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Journal Abstract Search

219 related items for PubMed ID: 23279377

  • 1.
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  • 2. Calcium/calmodulin-dependent protein kinase II contributes to cardiac arrhythmogenesis in heart failure.
    Sag CM, Wadsack DP, Khabbazzadeh S, Abesser M, Grefe C, Neumann K, Opiela MK, Backs J, Olson EN, Brown JH, Neef S, Maier SK, Maier LS.
    Circ Heart Fail; 2009 Nov; 2(6):664-75. PubMed ID: 19919992
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  • 3. CaMKII inhibition in heart failure, beneficial, harmful, or both.
    Cheng J, Xu L, Lai D, Guilbert A, Lim HJ, Keskanokwong T, Wang Y.
    Am J Physiol Heart Circ Physiol; 2012 Apr 01; 302(7):H1454-65. PubMed ID: 22287581
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  • 5. {beta}1-Adrenergic receptor activation induces mouse cardiac myocyte death through both L-type calcium channel-dependent and -independent pathways.
    Wang W, Zhang H, Gao H, Kubo H, Berretta RM, Chen X, Houser SR.
    Am J Physiol Heart Circ Physiol; 2010 Aug 01; 299(2):H322-31. PubMed ID: 20495143
    [Abstract] [Full Text] [Related]

  • 6. Reactive oxygen species-activated Ca/calmodulin kinase IIδ is required for late I(Na) augmentation leading to cellular Na and Ca overload.
    Wagner S, Ruff HM, Weber SL, Bellmann S, Sowa T, Schulte T, Anderson ME, Grandi E, Bers DM, Backs J, Belardinelli L, Maier LS.
    Circ Res; 2011 Mar 04; 108(5):555-65. PubMed ID: 21252154
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  • 7. miR-1 overexpression enhances Ca(2+) release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII-dependent hyperphosphorylation of RyR2.
    Terentyev D, Belevych AE, Terentyeva R, Martin MM, Malana GE, Kuhn DE, Abdellatif M, Feldman DS, Elton TS, Györke S.
    Circ Res; 2009 Feb 27; 104(4):514-21. PubMed ID: 19131648
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  • 8. CaMKII inhibition has dual effects on spontaneous Ca2+ release and Ca2+ alternans in ventricular cardiomyocytes from mice with a gain-of-function RyR2 mutation.
    Sadredini M, Haugsten Hansen M, Frisk M, Louch WE, Lehnart SE, Sjaastad I, Stokke MK.
    Am J Physiol Heart Circ Physiol; 2021 Aug 01; 321(2):H446-H460. PubMed ID: 34270372
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  • 9. CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation.
    Grimm M, Ling H, Willeford A, Pereira L, Gray CB, Erickson JR, Sarma S, Respress JL, Wehrens XH, Bers DM, Brown JH.
    J Mol Cell Cardiol; 2015 Aug 01; 85():282-91. PubMed ID: 26080362
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  • 10. CaMKIIδ Met281/282 oxidation is not required for recovery of calcium transients during acidosis.
    Kreitmeier KG, Tarnowski D, Nanadikar MS, Baier MJ, Wagner S, Katschinski DM, Maier LS, Sag CM.
    Am J Physiol Heart Circ Physiol; 2021 Mar 01; 320(3):H1199-H1212. PubMed ID: 33449853
    [Abstract] [Full Text] [Related]

  • 11. CaMKII-dependent SR Ca leak contributes to doxorubicin-induced impaired Ca handling in isolated cardiac myocytes.
    Sag CM, Köhler AC, Anderson ME, Backs J, Maier LS.
    J Mol Cell Cardiol; 2011 Nov 01; 51(5):749-59. PubMed ID: 21819992
    [Abstract] [Full Text] [Related]

  • 12. Hyperglycemia Acutely Increases Cytosolic Reactive Oxygen Species via O-linked GlcNAcylation and CaMKII Activation in Mouse Ventricular Myocytes.
    Lu S, Liao Z, Lu X, Katschinski DM, Mercola M, Chen J, Heller Brown J, Molkentin JD, Bossuyt J, Bers DM.
    Circ Res; 2020 May 08; 126(10):e80-e96. PubMed ID: 32134364
    [Abstract] [Full Text] [Related]

  • 13. Oxidative-stress-induced afterdepolarizations and calmodulin kinase II signaling.
    Xie LH, Chen F, Karagueuzian HS, Weiss JN.
    Circ Res; 2009 Jan 02; 104(1):79-86. PubMed ID: 19038865
    [Abstract] [Full Text] [Related]

  • 14. NADPH oxidase 2 mediates angiotensin II-dependent cellular arrhythmias via PKA and CaMKII.
    Wagner S, Dantz C, Flebbe H, Azizian A, Sag CM, Engels S, Möllencamp J, Dybkova N, Islam T, Shah AM, Maier LS.
    J Mol Cell Cardiol; 2014 Oct 02; 75():206-15. PubMed ID: 25073061
    [Abstract] [Full Text] [Related]

  • 15. Phospholamban ablation rescues the enhanced propensity to arrhythmias of mice with CaMKII-constitutive phosphorylation of RyR2 at site S2814.
    Mazzocchi G, Sommese L, Palomeque J, Felice JI, Di Carlo MN, Fainstein D, Gonzalez P, Contreras P, Skapura D, McCauley MD, Lascano EC, Negroni JA, Kranias EG, Wehrens XH, Valverde CA, Mattiazzi A.
    J Physiol; 2016 Jun 01; 594(11):3005-30. PubMed ID: 26695843
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  • 18. Enhanced late INa induces proarrhythmogenic SR Ca leak in a CaMKII-dependent manner.
    Sag CM, Mallwitz A, Wagner S, Hartmann N, Schotola H, Fischer TH, Ungeheuer N, Herting J, Shah AM, Maier LS, Sossalla S, Unsöld B.
    J Mol Cell Cardiol; 2014 Nov 01; 76():94-105. PubMed ID: 25173923
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  • 19. The novel CaMKII inhibitor GS-680 reduces diastolic SR Ca leak and prevents CaMKII-dependent pro-arrhythmic activity.
    Lebek S, Plößl A, Baier M, Mustroph J, Tarnowski D, Lücht CM, Schopka S, Flörchinger B, Schmid C, Zausig Y, Pagratis N, Marchand B, Koltun DO, Hung WK, Ahmadyar S, Belardinelli L, Maier LS, Wagner S.
    J Mol Cell Cardiol; 2018 May 01; 118():159-168. PubMed ID: 29614261
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