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Journal Abstract Search

203 related items for PubMed ID: 23285138

  • 21. Downregulation of Mcl-1 through GSK-3β activation contributes to arsenic trioxide-induced apoptosis in acute myeloid leukemia cells.
    Wang R, Xia L, Gabrilove J, Waxman S, Jing Y.
    Leukemia; 2013 Feb; 27(2):315-24. PubMed ID: 22751450
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  • 22. Alteration in miRNA gene expression pattern in acute promyelocytic leukemia cell induced by arsenic trioxide: a possible mechanism to explain arsenic multi-target action.
    Ghaffari SH, Bashash D, Dizaji MZ, Ghavamzadeh A, Alimoghaddam K.
    Tumour Biol; 2012 Feb; 33(1):157-72. PubMed ID: 22072212
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  • 23. Bone marrow stroma-induced resistance of chronic lymphocytic leukemia cells to arsenic trioxide involves Mcl-1 upregulation and is overcome by inhibiting the PI3Kδ or PKCβ signaling pathways.
    Amigo-Jiménez I, Bailón E, Aguilera-Montilla N, Terol MJ, García-Marco JA, García-Pardo A.
    Oncotarget; 2015 Dec 29; 6(42):44832-48. PubMed ID: 26540567
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  • 24. Induction of apoptosis in arsenic trioxide-treated lung cancer A549 cells by buthionine sulfoximine.
    Han YH, Kim SZ, Kim SH, Park WH.
    Mol Cells; 2008 Aug 31; 26(2):158-64. PubMed ID: 18596414
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  • 25. Proteomic and functional analyses reveal a dual molecular mechanism underlying arsenic-induced apoptosis in human multiple myeloma cells.
    Ge F, Lu XP, Zeng HL, He QY, Xiong S, Jin L, He QY.
    J Proteome Res; 2009 Jun 31; 8(6):3006-19. PubMed ID: 19364129
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  • 26. Bcl-2 hijacks the arsenic trioxide resistance in SH-SY5Y cells.
    Wang J, Peng X, Yang D, Guo M, Xu X, Yin F, Wang Y, Huang J, Zhan L, Qi Z.
    J Cell Mol Med; 2022 Jan 31; 26(2):563-569. PubMed ID: 34910369
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  • 27. Potentiation of arsenic trioxide-induced apoptosis by 8-bromo-7-methoxychrysin in human leukemia cells involves depletion of intracellular reduced glutathione.
    Xiao G, Tang X, Yao C, Wang C.
    Acta Biochim Biophys Sin (Shanghai); 2011 Sep 31; 43(9):712-21. PubMed ID: 21785114
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  • 29. Arsenic trioxide inhibits the growth of Calu-6 cells via inducing a G2 arrest of the cell cycle and apoptosis accompanied with the depletion of GSH.
    Han YH, Kim SZ, Kim SH, Park WH.
    Cancer Lett; 2008 Oct 18; 270(1):40-55. PubMed ID: 18539383
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  • 33. Characterization of molecular events in a series of bladder urothelial carcinoma cell lines with progressive resistance to arsenic trioxide.
    Hour TC, Huang CY, Lin CC, Chen J, Guan JY, Lee JM, Pu YS.
    Anticancer Drugs; 2004 Sep 18; 15(8):779-85. PubMed ID: 15494640
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  • 35. Arsenic trioxide cytotoxicity in steroid and chemotherapy-resistant myeloma cell lines: enhancement of apoptosis by manipulation of cellular redox state.
    Gartenhaus RB, Prachand SN, Paniaqua M, Li Y, Gordon LI.
    Clin Cancer Res; 2002 Feb 18; 8(2):566-72. PubMed ID: 11839678
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  • 36. Ascorbic acid enhances arsenic trioxide-induced cytotoxicity in multiple myeloma cells.
    Grad JM, Bahlis NJ, Reis I, Oshiro MM, Dalton WS, Boise LH.
    Blood; 2001 Aug 01; 98(3):805-13. PubMed ID: 11468182
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  • 37. Arsenic trioxide-induced apoptosis in myeloma cells: p53-dependent G1 or G2/M cell cycle arrest, activation of caspase-8 or caspase-9, and synergy with APO2/TRAIL.
    Liu Q, Hilsenbeck S, Gazitt Y.
    Blood; 2003 May 15; 101(10):4078-87. PubMed ID: 12531793
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