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1149 related items for PubMed ID: 23158927

  • 1. Diallyl trisufide (DATS) suppresses high glucose-induced cardiomyocyte apoptosis by inhibiting JNK/NFκB signaling via attenuating ROS generation.
    Kuo WW, Wang WJ, Tsai CY, Way CL, Hsu HH, Chen LM.
    Int J Cardiol; 2013 Sep 20; 168(1):270-80. PubMed ID: 23158927
    [Abstract] [Full Text] [Related]

  • 2. Antioxidant effects of diallyl trisulfide on high glucose-induced apoptosis are mediated by the PI3K/Akt-dependent activation of Nrf2 in cardiomyocytes.
    Tsai CY, Wang CC, Lai TY, Tsu HN, Wang CH, Liang HY, Kuo WW.
    Int J Cardiol; 2013 Sep 30; 168(2):1286-97. PubMed ID: 23453443
    [Abstract] [Full Text] [Related]

  • 3. Diallyl trisulfide suppresses doxorubicin-induced cardiomyocyte apoptosis by inhibiting MAPK/NF-κB signaling through attenuation of ROS generation.
    Wen SY, Tsai CY, Pai PY, Chen YW, Yang YC, Aneja R, Huang CY, Kuo WW.
    Environ Toxicol; 2018 Jan 30; 33(1):93-103. PubMed ID: 29087013
    [Abstract] [Full Text] [Related]

  • 4. NADPH oxidase-derived superoxide anion-induced apoptosis is mediated via the JNK-dependent activation of NF-κB in cardiomyocytes exposed to high glucose.
    Tsai KH, Wang WJ, Lin CW, Pai P, Lai TY, Tsai CY, Kuo WW.
    J Cell Physiol; 2012 Apr 30; 227(4):1347-57. PubMed ID: 21604272
    [Abstract] [Full Text] [Related]

  • 5. Diallyl trisulfide induces apoptosis in human breast cancer cells through ROS-mediated activation of JNK and AP-1.
    Na HK, Kim EH, Choi MA, Park JM, Kim DH, Surh YJ.
    Biochem Pharmacol; 2012 Nov 15; 84(10):1241-50. PubMed ID: 22981381
    [Abstract] [Full Text] [Related]

  • 6. Diallyl trisulfide protects against high glucose-induced cardiac apoptosis by stimulating the production of cystathionine gamma-lyase-derived hydrogen sulfide.
    Tsai CY, Wen SY, Shibu MA, Yang YC, Peng H, Wang B, Wei YM, Chang HY, Lee CY, Huang CY, Kuo WW.
    Int J Cardiol; 2015 Sep 15; 195():300-10. PubMed ID: 26056963
    [Abstract] [Full Text] [Related]

  • 7. Diallyl Trisulfide (DATS) Suppresses AGE-Induced Cardiomyocyte Apoptosis by Targeting ROS-Mediated PKCδ Activation.
    Hsieh DJ, Ng SC, Zeng RY, Padma VV, Huang CY, Kuo WW.
    Int J Mol Sci; 2020 Apr 09; 21(7):. PubMed ID: 32283691
    [Abstract] [Full Text] [Related]

  • 8. c-Jun NH(2)-terminal kinase signaling axis regulates diallyl trisulfide-induced generation of reactive oxygen species and cell cycle arrest in human prostate cancer cells.
    Antosiewicz J, Herman-Antosiewicz A, Marynowski SW, Singh SV.
    Cancer Res; 2006 May 15; 66(10):5379-86. PubMed ID: 16707465
    [Abstract] [Full Text] [Related]

  • 9. Apoptosis induction in human lung adenocarcinoma cells by oil-soluble allyl sulfides: triggers, pathways, and modulators.
    Wu XJ, Hu Y, Lamy E, Mersch-Sundermann V.
    Environ Mol Mutagen; 2009 Apr 15; 50(3):266-75. PubMed ID: 19197990
    [Abstract] [Full Text] [Related]

