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954 related items for PubMed ID: 23338126

  • 1. Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibition of the p38 MAPK pathway in H9c2 cells.
    Guo R, Lin J, Xu W, Shen N, Mo L, Zhang C, Feng J.
    Int J Mol Med; 2013 Mar; 31(3):644-50. PubMed ID: 23338126
    [Abstract] [Full Text] [Related]

  • 2. Exogenous hydrogen sulfide protects H9c2 cardiac cells against high glucose-induced injury by inhibiting the activities of the p38 MAPK and ERK1/2 pathways.
    Xu W, Wu W, Chen J, Guo R, Lin J, Liao X, Feng J.
    Int J Mol Med; 2013 Oct; 32(4):917-25. PubMed ID: 23912965
    [Abstract] [Full Text] [Related]

  • 3. Exogenous hydrogen sulfide protects against doxorubicin-induced inflammation and cytotoxicity by inhibiting p38MAPK/NFκB pathway in H9c2 cardiac cells.
    Guo R, Wu K, Chen J, Mo L, Hua X, Zheng D, Chen P, Chen G, Xu W, Feng J.
    Cell Physiol Biochem; 2013 Oct; 32(6):1668-80. PubMed ID: 24356372
    [Abstract] [Full Text] [Related]

  • 4. Naringin inhibits ROS-activated MAPK pathway in high glucose-induced injuries in H9c2 cardiac cells.
    Chen J, Guo R, Yan H, Tian L, You Q, Li S, Huang R, Wu K.
    Basic Clin Pharmacol Toxicol; 2014 Apr; 114(4):293-304. PubMed ID: 24118820
    [Abstract] [Full Text] [Related]

  • 5. Exogenous H2S protects H9c2 cardiac cells against high glucose-induced injury and inflammation by inhibiting the activation of the NF-κB and IL-1β pathways.
    Xu W, Chen J, Lin J, Liu D, Mo L, Pan W, Feng J, Wu W, Zheng D.
    Int J Mol Med; 2015 Jan; 35(1):177-86. PubMed ID: 25412187
    [Abstract] [Full Text] [Related]

  • 6. Hydrogen sulfide protects H9c2 cardiac cells against doxorubicin-induced cytotoxicity through the PI3K/Akt/FoxO3a pathway.
    Liu MH, Zhang Y, He J, Tan TP, Wu SJ, Guo DM, He H, Peng J, Tang ZH, Jiang ZS.
    Int J Mol Med; 2016 Jun; 37(6):1661-8. PubMed ID: 27081862
    [Abstract] [Full Text] [Related]

  • 7. Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibiting reactive oxygen species-activated extracellular signal-regulated kinase 1/2 in H9c2 cardiac myocytes.
    Liu MH, Lin XL, Zhang Y, He J, Tan TP, Wu SJ, Liu J, Tian W, Chen L, Yu S, Li J, Yuan C.
    Mol Med Rep; 2015 Nov; 12(5):6841-8. PubMed ID: 26299281
    [Abstract] [Full Text] [Related]

  • 8. Exogenous hydrogen sulfide alleviates high glucose-induced cardiotoxicity via inhibition of leptin signaling in H9c2 cells.
    Zhuang XD, Hu X, Long M, Dong XB, Liu DH, Liao XX.
    Mol Cell Biochem; 2014 Jun; 391(1-2):147-55. PubMed ID: 24687304
    [Abstract] [Full Text] [Related]

  • 9. ATP-sensitive K⁺ channels contribute to the protective effects of exogenous hydrogen sulfide against high glucose-induced injury in H9c2 cardiac cells.
    Liang W, Chen J, Mo L, Ke X, Zhang W, Zheng D, Pan W, Wu S, Feng J, Song M, Liao X.
    Int J Mol Med; 2016 Mar; 37(3):763-72. PubMed ID: 26820501
    [Abstract] [Full Text] [Related]

  • 10. Inhibition of the leptin-induced activation of the p38 MAPK pathway contributes to the protective effects of naringin against high glucose-induced injury in H9c2 cardiac cells.
    Chen J, Mo H, Guo R, You Q, Huang R, Wu K.
    Int J Mol Med; 2014 Mar; 33(3):605-12. PubMed ID: 24399619
    [Abstract] [Full Text] [Related]

