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406 related items for PubMed ID: 23444197

  • 1. Melatonin prevents dopaminergic cell loss induced by lentiviral vectors expressing A30P mutant alpha-synuclein.
    Brito-Armas JM, Baekelandt V, Castro-Hernández JR, González-Hernández T, Rodríguez M, Castro R.
    Histol Histopathol; 2013 Aug; 28(8):999-1006. PubMed ID: 23444197
    [Abstract] [Full Text] [Related]

  • 2. Lentiviral nigral delivery of GDNF does not prevent neurodegeneration in a genetic rat model of Parkinson's disease.
    Lo Bianco C, Déglon N, Pralong W, Aebischer P.
    Neurobiol Dis; 2004 Nov; 17(2):283-9. PubMed ID: 15474365
    [Abstract] [Full Text] [Related]

  • 3. alpha -Synucleinopathy and selective dopaminergic neuron loss in a rat lentiviral-based model of Parkinson's disease.
    Lo Bianco C, Ridet JL, Schneider BL, Deglon N, Aebischer P.
    Proc Natl Acad Sci U S A; 2002 Aug 06; 99(16):10813-8. PubMed ID: 12122208
    [Abstract] [Full Text] [Related]

  • 4. AAV1/2-induced overexpression of A53T-α-synuclein in the substantia nigra results in degeneration of the nigrostriatal system with Lewy-like pathology and motor impairment: a new mouse model for Parkinson's disease.
    Ip CW, Klaus LC, Karikari AA, Visanji NP, Brotchie JM, Lang AE, Volkmann J, Koprich JB.
    Acta Neuropathol Commun; 2017 Feb 01; 5(1):11. PubMed ID: 28143577
    [Abstract] [Full Text] [Related]

  • 5. Generation of a alpha-synuclein-based rat model of Parkinson's disease.
    Recchia A, Rota D, Debetto P, Peroni D, Guidolin D, Negro A, Skaper SD, Giusti P.
    Neurobiol Dis; 2008 Apr 01; 30(1):8-18. PubMed ID: 18313315
    [Abstract] [Full Text] [Related]

  • 6. Lentiviral vector delivery of parkin prevents dopaminergic degeneration in an alpha-synuclein rat model of Parkinson's disease.
    Lo Bianco C, Schneider BL, Bauer M, Sajadi A, Brice A, Iwatsubo T, Aebischer P.
    Proc Natl Acad Sci U S A; 2004 Dec 14; 101(50):17510-5. PubMed ID: 15576511
    [Abstract] [Full Text] [Related]

  • 7. Overexpression of alpha-synuclein in rat substantia nigra results in loss of dopaminergic neurons, phosphorylation of alpha-synuclein and activation of caspase-9: resemblance to pathogenetic changes in Parkinson's disease.
    Yamada M, Iwatsubo T, Mizuno Y, Mochizuki H.
    J Neurochem; 2004 Oct 14; 91(2):451-61. PubMed ID: 15447678
    [Abstract] [Full Text] [Related]

  • 8. Dopaminergic neuron loss and up-regulation of chaperone protein mRNA induced by targeted over-expression of alpha-synuclein in mouse substantia nigra.
    St Martin JL, Klucken J, Outeiro TF, Nguyen P, Keller-McGandy C, Cantuti-Castelvetri I, Grammatopoulos TN, Standaert DG, Hyman BT, McLean PJ.
    J Neurochem; 2007 Mar 14; 100(6):1449-57. PubMed ID: 17241127
    [Abstract] [Full Text] [Related]

  • 9. rAAV2/7 vector-mediated overexpression of alpha-synuclein in mouse substantia nigra induces protein aggregation and progressive dose-dependent neurodegeneration.
    Oliveras-Salvá M, Van der Perren A, Casadei N, Stroobants S, Nuber S, D'Hooge R, Van den Haute C, Baekelandt V.
    Mol Neurodegener; 2013 Nov 25; 8():44. PubMed ID: 24267638
    [Abstract] [Full Text] [Related]

