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193 related items for PubMed ID: 23503750
1. Remission of nephrotic syndrome diminishes urinary plasmin content and abolishes activation of ENaC. Andersen RF, Buhl KB, Jensen BL, Svenningsen P, Friis UG, Jespersen B, Rittig S. Pediatr Nephrol; 2013 Aug; 28(8):1227-34. PubMed ID: 23503750 [Abstract] [Full Text] [Related]
3. Association of Urinary Plasminogen-Plasmin with Edema and Epithelial Sodium Channel Activation in Patients with Nephrotic Syndrome. Chen JL, Wang L, Yao XM, Zang YJ, Wang Y, Li ZJ, Pearce D, Wang H. Am J Nephrol; 2019 Nov; 50(2):92-104. PubMed ID: 31269481 [Abstract] [Full Text] [Related]
4. Aberrant glomerular filtration of urokinase-type plasminogen activator in nephrotic syndrome leads to amiloride-sensitive plasminogen activation in urine. Stæhr M, Buhl KB, Andersen RF, Svenningsen P, Nielsen F, Hinrichs GR, Bistrup C, Jensen BL. Am J Physiol Renal Physiol; 2015 Aug 01; 309(3):F235-41. PubMed ID: 25972510 [Abstract] [Full Text] [Related]
5. Plasmin in nephrotic urine activates the epithelial sodium channel. Svenningsen P, Bistrup C, Friis UG, Bertog M, Haerteis S, Krueger B, Stubbe J, Jensen ON, Thiesson HC, Uhrenholt TR, Jespersen B, Jensen BL, Korbmacher C, Skøtt O. J Am Soc Nephrol; 2009 Feb 01; 20(2):299-310. PubMed ID: 19073825 [Abstract] [Full Text] [Related]
6. Diabetic nephropathy is associated with increased urine excretion of proteases plasmin, prostasin and urokinase and activation of amiloride-sensitive current in collecting duct cells. Andersen H, Friis UG, Hansen PB, Svenningsen P, Henriksen JE, Jensen BL. Nephrol Dial Transplant; 2015 May 01; 30(5):781-9. PubMed ID: 25609736 [Abstract] [Full Text] [Related]
8. Plasmin in urine from patients with type 2 diabetes and treatment-resistant hypertension activates ENaC in vitro. Buhl KB, Oxlund CS, Friis UG, Svenningsen P, Bistrup C, Jacobsen IA, Jensen BL. J Hypertens; 2014 Aug 01; 32(8):1672-7; discussion 1677. PubMed ID: 24805959 [Abstract] [Full Text] [Related]
9. Prostasin-dependent activation of epithelial Na+ channels by low plasmin concentrations. Svenningsen P, Uhrenholt TR, Palarasah Y, Skjødt K, Jensen BL, Skøtt O. Am J Physiol Regul Integr Comp Physiol; 2009 Dec 01; 297(6):R1733-41. PubMed ID: 19793956 [Abstract] [Full Text] [Related]
10. Over- or underfill: not all nephrotic states are created equal. Bockenhauer D. Pediatr Nephrol; 2013 Aug 01; 28(8):1153-6. PubMed ID: 23529637 [Abstract] [Full Text] [Related]
11. Urokinase-type plasminogen activator contributes to amiloride-sensitive sodium retention in nephrotic range glomerular proteinuria in mice. Hinrichs GR, Weyer K, Friis UG, Svenningsen P, Lund IK, Nielsen R, Mollet G, Antignac C, Bistrup C, Jensen BL, Birn H. Acta Physiol (Oxf); 2019 Dec 01; 227(4):e13362. PubMed ID: 31423748 [Abstract] [Full Text] [Related]
13. Nephrotic syndrome is associated with increased plasma K+ concentration, intestinal K+ losses, and attenuated urinary K+ excretion: a study in rats and humans. Ydegaard R, Svenningsen P, Bistrup C, Andersen RF, Stubbe J, Buhl KB, Marcussen N, Hinrichs GR, Iraqi H, Zamani R, Dimke H, Jensen BL. Am J Physiol Renal Physiol; 2019 Dec 01; 317(6):F1549-F1562. PubMed ID: 31566427 [Abstract] [Full Text] [Related]
14. Amiloride lowers blood pressure and attenuates urine plasminogen activation in patients with treatment-resistant hypertension. Oxlund CS, Buhl KB, Jacobsen IA, Hansen MR, Gram J, Henriksen JE, Schousboe K, Tarnow L, Jensen BL. J Am Soc Hypertens; 2014 Dec 01; 8(12):872-81. PubMed ID: 25492830 [Abstract] [Full Text] [Related]
15. Association of Plasminuria with Overhydration in Patients with CKD. Schork A, Woern M, Kalbacher H, Voelter W, Nacken R, Bertog M, Haerteis S, Korbmacher C, Heyne N, Peter A, Häring HU, Artunc F. Clin J Am Soc Nephrol; 2016 May 06; 11(5):761-769. PubMed ID: 26933188 [Abstract] [Full Text] [Related]
16. Cathepsin B increases ENaC activity leading to hypertension early in nephrotic syndrome. Larionov A, Dahlke E, Kunke M, Zanon Rodriguez L, Schiessl IM, Magnin JL, Kern U, Alli AA, Mollet G, Schilling O, Castrop H, Theilig F. J Cell Mol Med; 2019 Oct 06; 23(10):6543-6553. PubMed ID: 31368174 [Abstract] [Full Text] [Related]
17. Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome. Xiao M, Bohnert BN, Aypek H, Kretz O, Grahammer F, Aukschun U, Wörn M, Janessa A, Essigke D, Daniel C, Amann K, Huber TB, Plow EF, Birkenfeld AL, Artunc F. Acta Physiol (Oxf); 2021 Jan 06; 231(1):e13512. PubMed ID: 32455507 [Abstract] [Full Text] [Related]
18. Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice. Haerteis S, Schork A, Dörffel T, Bohnert BN, Nacken R, Wörn M, Xiao M, Essigke D, Janessa A, Schmaier AH, Feener EP, Häring HU, Bertog M, Korbmacher C, Artunc F. Acta Physiol (Oxf); 2018 Sep 06; 224(1):e13060. PubMed ID: 29489063 [Abstract] [Full Text] [Related]
19. Hydronephrosis is associated with elevated plasmin in urine in pediatric patients and rats and changes in NCC and γ-ENaC abundance in rat kidney. Zachar R, Al-Mashhadi A, Dimke H, Svenningsen P, Jensen BL, Carlström M. Am J Physiol Renal Physiol; 2018 Sep 01; 315(3):F547-F557. PubMed ID: 29767556 [Abstract] [Full Text] [Related]
20. Is urinary excretion of plasminogen associated with development of pre-eclampsia? An observational, explorative case-control study. Nielsen LH, Kronborg C, Vittinghus E, Kitlen G, Jensen BL, Knudsen UB, Ovesen PG. BMJ Open; 2019 Jun 21; 9(6):e026489. PubMed ID: 31230006 [Abstract] [Full Text] [Related] Page: [Next] [New Search]