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142 related items for PubMed ID: 23564132

  • 1. Substance P antagonists as a novel intervention for brain edema and raised intracranial pressure.
    Gabrielian L, Helps SC, Thornton E, Turner RJ, Leonard AV, Vink R.
    Acta Neurochir Suppl; 2013; 118():201-4. PubMed ID: 23564132
    [Abstract] [Full Text] [Related]

  • 2. Characterizing the role of the neuropeptide substance P in experimental subarachnoid hemorrhage.
    Barry CM, Helps SC, den Heuvel Cv, Vink R.
    Brain Res; 2011 May 10; 1389():143-51. PubMed ID: 21377453
    [Abstract] [Full Text] [Related]

  • 3. A substance P antagonist improves outcome when administered 4 h after onset of ischaemic stroke.
    Turner RJ, Helps SC, Thornton E, Vink R.
    Brain Res; 2011 Jun 01; 1393():84-90. PubMed ID: 21466790
    [Abstract] [Full Text] [Related]

  • 4. The effect of an NK1 receptor antagonist on blood spinal cord barrier permeability following balloon compression-induced spinal cord injury.
    Leonard AV, Vink R.
    Acta Neurochir Suppl; 2013 Jun 01; 118():303-6. PubMed ID: 23564154
    [Abstract] [Full Text] [Related]

  • 5. Substance P is associated with the development of brain edema and functional deficits after traumatic brain injury.
    Donkin JJ, Nimmo AJ, Cernak I, Blumbergs PC, Vink R.
    J Cereb Blood Flow Metab; 2009 Aug 01; 29(8):1388-98. PubMed ID: 19436311
    [Abstract] [Full Text] [Related]

  • 6. Inhibition of myosin light chain kinase reduces brain edema formation after traumatic brain injury.
    Luh C, Kuhlmann CR, Ackermann B, Timaru-Kast R, Luhmann HJ, Behl C, Werner C, Engelhard K, Thal SC.
    J Neurochem; 2010 Feb 01; 112(4):1015-25. PubMed ID: 19943851
    [Abstract] [Full Text] [Related]

  • 7. NK1 tachykinin receptor antagonist treatment reduces cerebral edema and intracranial pressure in an ovine model of ischemic stroke.
    Sorby-Adams AJ, Marian OC, Bilecki IM, Elms LE, Yassi N, Hood RJ, Coller JK, Stuckey SM, Kimberly WT, Farr TD, Leonard AV, Thornton E, Vink R, Turner RJ.
    J Cereb Blood Flow Metab; 2024 Oct 01; 44(10):1759-1773. PubMed ID: 38546535
    [Abstract] [Full Text] [Related]

  • 8. Mechanisms of cerebral edema in traumatic brain injury: therapeutic developments.
    Donkin JJ, Vink R.
    Curr Opin Neurol; 2010 Jun 01; 23(3):293-9. PubMed ID: 20168229
    [Abstract] [Full Text] [Related]

  • 9. Combined tissue plasminogen activator and an NK1 tachykinin receptor antagonist: an effective treatment for reperfusion injury following acute ischemic stroke in rats.
    Turner RJ, Vink R.
    Neuroscience; 2012 Sep 18; 220():1-10. PubMed ID: 22750240
    [Abstract] [Full Text] [Related]

  • 10. Treatment with the NK1 antagonist emend reduces blood brain barrier dysfunction and edema formation in an experimental model of brain tumors.
    Harford-Wright E, Lewis KM, Ghabriel MN, Vink R.
    PLoS One; 2014 Sep 18; 9(5):e97002. PubMed ID: 24818961
    [Abstract] [Full Text] [Related]

  • 11. Role of vasopressin V(1a) and V2 receptors for the development of secondary brain damage after traumatic brain injury in mice.
    Trabold R, Krieg S, Schöller K, Plesnila N.
    J Neurotrauma; 2008 Dec 18; 25(12):1459-65. PubMed ID: 19118456
    [Abstract] [Full Text] [Related]

  • 12. Substance P in traumatic brain injury.
    Donkin JJ, Turner RJ, Hassan I, Vink R.
    Prog Brain Res; 2007 Dec 18; 161():97-109. PubMed ID: 17618972
    [Abstract] [Full Text] [Related]

  • 13. Magnesium enhances the beneficial effects of NK1 antagonist administration on blood-brain barrier permeability and motor outcome after traumatic brain injury.
    Ameliorate JL, Ghabriel MN, Vink R.
    Magnes Res; 2017 Aug 01; 30(3):88-97. PubMed ID: 29256408
    [Abstract] [Full Text] [Related]

  • 14. Aquaporin-4 facilitates reabsorption of excess fluid in vasogenic brain edema.
    Papadopoulos MC, Manley GT, Krishna S, Verkman AS.
    FASEB J; 2004 Aug 01; 18(11):1291-3. PubMed ID: 15208268
    [Abstract] [Full Text] [Related]

  • 15. Neurogenic inflammation is associated with development of edema and functional deficits following traumatic brain injury in rats.
    Nimmo AJ, Cernak I, Heath DL, Hu X, Bennett CJ, Vink R.
    Neuropeptides; 2004 Feb 01; 38(1):40-7. PubMed ID: 15003715
    [Abstract] [Full Text] [Related]

  • 16. Inhibition of neurogenic inflammation as a novel treatment for ischemic stroke.
    Turner R, Vink R.
    Timely Top Med Cardiovasc Dis; 2007 Oct 01; 11():E24. PubMed ID: 18297137
    [Abstract] [Full Text] [Related]

  • 17. Intracranial pressure changes following traumatic brain injury in rats: lack of significant change in the absence of mass lesions or hypoxia.
    Gabrielian L, Willshire LW, Helps SC, van den Heuvel C, Mathias J, Vink R.
    J Neurotrauma; 2011 Oct 01; 28(10):2103-11. PubMed ID: 21657835
    [Abstract] [Full Text] [Related]

  • 18. The role of vasopressin V1A receptors in cytotoxic brain edema formation following brain injury.
    Kleindienst A, Dunbar JG, Glisson R, Marmarou A.
    Acta Neurochir (Wien); 2013 Jan 01; 155(1):151-64. PubMed ID: 23188468
    [Abstract] [Full Text] [Related]

  • 19. Targeting classical but not neurogenic inflammation reduces peritumoral oedema in secondary brain tumours.
    Lewis KM, Harford-Wright E, Vink R, Ghabriel MN.
    J Neuroimmunol; 2012 Sep 15; 250(1-2):59-65. PubMed ID: 22722013
    [Abstract] [Full Text] [Related]

  • 20. Simvastatin in traumatic brain injury: effect on brain edema mechanisms.
    Béziaud T, Ru Chen X, El Shafey N, Fréchou M, Teng F, Palmier B, Beray-Berthat V, Soustrat M, Margaill I, Plotkine M, Marchand-Leroux C, Besson VC.
    Crit Care Med; 2011 Oct 15; 39(10):2300-7. PubMed ID: 21666443
    [Abstract] [Full Text] [Related]

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