  • 10. Diallyl trisulfide-induced apoptosis in human prostate cancer cells involves c-Jun N-terminal kinase and extracellular-signal regulated kinase-mediated phosphorylation of Bcl-2.
    Xiao D, Choi S, Johnson DE, Vogel VG, Johnson CS, Trump DL, Lee YJ, Singh SV.
    Oncogene; 2004 Jul 22; 23(33):5594-606. PubMed ID: 15184882
    [Abstract] [Full Text] [Related]

  • 11. Diallyl trisulfide-induced apoptosis of bladder cancer cells is caspase-dependent and regulated by PI3K/Akt and JNK pathways.
    Shin DY, Kim GY, Hwang HJ, Kim WJ, Choi YH.
    Environ Toxicol Pharmacol; 2014 Jan 22; 37(1):74-83. PubMed ID: 24309133
    [Abstract] [Full Text] [Related]

  • 12. Exogenous H2S protects H9c2 cardiac cells against high glucose-induced injury and inflammation by inhibiting the activation of the NF-κB and IL-1β pathways.
    Xu W, Chen J, Lin J, Liu D, Mo L, Pan W, Feng J, Wu W, Zheng D.
    Int J Mol Med; 2015 Jan 22; 35(1):177-86. PubMed ID: 25412187
    [Abstract] [Full Text] [Related]

  • 13. Exogenous spermine ameliorates high glucose-induced cardiomyocytic apoptosis via decreasing reactive oxygen species accumulation through inhibiting p38/JNK and JAK2 pathways.
    He Y, Yang J, Li H, Shao H, Wei C, Wang Y, Li M, Xu C.
    Int J Clin Exp Pathol; 2015 Jan 22; 8(12):15537-49. PubMed ID: 26884823
    [Abstract] [Full Text] [Related]

  • 14. Diallyl disulfide induces apoptosis in human leukemia HL-60 cells through activation of JNK mediated by reactive oxygen.
    Yi L, Ji XX, Lin M, Tan H, Tang Y, Wen L, Ma YH, Su Q.
    Pharmazie; 2010 Sep 22; 65(9):693-8. PubMed ID: 21038848
    [Abstract] [Full Text] [Related]

  • 15. Diallyl trisufide protects against oxygen glucose deprivation -induced apoptosis by scavenging free radicals via the PI3K/Akt -mediated Nrf2/HO-1 signaling pathway in B35 neural cells.
    Xu XH, Li GL, Wang BA, Qin Y, Bai SR, Rong J, Deng T, Li Q.
    Brain Res; 2015 Jul 21; 1614():38-50. PubMed ID: 25896937
    [Abstract] [Full Text] [Related]

  • 16. The involvement of Nrf2 in the protective effects of diallyl disulfide on carbon tetrachloride-induced hepatic oxidative damage and inflammatory response in rats.
    Lee IC, Kim SH, Baek HS, Moon C, Kang SS, Kim SH, Kim YB, Shin IS, Kim JC.
    Food Chem Toxicol; 2014 Jan 21; 63():174-85. PubMed ID: 24246655
    [Abstract] [Full Text] [Related]

  • 17. Diallyl Trisulfide Suppresses High-Glucose-Induced Cardiomyocyte Apoptosis by Targeting Reactive Oxygen Species-Mediated Hypoxia-Inducible Factor-1α/Insulin-like Growth Factor Binding Protein 3 Activation.
    Lin KH, Wei YM, Liu CH, Liu JS, Huang IC, Viswanadha VP, Huang CY, Kuo WW.
    J Agric Food Chem; 2021 Oct 06; 69(39):11696-11708. PubMed ID: 34558885
    [Abstract] [Full Text] [Related]

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  • 20. Propofol post-conditioning protects against cardiomyocyte apoptosis in hypoxia/reoxygenation injury by suppressing nuclear factor-kappa B translocation via extracellular signal-regulated kinase mitogen-activated protein kinase pathway.
    Li H, Tan J, Zou Z, Huang CG, Shi XY.
    Eur J Anaesthesiol; 2011 Jul 06; 28(7):525-34. PubMed ID: 21666544
    [Abstract] [Full Text] [Related]

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