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Journal Abstract Search

246 related items for PubMed ID: 23344574

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  • 3. Persistence of 1,25D-induced hypercalciuria in alendronate-treated genetic hypercalciuric stone-forming rats fed a low-calcium diet.
    Frick KK, Asplin JR, Culbertson CD, Granja I, Krieger NS, Bushinsky DA.
    Am J Physiol Renal Physiol; 2014 May 01; 306(9):F1081-7. PubMed ID: 24573387
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  • 4. 1,25(OH)₂D₃ induces a mineralization defect and loss of bone mineral density in genetic hypercalciuric stone-forming rats.
    Ng AH, Frick KK, Krieger NS, Asplin JR, Cohen-McFarlane M, Culbertson CD, Kyker-Snowman K, Grynpas MD, Bushinsky DA.
    Calcif Tissue Int; 2014 May 01; 94(5):531-43. PubMed ID: 24481706
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  • 7. Mechanism and function of high vitamin D receptor levels in genetic hypercalciuric stone-forming rats.
    Karnauskas AJ, van Leeuwen JP, van den Bemd GJ, Kathpalia PP, DeLuca HF, Bushinsky DA, Favus MJ.
    J Bone Miner Res; 2005 Mar 01; 20(3):447-54. PubMed ID: 15746989
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  • 15. Vitamin D receptor (VDR) contributes to the development of hypercalciuria by sensitizing VDR target genes to vitamin D in a genetic hypercalciuric stone-forming (GHS) rat model.
    Guo S, Chia W, Wang H, Bushinsky DA, Zhong B, Favus MJ.
    Genes Dis; 2022 May 01; 9(3):797-806. PubMed ID: 35782986
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  • 16. Increased sensitivity to 1,25(OH)2D3 in bone from genetic hypercalciuric rats.
    Krieger NS, Stathopoulos VM, Bushinsky DA.
    Am J Physiol; 1996 Jul 01; 271(1 Pt 1):C130-5. PubMed ID: 8760038
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