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Journal Abstract Search

597 related items for PubMed ID: 23220201

  • 1. Normalization of Dyrk1A expression by AAV2/1-shDyrk1A attenuates hippocampal-dependent defects in the Ts65Dn mouse model of Down syndrome.
    Altafaj X, Martín ED, Ortiz-Abalia J, Valderrama A, Lao-Peregrín C, Dierssen M, Fillat C.
    Neurobiol Dis; 2013 Apr; 52():117-27. PubMed ID: 23220201
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  • 5. Abnormal mineralization of the Ts65Dn Down syndrome mouse appendicular skeleton begins during embryonic development in a Dyrk1a-independent manner.
    Blazek JD, Malik AM, Tischbein M, Arbones ML, Moore CS, Roper RJ.
    Mech Dev; 2015 May; 136():133-42. PubMed ID: 25556111
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  • 6. Overexpression of Dyrk1A is implicated in several cognitive, electrophysiological and neuromorphological alterations found in a mouse model of Down syndrome.
    García-Cerro S, Martínez P, Vidal V, Corrales A, Flórez J, Vidal R, Rueda N, Arbonés ML, Martínez-Cué C.
    PLoS One; 2014 May; 9(9):e106572. PubMed ID: 25188425
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  • 8. Triplication of DYRK1A causes retinal structural and functional alterations in Down syndrome.
    Laguna A, Barallobre MJ, Marchena MÁ, Mateus C, Ramírez E, Martínez-Cue C, Delabar JM, Castelo-Branco M, de la Villa P, Arbonés ML.
    Hum Mol Genet; 2013 Jul 15; 22(14):2775-84. PubMed ID: 23512985
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  • 10. Environmental enrichment rescues DYRK1A activity and hippocampal adult neurogenesis in TgDyrk1A.
    Pons-Espinal M, Martinez de Lagran M, Dierssen M.
    Neurobiol Dis; 2013 Dec 15; 60():18-31. PubMed ID: 23969234
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  • 12. Constitutive Dyrk1A is abnormally expressed in Alzheimer disease, Down syndrome, Pick disease, and related transgenic models.
    Ferrer I, Barrachina M, Puig B, Martínez de Lagrán M, Martí E, Avila J, Dierssen M.
    Neurobiol Dis; 2005 Nov 15; 20(2):392-400. PubMed ID: 16242644
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  • 13. DYRK1A BAC transgenic mice show altered synaptic plasticity with learning and memory defects.
    Ahn KJ, Jeong HK, Choi HS, Ryoo SR, Kim YJ, Goo JS, Choi SY, Han JS, Ha I, Song WJ.
    Neurobiol Dis; 2006 Jun 15; 22(3):463-72. PubMed ID: 16455265
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  • 18. Evidence that increased Kcnj6 gene dose is necessary for deficits in behavior and dentate gyrus synaptic plasticity in the Ts65Dn mouse model of Down syndrome.
    Kleschevnikov AM, Yu J, Kim J, Lysenko LV, Zeng Z, Yu YE, Mobley WC.
    Neurobiol Dis; 2017 Jul 15; 103():1-10. PubMed ID: 28342823
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  • 20. Dual-specificity tyrosine(Y)-phosphorylation regulated kinase 1A-mediated phosphorylation of amyloid precursor protein: evidence for a functional link between Down syndrome and Alzheimer's disease.
    Ryoo SR, Cho HJ, Lee HW, Jeong HK, Radnaabazar C, Kim YS, Kim MJ, Son MY, Seo H, Chung SH, Song WJ.
    J Neurochem; 2008 Mar 15; 104(5):1333-44. PubMed ID: 18005339
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