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Journal Abstract Search

399 related items for PubMed ID: 23333620

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  • 3. Different dynamic movements of wild-type and pathogenic VCPs and their cofactors to damaged mitochondria in a Parkin-mediated mitochondrial quality control system.
    Kimura Y, Fukushi J, Hori S, Matsuda N, Okatsu K, Kakiyama Y, Kawawaki J, Kakizuka A, Tanaka K.
    Genes Cells; 2013 Dec; 18(12):1131-43. PubMed ID: 24215292
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  • 4. Inclusion body myopathy-associated mutations in p97/VCP impair endoplasmic reticulum-associated degradation.
    Weihl CC, Dalal S, Pestronk A, Hanson PI.
    Hum Mol Genet; 2006 Jan 15; 15(2):189-99. PubMed ID: 16321991
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  • 7. Transgenic mice expressing mutant forms VCP/p97 recapitulate the full spectrum of IBMPFD including degeneration in muscle, brain and bone.
    Custer SK, Neumann M, Lu H, Wright AC, Taylor JP.
    Hum Mol Genet; 2010 May 01; 19(9):1741-55. PubMed ID: 20147319
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  • 9. Inclusion body myopathy, Paget's disease of the bone and fronto-temporal dementia: a disorder of autophagy.
    Ju JS, Weihl CC.
    Hum Mol Genet; 2010 Apr 15; 19(R1):R38-45. PubMed ID: 20410287
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  • 10. A novel VCP mutation as the cause of atypical IBMPFD in a Chinese family.
    Gu JM, Ke YH, Yue H, Liu YJ, Zhang Z, Zhang H, Hu WW, Wang C, He JW, Hu YQ, Li M, Fu WZ, Zhang ZL.
    Bone; 2013 Jan 15; 52(1):9-16. PubMed ID: 23000505
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  • 13. Nucleocytoplasmic shuttling of valosin-containing protein (VCP/p97) regulated by its N domain and C-terminal region.
    Song C, Wang Q, Song C, Lockett SJ, Colburn NH, Li CC, Wang JM, Rogers TJ.
    Biochim Biophys Acta; 2015 Jan 15; 1853(1):222-32. PubMed ID: 25447673
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  • 14. Pathogenic VCP/TER94 alleles are dominant actives and contribute to neurodegeneration by altering cellular ATP level in a Drosophila IBMPFD model.
    Chang YC, Hung WT, Chang YC, Chang HC, Wu CL, Chiang AS, Jackson GR, Sang TK.
    PLoS Genet; 2011 Feb 03; 7(2):e1001288. PubMed ID: 21304887
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  • 15. Structural and functional deviations in disease-associated p97 mutants.
    Tang WK, Xia D.
    J Struct Biol; 2012 Aug 03; 179(2):83-92. PubMed ID: 22579784
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  • 16. Imbalances in p97 co-factor interactions in human proteinopathy.
    Fernández-Sáiz V, Buchberger A.
    EMBO Rep; 2010 Jun 03; 11(6):479-85. PubMed ID: 20414249
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  • 17. Pathological consequences of VCP mutations on human striated muscle.
    Hübbers CU, Clemen CS, Kesper K, Böddrich A, Hofmann A, Kämäräinen O, Tolksdorf K, Stumpf M, Reichelt J, Roth U, Krause S, Watts G, Kimonis V, Wattjes MP, Reimann J, Thal DR, Biermann K, Evert BO, Lochmüller H, Wanker EE, Schoser BG, Noegel AA, Schröder R.
    Brain; 2007 Feb 03; 130(Pt 2):381-93. PubMed ID: 16984901
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  • 18. Rescue of growth defects of yeast cdc48 mutants by pathogenic IBMPFD-VCPs.
    Takata T, Kimura Y, Ohnuma Y, Kawawaki J, Kakiyama Y, Tanaka K, Kakizuka A.
    J Struct Biol; 2012 Aug 03; 179(2):93-103. PubMed ID: 22728077
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  • 19. Valosin-containing protein (VCP) is required for autophagy and is disrupted in VCP disease.
    Ju JS, Fuentealba RA, Miller SE, Jackson E, Piwnica-Worms D, Baloh RH, Weihl CC.
    J Cell Biol; 2009 Dec 14; 187(6):875-88. PubMed ID: 20008565
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  • 20. The role of a novel p97/valosin-containing protein-interacting motif of gp78 in endoplasmic reticulum-associated degradation.
    Ballar P, Shen Y, Yang H, Fang S.
    J Biol Chem; 2006 Nov 17; 281(46):35359-68. PubMed ID: 16987818
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