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583 related items for PubMed ID: 23355489

  • 1. Ataxia telangiectasia mutated (ATM) is dispensable for endonuclease I-SceI-induced homologous recombination in mouse embryonic stem cells.
    Rass E, Chandramouly G, Zha S, Alt FW, Xie A.
    J Biol Chem; 2013 Mar 08; 288(10):7086-95. PubMed ID: 23355489
    [Abstract] [Full Text] [Related]

  • 2. Mouse but not human embryonic stem cells are deficient in rejoining of ionizing radiation-induced DNA double-strand breaks.
    Bañuelos CA, Banáth JP, MacPhail SH, Zhao J, Eaves CA, O'Connor MD, Lansdorp PM, Olive PL.
    DNA Repair (Amst); 2008 Sep 01; 7(9):1471-83. PubMed ID: 18602349
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  • 3. Functional interaction of H2AX, NBS1, and p53 in ATM-dependent DNA damage responses and tumor suppression.
    Kang J, Ferguson D, Song H, Bassing C, Eckersdorff M, Alt FW, Xu Y.
    Mol Cell Biol; 2005 Jan 01; 25(2):661-70. PubMed ID: 15632067
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  • 4. DNA-PKcs plays a dominant role in the regulation of H2AX phosphorylation in response to DNA damage and cell cycle progression.
    An J, Huang YC, Xu QZ, Zhou LJ, Shang ZF, Huang B, Wang Y, Liu XD, Wu DC, Zhou PK.
    BMC Mol Biol; 2010 Mar 06; 11():18. PubMed ID: 20205745
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  • 5. Targeting protein for xenopus kinesin-like protein 2 (TPX2) regulates γ-histone 2AX (γ-H2AX) levels upon ionizing radiation.
    Neumayer G, Helfricht A, Shim SY, Le HT, Lundin C, Belzil C, Chansard M, Yu Y, Lees-Miller SP, Gruss OJ, van Attikum H, Helleday T, Nguyen MD.
    J Biol Chem; 2012 Dec 07; 287(50):42206-22. PubMed ID: 23045526
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  • 9. Inhibition of poly(ADP-ribose) polymerase (PARP) and ataxia telangiectasia mutated (ATM) on the chemosensitivity of mantle cell lymphoma to agents that induce DNA strand breaks.
    Golla RM, Li M, Shen Y, Ji M, Yan Y, Fu K, Greiner TC, McKeithan TW, Chan WC.
    Hematol Oncol; 2012 Dec 07; 30(4):175-9. PubMed ID: 22170260
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  • 11. Ataxia telangiectasia mutated (ATM) signaling network is modulated by a novel poly(ADP-ribose)-dependent pathway in the early response to DNA-damaging agents.
    Haince JF, Kozlov S, Dawson VL, Dawson TM, Hendzel MJ, Lavin MF, Poirier GG.
    J Biol Chem; 2007 Jun 01; 282(22):16441-53. PubMed ID: 17428792
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  • 13. ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation.
    Stiff T, O'Driscoll M, Rief N, Iwabuchi K, Löbrich M, Jeggo PA.
    Cancer Res; 2004 Apr 01; 64(7):2390-6. PubMed ID: 15059890
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  • 14. Live-cell tracking of γ-H2AX kinetics reveals the distinct modes of ATM and DNA-PK in the immediate response to DNA damage.
    Trakarnphornsombat W, Kimura H.
    J Cell Sci; 2023 Apr 15; 136(8):. PubMed ID: 36999484
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  • 15. ATM damage response and XLF repair factor are functionally redundant in joining DNA breaks.
    Zha S, Guo C, Boboila C, Oksenych V, Cheng HL, Zhang Y, Wesemann DR, Yuen G, Patel H, Goff PH, Dubois RL, Alt FW.
    Nature; 2011 Jan 13; 469(7329):250-4. PubMed ID: 21160472
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  • 16. Clustered DNA damage induces pan-nuclear H2AX phosphorylation mediated by ATM and DNA-PK.
    Meyer B, Voss KO, Tobias F, Jakob B, Durante M, Taucher-Scholz G.
    Nucleic Acids Res; 2013 Jul 13; 41(12):6109-18. PubMed ID: 23620287
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  • 17. Differential requirement for H2AX and 53BP1 in organismal development and genome maintenance in the absence of poly(ADP)ribosyl polymerase 1.
    Orsburn B, Escudero B, Prakash M, Gesheva S, Liu G, Huso DL, Franco S.
    Mol Cell Biol; 2010 May 13; 30(10):2341-52. PubMed ID: 20231360
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  • 18. Assessment of ATM phosphorylation on Ser-1981 induced by DNA topoisomerase I and II inhibitors in relation to Ser-139-histone H2AX phosphorylation, cell cycle phase, and apoptosis.
    Kurose A, Tanaka T, Huang X, Halicka HD, Traganos F, Dai W, Darzynkiewicz Z.
    Cytometry A; 2005 Nov 13; 68(1):1-9. PubMed ID: 16184611
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  • 19. Role of autophagy in chemoresistance: regulation of the ATM-mediated DNA-damage signaling pathway through activation of DNA-PKcs and PARP-1.
    Yoon JH, Ahn SG, Lee BH, Jung SH, Oh SH.
    Biochem Pharmacol; 2012 Mar 15; 83(6):747-57. PubMed ID: 22226932
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