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Journal Abstract Search

513 related items for PubMed ID: 23080121

  • 1. Mre11 ATLD17/18 mutation retains Tel1/ATM activity but blocks DNA double-strand break repair.
    Limbo O, Moiani D, Kertokalio A, Wyman C, Tainer JA, Russell P.
    Nucleic Acids Res; 2012 Dec; 40(22):11435-49. PubMed ID: 23080121
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  • 2. Mre11 dimers coordinate DNA end bridging and nuclease processing in double-strand-break repair.
    Williams RS, Moncalian G, Williams JS, Yamada Y, Limbo O, Shin DS, Groocock LM, Cahill D, Hitomi C, Guenther G, Moiani D, Carney JP, Russell P, Tainer JA.
    Cell; 2008 Oct 03; 135(1):97-109. PubMed ID: 18854158
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  • 4. The role of MRN in the S-phase DNA damage checkpoint is independent of its Ctp1-dependent roles in double-strand break repair and checkpoint signaling.
    Porter-Goff ME, Rhind N.
    Mol Biol Cell; 2009 Apr 03; 20(7):2096-107. PubMed ID: 19211838
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  • 8. Nonhomologous End-Joining with Minimal Sequence Loss Is Promoted by the Mre11-Rad50-Nbs1-Ctp1 Complex in Schizosaccharomyces pombe.
    Li Y, Wang J, Zhou G, Lajeunesse M, Le N, Stawicki BN, Corcino YL, Berkner KL, Runge KW.
    Genetics; 2017 May 03; 206(1):481-496. PubMed ID: 28292918
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  • 9. Ctp1-dependent clipping and resection of DNA double-strand breaks by Mre11 endonuclease complex are not genetically separable.
    Jensen KL, Russell P.
    Nucleic Acids Res; 2016 Sep 30; 44(17):8241-9. PubMed ID: 27325741
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  • 10. Mre11-Rad50-dependent activity of ATM/Tel1 at DNA breaks and telomeres in the absence of Nbs1.
    Limbo O, Yamada Y, Russell P.
    Mol Biol Cell; 2018 Jun 01; 29(11):1389-1399. PubMed ID: 29851556
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  • 11. Mre11-Rad50-Nbs1 is a keystone complex connecting DNA repair machinery, double-strand break signaling, and the chromatin template.
    Williams RS, Williams JS, Tainer JA.
    Biochem Cell Biol; 2007 Aug 01; 85(4):509-20. PubMed ID: 17713585
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  • 12. Nbs1 flexibly tethers Ctp1 and Mre11-Rad50 to coordinate DNA double-strand break processing and repair.
    Williams RS, Dodson GE, Limbo O, Yamada Y, Williams JS, Guenther G, Classen S, Glover JN, Iwasaki H, Russell P, Tainer JA.
    Cell; 2009 Oct 02; 139(1):87-99. PubMed ID: 19804755
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  • 13. Ctp1 is a cell-cycle-regulated protein that functions with Mre11 complex to control double-strand break repair by homologous recombination.
    Limbo O, Chahwan C, Yamada Y, de Bruin RA, Wittenberg C, Russell P.
    Mol Cell; 2007 Oct 12; 28(1):134-46. PubMed ID: 17936710
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  • 14. Molecular insights into the activation of Mre11-Rad50 endonuclease activity by Sae2/CtIP.
    Nicolas Y, Bret H, Cannavo E, Acharya A, Cejka P, Borde V, Guerois R.
    Mol Cell; 2024 Jun 20; 84(12):2223-2237.e4. PubMed ID: 38870937
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  • 15. Mutation of Conserved Mre11 Residues Alter Protein Dynamics to Separate Nuclease Functions.
    Rahman S, Beikzadeh M, Canny MD, Kaur N, Latham MP.
    J Mol Biol; 2020 May 01; 432(10):3289-3308. PubMed ID: 32246962
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  • 16. Mre11 nuclease activity has essential roles in DNA repair and genomic stability distinct from ATM activation.
    Buis J, Wu Y, Deng Y, Leddon J, Westfield G, Eckersdorff M, Sekiguchi JM, Chang S, Ferguson DO.
    Cell; 2008 Oct 03; 135(1):85-96. PubMed ID: 18854157
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  • 17. Molecular characterization of the role of the Schizosaccharomyces pombe nip1+/ctp1+ gene in DNA double-strand break repair in association with the Mre11-Rad50-Nbs1 complex.
    Akamatsu Y, Murayama Y, Yamada T, Nakazaki T, Tsutsui Y, Ohta K, Iwasaki H.
    Mol Cell Biol; 2008 Jun 03; 28(11):3639-51. PubMed ID: 18378696
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  • 18. Structural biochemistry and interaction architecture of the DNA double-strand break repair Mre11 nuclease and Rad50-ATPase.
    Hopfner KP, Karcher A, Craig L, Woo TT, Carney JP, Tainer JA.
    Cell; 2001 May 18; 105(4):473-85. PubMed ID: 11371344
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  • 19. A mutant allele of MRE11 found in mismatch repair-deficient tumor cells suppresses the cellular response to DNA replication fork stress in a dominant negative manner.
    Wen Q, Scorah J, Phear G, Rodgers G, Rodgers S, Meuth M.
    Mol Biol Cell; 2008 Apr 18; 19(4):1693-705. PubMed ID: 18256278
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