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Journal Abstract Search

271 related items for PubMed ID: 22874425

  • 1. Excess protein O-GlcNAcylation and the progression of diabetic cardiomyopathy.
    Fricovsky ES, Suarez J, Ihm SH, Scott BT, Suarez-Ramirez JA, Banerjee I, Torres-Gonzalez M, Wang H, Ellrott I, Maya-Ramos L, Villarreal F, Dillmann WH.
    Am J Physiol Regul Integr Comp Physiol; 2012 Oct 01; 303(7):R689-99. PubMed ID: 22874425
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  • 2. Diabetes and the accompanying hyperglycemia impairs cardiomyocyte calcium cycling through increased nuclear O-GlcNAcylation.
    Clark RJ, McDonough PM, Swanson E, Trost SU, Suzuki M, Fukuda M, Dillmann WH.
    J Biol Chem; 2003 Nov 07; 278(45):44230-7. PubMed ID: 12941958
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  • 4. Fine-tuning the cardiac O-GlcNAcylation regulatory enzymes governs the functional and structural phenotype of the diabetic heart.
    Prakoso D, Lim SY, Erickson JR, Wallace RS, Lees JG, Tate M, Kiriazis H, Donner DG, Henstridge DC, Davey JR, Qian H, Deo M, Parry LJ, Davidoff AJ, Gregorevic P, Chatham JC, De Blasio MJ, Ritchie RH.
    Cardiovasc Res; 2022 Jan 07; 118(1):212-225. PubMed ID: 33576380
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  • 5. Inhibition of phospholamban phosphorylation by O-GlcNAcylation: implications for diabetic cardiomyopathy.
    Yokoe S, Asahi M, Takeda T, Otsu K, Taniguchi N, Miyoshi E, Suzuki K.
    Glycobiology; 2010 Oct 07; 20(10):1217-26. PubMed ID: 20484118
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  • 11. O-GlcNAcylation involvement in high glucose-induced cardiac hypertrophy via ERK1/2 and cyclin D2.
    Ding F, Yu L, Wang M, Xu S, Xia Q, Fu G.
    Amino Acids; 2013 Aug 07; 45(2):339-49. PubMed ID: 23665912
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  • 12. Sustained Increases in Cardiomyocyte Protein O-Linked β-N-Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment.
    Ha CM, Bakshi S, Brahma MK, Potter LA, Chang SF, Sun Z, Benavides GA, He L, Umbarkar P, Zou L, Curfman S, Sunny S, Paterson AJ, Rajasekaran NS, Barnes JW, Zhang J, Lal H, Xie M, Darley-Usmar VM, Chatham JC, Wende AR.
    J Am Heart Assoc; 2023 Oct 03; 12(19):e029898. PubMed ID: 37750556
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  • 15. Hyperglycemia Acutely Increases Cytosolic Reactive Oxygen Species via O-linked GlcNAcylation and CaMKII Activation in Mouse Ventricular Myocytes.
    Lu S, Liao Z, Lu X, Katschinski DM, Mercola M, Chen J, Heller Brown J, Molkentin JD, Bossuyt J, Bers DM.
    Circ Res; 2020 May 08; 126(10):e80-e96. PubMed ID: 32134364
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  • 16. Type 1 diabetic cardiomyopathy in the Akita (Ins2WT/C96Y) mouse model is characterized by lipotoxicity and diastolic dysfunction with preserved systolic function.
    Basu R, Oudit GY, Wang X, Zhang L, Ussher JR, Lopaschuk GD, Kassiri Z.
    Am J Physiol Heart Circ Physiol; 2009 Dec 08; 297(6):H2096-108. PubMed ID: 19801494
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  • 17. Activation of AKT by O-linked N-acetylglucosamine induces vascular calcification in diabetes mellitus.
    Heath JM, Sun Y, Yuan K, Bradley WE, Litovsky S, Dell'Italia LJ, Chatham JC, Wu H, Chen Y.
    Circ Res; 2014 Mar 28; 114(7):1094-102. PubMed ID: 24526702
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  • 18. Adenovirus-mediated overexpression of O-GlcNAcase improves contractile function in the diabetic heart.
    Hu Y, Belke D, Suarez J, Swanson E, Clark R, Hoshijima M, Dillmann WH.
    Circ Res; 2005 May 13; 96(9):1006-13. PubMed ID: 15817886
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  • 19. Role for high-glucose-induced protein O-GlcNAcylation in stimulating cardiac fibroblast collagen synthesis.
    Aguilar H, Fricovsky E, Ihm S, Schimke M, Maya-Ramos L, Aroonsakool N, Ceballos G, Dillmann W, Villarreal F, Ramirez-Sanchez I.
    Am J Physiol Cell Physiol; 2014 May 01; 306(9):C794-804. PubMed ID: 24553187
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  • 20. O-GlcNAcylation of 8-Oxoguanine DNA Glycosylase (Ogg1) Impairs Oxidative Mitochondrial DNA Lesion Repair in Diabetic Hearts.
    Cividini F, Scott BT, Dai A, Han W, Suarez J, Diaz-Juarez J, Diemer T, Casteel DE, Dillmann WH.
    J Biol Chem; 2016 Dec 16; 291(51):26515-26528. PubMed ID: 27816939
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