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Journal Abstract Search


406 related items for PubMed ID: 23444197

  • 21.
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  • 22. Motor dysfunction and gliosis with preserved dopaminergic markers in human alpha-synuclein A30P transgenic mice.
    Gomez-Isla T, Irizarry MC, Mariash A, Cheung B, Soto O, Schrump S, Sondel J, Kotilinek L, Day J, Schwarzschild MA, Cha JH, Newell K, Miller DW, Uéda K, Young AB, Hyman BT, Ashe KH.
    Neurobiol Aging; 2003; 24(2):245-58. PubMed ID: 12498958
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  • 26. Depressive-like phenotype induced by AAV-mediated overexpression of human α-synuclein in midbrain dopaminergic neurons.
    Caudal D, Alvarsson A, Björklund A, Svenningsson P.
    Exp Neurol; 2015 Nov; 273():243-52. PubMed ID: 26363495
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  • 29. Progressive neurodegenerative and behavioural changes induced by AAV-mediated overexpression of α-synuclein in midbrain dopamine neurons.
    Decressac M, Mattsson B, Lundblad M, Weikop P, Björklund A.
    Neurobiol Dis; 2012 Mar; 45(3):939-53. PubMed ID: 22182688
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  • 31. Familial Parkinson mutant alpha-synuclein causes dopamine neuron dysfunction in transgenic Caenorhabditis elegans.
    Kuwahara T, Koyama A, Gengyo-Ando K, Masuda M, Kowa H, Tsunoda M, Mitani S, Iwatsubo T.
    J Biol Chem; 2006 Jan 06; 281(1):334-40. PubMed ID: 16260788
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  • 32. α-synuclein aggregation reduces nigral myocyte enhancer factor-2D in idiopathic and experimental Parkinson's disease.
    Chu Y, Mickiewicz AL, Kordower JH.
    Neurobiol Dis; 2011 Jan 06; 41(1):71-82. PubMed ID: 20816781
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  • 33. Human alpha-synuclein over-expression increases intracellular reactive oxygen species levels and susceptibility to dopamine.
    Junn E, Mouradian MM.
    Neurosci Lett; 2002 Mar 08; 320(3):146-50. PubMed ID: 11852183
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  • 34. Mitochondrial associated metabolic proteins are selectively oxidized in A30P alpha-synuclein transgenic mice--a model of familial Parkinson's disease.
    Poon HF, Frasier M, Shreve N, Calabrese V, Wolozin B, Butterfield DA.
    Neurobiol Dis; 2005 Apr 08; 18(3):492-8. PubMed ID: 15755676
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  • 35. Ser129D mutant alpha-synuclein induces earlier motor dysfunction while S129A results in distinctive pathology in a rat model of Parkinson's disease.
    Febbraro F, Sahin G, Farran A, Soares S, Jensen PH, Kirik D, Romero-Ramos M.
    Neurobiol Dis; 2013 Aug 08; 56():47-58. PubMed ID: 23567651
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  • 37. Ventral tegmental area dopamine neurons are resistant to human mutant alpha-synuclein overexpression.
    Maingay M, Romero-Ramos M, Carta M, Kirik D.
    Neurobiol Dis; 2006 Sep 08; 23(3):522-32. PubMed ID: 16806952
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  • 38. Motor deficits and beta oscillations are dissociable in an alpha-synuclein model of Parkinson's disease.
    Brys I, Nunes J, Fuentes R.
    Eur J Neurosci; 2017 Aug 08; 46(3):1906-1917. PubMed ID: 28370471
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  • 39. Lack of additive role of ageing in nigrostriatal neurodegeneration triggered by α-synuclein overexpression.
    Bourdenx M, Dovero S, Engeln M, Bido S, Bastide MF, Dutheil N, Vollenweider I, Baud L, Piron C, Grouthier V, Boraud T, Porras G, Li Q, Baekelandt V, Scheller D, Michel A, Fernagut PO, Georges F, Courtine G, Bezard E, Dehay B.
    Acta Neuropathol Commun; 2015 Jul 25; 3():46. PubMed ID: 26205255
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  • 40. Aggregation of αSynuclein promotes progressive in vivo neurotoxicity in adult rat dopaminergic neurons.
    Taschenberger G, Garrido M, Tereshchenko Y, Bähr M, Zweckstetter M, Kügler S.
    Acta Neuropathol; 2012 May 25; 123(5):671-83. PubMed ID: 22167382
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