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Journal Abstract Search

237 related items for PubMed ID: 23219306

  • 21. Biological and clinical characteristics of individuals at risk for spinocerebellar ataxia types 1, 2, 3, and 6 in the longitudinal RISCA study: analysis of baseline data.
    Jacobi H, Reetz K, du Montcel ST, Bauer P, Mariotti C, Nanetti L, Rakowicz M, Sulek A, Durr A, Charles P, Filla A, Antenora A, Schöls L, Schicks J, Infante J, Kang JS, Timmann D, Di Fabio R, Masciullo M, Baliko L, Melegh B, Boesch S, Bürk K, Peltz A, Schulz JB, Dufaure-Garé I, Klockgether T.
    Lancet Neurol; 2013 Jul; 12(7):650-8. PubMed ID: 23707147
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  • 25. Conversion of individuals at risk for spinocerebellar ataxia types 1, 2, 3, and 6 to manifest ataxia (RISCA): a longitudinal cohort study.
    Jacobi H, du Montcel ST, Romanzetti S, Harmuth F, Mariotti C, Nanetti L, Rakowicz M, Makowicz G, Durr A, Monin ML, Filla A, Roca A, Schöls L, Hengel H, Infante J, Kang JS, Timmann D, Casali C, Masciullo M, Baliko L, Melegh B, Nachbauer W, Bürk-Gergs K, Schulz JB, Riess O, Reetz K, Klockgether T.
    Lancet Neurol; 2020 Sep; 19(9):738-747. PubMed ID: 32822634
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  • 27. Gray matter volume deficits in spinocerebellar ataxia: an optimized voxel based morphometric study.
    Goel G, Pal PK, Ravishankar S, Venkatasubramanian G, Jayakumar PN, Krishna N, Purushottam M, Saini J, Faruq M, Mukherji M, Jain S.
    Parkinsonism Relat Disord; 2011 Aug; 17(7):521-7. PubMed ID: 21600833
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  • 28. Long-term disease progression in spinocerebellar ataxia types 1, 2, 3, and 6: a longitudinal cohort study.
    Jacobi H, du Montcel ST, Bauer P, Giunti P, Cook A, Labrum R, Parkinson MH, Durr A, Brice A, Charles P, Marelli C, Mariotti C, Nanetti L, Panzeri M, Rakowicz M, Sulek A, Sobanska A, Schmitz-Hübsch T, Schöls L, Hengel H, Baliko L, Melegh B, Filla A, Antenora A, Infante J, Berciano J, van de Warrenburg BP, Timmann D, Szymanski S, Boesch S, Kang JS, Pandolfo M, Schulz JB, Molho S, Diallo A, Klockgether T.
    Lancet Neurol; 2015 Nov; 14(11):1101-8. PubMed ID: 26377379
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  • 32. [Copy number variation of trinucleotide repeat in dynamic mutation sites of autosomal dominant cerebellar ataxias related genes].
    Chen P, Ma M, Shang H, Su D, Zhang S, Yang Y.
    Zhonghua Yi Xue Yi Chuan Xue Za Zhi; 2009 Dec; 26(6):626-33. PubMed ID: 19953483
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  • 33. Consistent affection of the central somatosensory system in spinocerebellar ataxia type 2 and type 3 and its significance for clinical symptoms and rehabilitative therapy.
    Rüb U, Seidel K, Ozerden I, Gierga K, Brunt ER, Schöls L, de Vos RA, den Dunnen W, Schultz C, Auburger G, Deller T.
    Brain Res Rev; 2007 Feb; 53(2):235-49. PubMed ID: 17014911
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  • 36. Peripheral Neuropathy in Spinocerebellar Ataxia Type 1, 2, 3, and 6.
    Linnemann C, Tezenas du Montcel S, Rakowicz M, Schmitz-Hübsch T, Szymanski S, Berciano J, van de Warrenburg BP, Pedersen K, Depondt C, Rola R, Klockgether T, García A, Mutlu G, Schöls L.
    Cerebellum; 2016 Apr; 15(2):165-73. PubMed ID: 26054379
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  • 38. No parkinsonism in SCA2 and SCA3 despite severe neurodegeneration of the dopaminergic substantia nigra.
    Schöls L, Reimold M, Seidel K, Globas C, Brockmann K, Hauser TK, Auburger G, Bürk K, den Dunnen W, Reischl G, Korf HW, Brunt ER, Rüb U.
    Brain; 2015 Nov; 138(Pt 11):3316-26. PubMed ID: 26362908
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  • 40. Motor cortical dysfunction develops in spinocerebellar ataxia type 3.
    Farrar MA, Vucic S, Nicholson G, Kiernan MC.
    Clin Neurophysiol; 2016 Nov; 127(11):3418-3424. PubMed ID: 27689815
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