  • 10. Rab1A over-expression prevents Golgi apparatus fragmentation and partially corrects motor deficits in an alpha-synuclein based rat model of Parkinson's disease.
    Coune PG, Bensadoun JC, Aebischer P, Schneider BL.
    J Parkinsons Dis; 2011 Nov 25; 1(4):373-87. PubMed ID: 23939344
    [Abstract] [Full Text] [Related]

  • 11. Reprint of: revisiting oxidative stress and mitochondrial dysfunction in the pathogenesis of Parkinson disease-resemblance to the effect of amphetamine drugs of abuse.
    Perfeito R, Cunha-Oliveira T, Rego AC.
    Free Radic Biol Med; 2013 Sep 25; 62():186-201. PubMed ID: 23743292
    [Abstract] [Full Text] [Related]

  • 12. Alpha-synuclein redistributes to neuromelanin lipid in the substantia nigra early in Parkinson's disease.
    Halliday GM, Ophof A, Broe M, Jensen PH, Kettle E, Fedorow H, Cartwright MI, Griffiths FM, Shepherd CE, Double KL.
    Brain; 2005 Nov 25; 128(Pt 11):2654-64. PubMed ID: 16000336
    [Abstract] [Full Text] [Related]

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  • 14. Dopaminergic cell loss induced by human A30P alpha-synuclein gene transfer to the rat substantia nigra.
    Klein RL, King MA, Hamby ME, Meyer EM.
    Hum Gene Ther; 2002 Mar 20; 13(5):605-12. PubMed ID: 11916484
    [Abstract] [Full Text] [Related]

  • 15. Changes in adult olfactory bulb neurogenesis in mice expressing the A30P mutant form of alpha-synuclein.
    Marxreiter F, Nuber S, Kandasamy M, Klucken J, Aigner R, Burgmayer R, Couillard-Despres S, Riess O, Winkler J, Winner B.
    Eur J Neurosci; 2009 Mar 20; 29(5):879-90. PubMed ID: 19291219
    [Abstract] [Full Text] [Related]

  • 16. Curcumin protects against A53T alpha-synuclein-induced toxicity in a PC12 inducible cell model for Parkinsonism.
    Liu Z, Yu Y, Li X, Ross CA, Smith WW.
    Pharmacol Res; 2011 May 20; 63(5):439-44. PubMed ID: 21237271
    [Abstract] [Full Text] [Related]

  • 17. A stress-enhanced model for discovery of disease-modifying gene: Ece1-suppresses the toxicity of α-synuclein A30P.
    Chen AY, Tully T.
    Neurobiol Dis; 2018 Jun 20; 114():153-163. PubMed ID: 29524599
    [Abstract] [Full Text] [Related]

  • 18. Alpha synuclein dimers and oligomers are increased in overexpressing conditions in vitro and in vivo.
    Marmolino D, Foerch P, Atienzar FA, Staelens L, Michel A, Scheller D.
    Mol Cell Neurosci; 2016 Mar 20; 71():92-101. PubMed ID: 26711807
    [Abstract] [Full Text] [Related]

  • 19. Melatonin protects against behavioral deficits, dopamine loss and oxidative stress in homocysteine model of Parkinson's disease.
    Paul R, Phukan BC, Justin Thenmozhi A, Manivasagam T, Bhattacharya P, Borah A.
    Life Sci; 2018 Jan 01; 192():238-245. PubMed ID: 29138117
    [Abstract] [Full Text] [Related]

  • 20. Haploinsufficiency for trkB and trkC receptors induces cell loss and accumulation of alpha-synuclein in the substantia nigra.
    von Bohlen und Halbach O, Minichiello L, Unsicker K.
    FASEB J; 2005 Oct 01; 19(12):1740-2. PubMed ID: 16037097
    [Abstract] [Full Text] [Related]

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