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Journal Abstract Search

197 related items for PubMed ID: 23263329

  • 1. Hierarchical accumulation of RyR post-translational modifications drives disease progression in dystrophic cardiomyopathy.
    Kyrychenko S, Poláková E, Kang C, Pocsai K, Ullrich ND, Niggli E, Shirokova N.
    Cardiovasc Res; 2013 Mar 15; 97(4):666-75. PubMed ID: 23263329
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  • 2. Hypersensitivity of excitation-contraction coupling in dystrophic cardiomyocytes.
    Ullrich ND, Fanchaouy M, Gusev K, Shirokova N, Niggli E.
    Am J Physiol Heart Circ Physiol; 2009 Dec 15; 297(6):H1992-2003. PubMed ID: 19783774
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  • 3. Intracellular calcium handling in ventricular myocytes from mdx mice.
    Williams IA, Allen DG.
    Am J Physiol Heart Circ Physiol; 2007 Feb 15; 292(2):H846-55. PubMed ID: 17012353
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  • 4. Oxidized CaMKII (Ca2+/Calmodulin-Dependent Protein Kinase II) Is Essential for Ventricular Arrhythmia in a Mouse Model of Duchenne Muscular Dystrophy.
    Wang Q, Quick AP, Cao S, Reynolds J, Chiang DY, Beavers D, Li N, Wang G, Rodney GG, Anderson ME, Wehrens XHT.
    Circ Arrhythm Electrophysiol; 2018 Apr 15; 11(4):e005682. PubMed ID: 29654126
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  • 5. Genetic inhibition of PKA phosphorylation of RyR2 prevents dystrophic cardiomyopathy.
    Sarma S, Li N, van Oort RJ, Reynolds C, Skapura DG, Wehrens XH.
    Proc Natl Acad Sci U S A; 2010 Jul 20; 107(29):13165-70. PubMed ID: 20615971
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  • 6. Crosstalk between RyR2 oxidation and phosphorylation contributes to cardiac dysfunction in mice with Duchenne muscular dystrophy.
    Wang Q, Wang W, Wang G, Rodney GG, Wehrens XH.
    J Mol Cell Cardiol; 2015 Dec 20; 89(Pt B):177-84. PubMed ID: 26555638
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  • 11. Calcium current properties in dystrophin-deficient ventricular cardiomyocytes from aged mdx mice.
    Rubi L, Todt H, Kubista H, Koenig X, Hilber K.
    Physiol Rep; 2018 Jan 20; 6(1):. PubMed ID: 29333726
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  • 12. Blunted cardiac beta-adrenergic response as an early indication of cardiac dysfunction in Duchenne muscular dystrophy.
    Li Y, Zhang S, Zhang X, Li J, Ai X, Zhang L, Yu D, Ge S, Peng Y, Chen X.
    Cardiovasc Res; 2014 Jul 01; 103(1):60-71. PubMed ID: 24812281
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  • 14. Calcium/calmodulin-dependent kinase II and nitric oxide synthase 1-dependent modulation of ryanodine receptors during β-adrenergic stimulation is restricted to the dyadic cleft.
    Dries E, Santiago DJ, Johnson DM, Gilbert G, Holemans P, Korte SM, Roderick HL, Sipido KR.
    J Physiol; 2016 Oct 15; 594(20):5923-5939. PubMed ID: 27121757
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  • 20. Oxidation- and CaMKII-mediated sarcoplasmic reticulum Ca(2+) leak triggers atrial fibrillation in aging.
    Guo X, Yuan S, Liu Z, Fang Q.
    J Cardiovasc Electrophysiol; 2014 Jun 15; 25(6):645-52. PubMed ID: 24576293
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