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155 related items for PubMed ID: 22902525
1. Nitrite- and nitroxyl-induced relaxation in porcine coronary (micro-) arteries: underlying mechanisms and role as endothelium-derived hyperpolarizing factor(s). Botden IP, Batenburg WW, de Vries R, Langendonk JG, Sijbrands EJ, Danser AH. Pharmacol Res; 2012 Nov; 66(5):409-18. PubMed ID: 22902525 [Abstract] [Full Text] [Related]
8. Soluble guanylate cyclase is required for systemic vasodilation but not positive inotropy induced by nitroxyl in the mouse. Zhu G, Groneberg D, Sikka G, Hori D, Ranek MJ, Nakamura T, Takimoto E, Paolocci N, Berkowitz DE, Friebe A, Kass DA. Hypertension; 2015 Feb; 65(2):385-92. PubMed ID: 25452469 [Abstract] [Full Text] [Related]
9. Redox variants of NO (NO{middle dot} and HNO) elicit vasorelaxation of resistance arteries via distinct mechanisms. Favaloro JL, Kemp-Harper BK. Am J Physiol Heart Circ Physiol; 2009 May; 296(5):H1274-80. PubMed ID: 19252101 [Abstract] [Full Text] [Related]
14. Mechanisms underlying activation of soluble guanylate cyclase by the nitroxyl donor Angeli's salt. Zeller A, Wenzl MV, Beretta M, Stessel H, Russwurm M, Koesling D, Schmidt K, Mayer B. Mol Pharmacol; 2009 Nov; 76(5):1115-22. PubMed ID: 19720727 [Abstract] [Full Text] [Related]
15. Role of sulfhydryl-dependent dimerization of soluble guanylyl cyclase in relaxation of porcine coronary artery to nitric oxide. Zheng X, Ying L, Liu J, Dou D, He Q, Leung SW, Man RY, Vanhoutte PM, Gao Y. Cardiovasc Res; 2011 Jun 01; 90(3):565-72. PubMed ID: 21248051 [Abstract] [Full Text] [Related]
16. Endothelium-derived hyperpolarizing factor and protein kinase G Iα activation: H2O2 versus S-nitrosothiols. Bautista-Niño PK, van der Stel M, Batenburg WW, de Vries R, Roks AJM, Danser AHJ. Eur J Pharmacol; 2018 May 15; 827():112-116. PubMed ID: 29550336 [Abstract] [Full Text] [Related]