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Journal Abstract Search

314 related items for PubMed ID: 22802227

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  • 2. Renal tubular NEDD4-2 deficiency causes NCC-mediated salt-dependent hypertension.
    Ronzaud C, Loffing-Cueni D, Hausel P, Debonneville A, Malsure SR, Fowler-Jaeger N, Boase NA, Perrier R, Maillard M, Yang B, Stokes JB, Koesters R, Kumar S, Hummler E, Loffing J, Staub O.
    J Clin Invest; 2013 Feb; 123(2):657-65. PubMed ID: 23348737
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  • 6. Mineralocorticoid receptors/epithelial Na(+) channels in the choroid plexus are involved in hypertensive mechanisms in stroke-prone spontaneously hypertensive rats.
    Nakano M, Hirooka Y, Matsukawa R, Ito K, Sunagawa K.
    Hypertens Res; 2013 Mar; 36(3):277-84. PubMed ID: 23096235
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  • 8. Effects of central sodium on epithelial sodium channels in rat brain.
    Wang HW, Amin MS, El-Shahat E, Huang BS, Tuana BS, Leenen FH.
    Am J Physiol Regul Integr Comp Physiol; 2010 Jul; 299(1):R222-33. PubMed ID: 20427723
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  • 10. An isoform of Nedd4-2 is critically involved in the renal adaptation to high salt intake in mice.
    Minegishi S, Ishigami T, Kino T, Chen L, Nakashima-Sasaki R, Araki N, Yatsu K, Fujita M, Umemura S.
    Sci Rep; 2016 Jun 03; 6():27137. PubMed ID: 27256588
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  • 11. Enhanced expression of epithelial sodium channels causes salt-induced hypertension in mice through inhibition of the α2-isoform of Na+, K+-ATPase.
    Leenen FH, Hou X, Wang HW, Ahmad M.
    Physiol Rep; 2015 May 03; 3(5):. PubMed ID: 25991719
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  • 13. Deletion of renal Nedd4-2 abolishes the effect of high sodium intake (HS) on Kir4.1, ENaC, and NCC and causes hypokalemia during high HS.
    Zhang DD, Duan XP, Xiao Y, Wu P, Gao ZX, Wang WH, Lin DH.
    Am J Physiol Renal Physiol; 2021 May 01; 320(5):F883-F896. PubMed ID: 33818128
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  • 17. Deficiency of renal cortical EGF increases ENaC activity and contributes to salt-sensitive hypertension.
    Pavlov TS, Levchenko V, O'Connor PM, Ilatovskaya DV, Palygin O, Mori T, Mattson DL, Sorokin A, Lombard JH, Cowley AW, Staruschenko A.
    J Am Soc Nephrol; 2013 Jun 01; 24(7):1053-62. PubMed ID: 23599382
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  • 19. Aldosterone responsiveness of the epithelial sodium channel (ENaC) in colon is increased in a mouse model for Liddle's syndrome.
    Bertog M, Cuffe JE, Pradervand S, Hummler E, Hartner A, Porst M, Hilgers KF, Rossier BC, Korbmacher C.
    J Physiol; 2008 Jan 15; 586(2):459-75. PubMed ID: 18006588
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  • 20. ROMK channels are inhibited in the aldosterone-sensitive distal nephron of renal tubule Nedd4-2-deficient mice.
    Zhang DD, Zheng JY, Duan XP, Lin DH, Wang WH.
    Am J Physiol Renal Physiol; 2022 Jan 01; 322(1):F55-F67. PubMed ID: 34843409
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