  • 11. Hydrogen sulfide attenuates doxorubicin‑induced cardiotoxicity by inhibiting calreticulin expression in H9c2 cells.
    Liu MH, Zhang Y, Lin XL, He J, Tan TP, Wu SJ, Yu S, Chen L, Chen YD, Fu HY, Yuan C, Li J.
    Mol Med Rep; 2015 Oct; 12(4):5197-202. PubMed ID: 26134131
    [Abstract] [Full Text] [Related]

  • 12. Hydrogen sulfide protects H9c2 cells against doxorubicin-induced cardiotoxicity through inhibition of endoplasmic reticulum stress.
    Wang XY, Yang CT, Zheng DD, Mo LQ, Lan AP, Yang ZL, Hu F, Chen PX, Liao XX, Feng JQ.
    Mol Cell Biochem; 2012 Apr; 363(1-2):419-26. PubMed ID: 22203419
    [Abstract] [Full Text] [Related]

  • 13. Exogenous hydrogen sulfide ameliorates high glucose-induced myocardial injury & inflammation via the CIRP-MAPK signaling pathway in H9c2 cardiac cells.
    Zhao HL, Wu BQ, Luo Y, Zhang WY, Hao YL, Liang JJ, Fang F, Liu W, Chen XH.
    Life Sci; 2018 Sep 01; 208():315-324. PubMed ID: 29857073
    [Abstract] [Full Text] [Related]

  • 14. Activation of the p38 MAPK/NF-κB pathway contributes to doxorubicin-induced inflammation and cytotoxicity in H9c2 cardiac cells.
    Guo RM, Xu WM, Lin JC, Mo LQ, Hua XX, Chen PX, Wu K, Zheng DD, Feng JQ.
    Mol Med Rep; 2013 Aug 01; 8(2):603-8. PubMed ID: 23807148
    [Abstract] [Full Text] [Related]

  • 15. Chitosan oligosaccharides prevent doxorubicin-induced oxidative stress and cardiac apoptosis through activating p38 and JNK MAPK mediated Nrf2/ARE pathway.
    Zhang Y, Ahmad KA, Khan FU, Yan S, Ihsan AU, Ding Q.
    Chem Biol Interact; 2019 May 25; 305():54-65. PubMed ID: 30928397
    [Abstract] [Full Text] [Related]

  • 16. Ghrelin inhibits doxorubicin cardiotoxicity by inhibiting excessive autophagy through AMPK and p38-MAPK.
    Wang X, Wang XL, Chen HL, Wu D, Chen JX, Wang XX, Li RL, He JH, Mo L, Cen X, Wei YQ, Jiang W.
    Biochem Pharmacol; 2014 Apr 01; 88(3):334-50. PubMed ID: 24522112
    [Abstract] [Full Text] [Related]

  • 17. Novel insights into the role of HSP90 in cytoprotection of H2S against chemical hypoxia-induced injury in H9c2 cardiac myocytes.
    Yang Z, Yang C, Xiao L, Liao X, Lan A, Wang X, Guo R, Chen P, Hu C, Feng J.
    Int J Mol Med; 2011 Sep 01; 28(3):397-403. PubMed ID: 21519787
    [Abstract] [Full Text] [Related]

  • 18. Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibiting the expression of peroxiredoxin III in H9c2 cells.
    Liu MH, Lin XL, Yuan C, He J, Tan TP, Wu SJ, Yu S, Chen L, Liu J, Tian W, Chen YD, Fu HY, Li J, Zhang Y.
    Mol Med Rep; 2016 Jan 01; 13(1):367-72. PubMed ID: 26573464
    [Abstract] [Full Text] [Related]

  • 19. Exogenous hydrogen sulfide protects against high glucose‑induced inflammation and cytotoxicity in H9c2 cardiac cells.
    Huang Z, Dong X, Zhuang X, Hu X, Wang L, Liao X.
    Mol Med Rep; 2016 Nov 01; 14(5):4911-4917. PubMed ID: 27748941
    [Abstract] [Full Text] [Related]

  • 20. Increasing ERK phosphorylation by inhibition of p38 activity protects against cadmium-induced apoptotic cell death through ERK/Drp1/p38 signaling axis in spermatocyte-derived GC-2spd cells.
    Seong JB, Bae YC, Lee HS, Huh JW, Lee SR, Lee HJ, Lee DS.
    Toxicol Appl Pharmacol; 2019 Dec 01; 384():114797. PubMed ID: 31676320
    [Abstract] [Full Text] [Related]

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