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Journal Abstract Search

150 related items for PubMed ID: 22941498

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  • 2. Helicobacter pylori cagL amino acid polymorphism D58E59 pave the way toward peptic ulcer disease while N58E59 is associated with gastric cancer in north of Iran.
    Cherati MR, Shokri-Shirvani J, Karkhah A, Rajabnia R, Nouri HR.
    Microb Pathog; 2017 Jun; 107():413-418. PubMed ID: 28433795
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  • 3. Virulence attributes of Helicobacter pylori isolates & their association with gastroduodenal disease.
    Saxena A, Shukla S, Prasad KN, Ghoshal UC.
    Indian J Med Res; 2011 May; 133(5):514-20. PubMed ID: 21623037
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  • 5. Genetic diversity and amino acid sequence polymorphism in Helicobacter pylori CagL hypervariable motif and its association with virulence markers and gastroduodenal diseases.
    Yadegar A, Mohabati Mobarez A, Zali MR.
    Cancer Med; 2019 Apr; 8(4):1619-1632. PubMed ID: 30873747
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  • 6. H. pylori isolates with amino acid sequence polymorphisms as presence of both HtrA-L171 & CagL-Y58/E59 increase the risk of gastric cancer.
    Yeh YC, Kuo HY, Chang WL, Yang HB, Lu CC, Cheng HC, Wu MS, Sheu BS.
    J Biomed Sci; 2019 Jan 05; 26(1):4. PubMed ID: 30611258
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  • 9. Association between polymorphisms in HLA-A, HLA-B, HLA-DR, and DQ genes from gastric cancer and duodenal ulcer patients and cagL among cagA-positive Helicobacter pylori strains: The first study in a Turkish population.
    Kocak BT, Saribas S, Demiryas S, Yilmaz E, Uysal O, Kepil N, Demirci M, Dınc HO, Akkus S, Gülergün R, Gareayaghi N, Dağdeviren HE, Ozbey D, Dağ HH, Tokman HB, Tasci I, Kocazeybek B.
    Infect Genet Evol; 2020 Aug 05; 82():104288. PubMed ID: 32179147
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  • 10. Preservation of Helicobacter pylori CagA Translocation and Host Cell Proinflammatory Responses in the Face of CagL Hypervariability at Amino Acid Residues 58/59.
    Tafreshi M, Zwickel N, Gorrell RJ, Kwok T.
    PLoS One; 2015 Aug 05; 10(7):e0133531. PubMed ID: 26196862
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  • 12. The association between cagL and cagA, vacAs-m, babA genes in patients with gastric cancer, duodenal ulcer, and non-ulcer dyspepsia related to Helicobacter pylori.
    Demiryas S, Caliskan R, Saribas S, Akkus S, Gareayaghi N, Kirmusaoglu S, Kepil N, Dinc H, Dag H, Dagdeviren E, Tokman HB, Kalayci F, Demirci M, Tasci I, Erzin Y, Bal K, Kocazeybek B.
    Acta Gastroenterol Belg; 2020 Aug 05; 83(3):385-392. PubMed ID: 33094584
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  • 13. Prevalence of cagA EPIYA motifs in Helicobacter pylori among dyspeptic patients in northeast Thailand.
    Chomvarin C, Phusri K, Sawadpanich K, Mairiang P, Namwat W, Wongkham C, Hahnvajanawong C.
    Southeast Asian J Trop Med Public Health; 2012 Jan 05; 43(1):105-15. PubMed ID: 23082560
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  • 14. Epstein-Barr virus DNA load and its association with Helicobacter pylori infection in gastroduodenal diseases.
    Shukla SK, Prasad KN, Tripathi A, Singh A, Saxena A, Ghoshal UC, Krishnani N, Husain N.
    Braz J Infect Dis; 2011 Jan 05; 15(6):583-90. PubMed ID: 22218519
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  • 15. [Molecular detection of Helicobacter pylori vacA and cagA genes in gastric tissue specimens of patients with peptic ulcer disease and non-ulcer dyspepsia].
    Karaman M, Abacıoğlu H, Topalak OS, Simşek I.
    Mikrobiyol Bul; 2011 Jan 05; 45(1):11-20. PubMed ID: 21341154
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  • 18. An RGD helper sequence in CagL of Helicobacter pylori assists in interactions with integrins and injection of CagA.
    Conradi J, Tegtmeyer N, Woźna M, Wissbrock M, Michalek C, Gagell C, Cover TL, Frank R, Sewald N, Backert S.
    Front Cell Infect Microbiol; 2012 Jan 05; 2():70. PubMed ID: 22919661
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