2):020903. PubMed ID: 25405546
    [Abstract] [Full Text] [Related]

  • 17. Aortic stenosis: a general overview of clinical, pathophysiological and therapeutic aspects.
    Akerström F, Barderas MG, Rodríguez-Padial L.
    Expert Rev Cardiovasc Ther; 2013 Feb 01; 11(2):239-50. PubMed ID: 23405844
    [Abstract] [Full Text] [Related]

  • 18. Heterogeneous multi-laminar tissue constructs as a platform to evaluate aortic valve matrix-dependent pathogenicity.
    Monroe MN, Nikonowicz RC, Grande-Allen KJ.
    Acta Biomater; 2019 Oct 01; 97():420-427. PubMed ID: 31362141
    [Abstract] [Full Text] [Related]

  • 19. Inflammation is associated with the remodeling of calcific aortic valve disease.
    Coté N, Mahmut A, Bosse Y, Couture C, Pagé S, Trahan S, Boulanger MC, Fournier D, Pibarot P, Mathieu P.
    Inflammation; 2013 Jun 01; 36(3):573-81. PubMed ID: 23225202
    [Abstract] [Full Text] [Related]

  • 20. A three-dimensional valve-on-chip microphysiological system implicates cell cycle progression, cholesterol metabolism and protein homeostasis in early calcific aortic valve disease progression.
    Tandon I, Woessner AE, Ferreira LA, Shamblin C, Vaca-Diez G, Walls A, Kuczwara P, Applequist A, Nascimento DF, Tandon S, Kim JW, Rausch M, Timek T, Padala M, Kinter MT, Province D, Byrum SD, Quinn KP, Balachandran K.
    Acta Biomater; 2024 Sep 15; 186():167-184. PubMed ID: 39084496
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 27.