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Journal Abstract Search

311 related items for PubMed ID: 23124096

  • 1. Overexpression of DYRK1A inhibits choline acetyltransferase induction by oleic acid in cellular models of Down syndrome.
    Hijazi M, Fillat C, Medina JM, Velasco A.
    Exp Neurol; 2013 Jan; 239():229-34. PubMed ID: 23124096
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  • 2. Restrained Phosphatidylcholine Synthesis in a Cellular Model of Down's Syndrome is Associated with the Overexpression of Dyrk1A.
    Hijazi M, Medina JM, Velasco A.
    Mol Neurobiol; 2017 Mar; 54(2):1092-1100. PubMed ID: 26803494
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  • 3. A commentary on: Overexpression of Dyrk1A inhibits choline acetyltransferase induction by oleic acid in cellular models of Down syndrome.
    Dierssen M.
    Exp Neurol; 2013 Sep; 247():110-2. PubMed ID: 23570900
    [No Abstract] [Full Text] [Related]

  • 4. Effect of DYRK1A activity inhibition on development of neuronal progenitors isolated from Ts65Dn mice.
    Mazur-Kolecka B, Golabek A, Kida E, Rabe A, Hwang YW, Adayev T, Wegiel J, Flory M, Kaczmarski W, Marchi E, Frackowiak J.
    J Neurosci Res; 2012 May; 90(5):999-1010. PubMed ID: 22252917
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  • 5. Dual-specificity tyrosine(Y)-phosphorylation regulated kinase 1A-mediated phosphorylation of amyloid precursor protein: evidence for a functional link between Down syndrome and Alzheimer's disease.
    Ryoo SR, Cho HJ, Lee HW, Jeong HK, Radnaabazar C, Kim YS, Kim MJ, Son MY, Seo H, Chung SH, Song WJ.
    J Neurochem; 2008 Mar; 104(5):1333-44. PubMed ID: 18005339
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  • 7. Form of dual-specificity tyrosine-(Y)-phosphorylation-regulated kinase 1A nonphosphorylated at tyrosine 145 and 147 is enriched in the nuclei of astroglial cells, adult hippocampal progenitors, and some cholinergic axon terminals.
    Kida E, Walus M, Jarząbek K, Palminiello S, Albertini G, Rabe A, Hwang YW, Golabek AA.
    Neuroscience; 2011 Nov 10; 195():112-27. PubMed ID: 21878370
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  • 8. Engineering DYRK1A overdosage yields Down syndrome-characteristic cortical splicing aberrations.
    Toiber D, Azkona G, Ben-Ari S, Torán N, Soreq H, Dierssen M.
    Neurobiol Dis; 2010 Oct 10; 40(1):348-59. PubMed ID: 20600907
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  • 11. Overexpression of Dyrk1A causes the defects in synaptic vesicle endocytosis.
    Kim Y, Park J, Song WJ, Chang S.
    Neurosignals; 2010 Oct 10; 18(3):164-72. PubMed ID: 21135538
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  • 13. Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes.
    García-Cerro S, Rueda N, Vidal V, Lantigua S, Martínez-Cué C.
    Neurobiol Dis; 2017 Oct 10; 106():76-88. PubMed ID: 28647555
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  • 17. Dyrk1A overexpression in immortalized hippocampal cells produces the neuropathological features of Down syndrome.
    Park J, Yang EJ, Yoon JH, Chung KC.
    Mol Cell Neurosci; 2007 Oct 10; 36(2):270-9. PubMed ID: 17720532
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  • 19. Epigallocatechin-3-gallate, a DYRK1A inhibitor, rescues cognitive deficits in Down syndrome mouse models and in humans.
    De la Torre R, De Sola S, Pons M, Duchon A, de Lagran MM, Farré M, Fitó M, Benejam B, Langohr K, Rodriguez J, Pujadas M, Bizot JC, Cuenca A, Janel N, Catuara S, Covas MI, Blehaut H, Herault Y, Delabar JM, Dierssen M.
    Mol Nutr Food Res; 2014 Feb 10; 58(2):278-88. PubMed ID: 24039182
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  • 20. Constitutive Dyrk1A is abnormally expressed in Alzheimer disease, Down syndrome, Pick disease, and related transgenic models.
    Ferrer I, Barrachina M, Puig B, Martínez de Lagrán M, Martí E, Avila J, Dierssen M.
    Neurobiol Dis; 2005 Nov 10; 20(2):392-400. PubMed ID: 16242644
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