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Journal Abstract Search

383 related items for PubMed ID: 23571523

  • 1. Estrogens increase cystathionine-γ-lyase expression and decrease inflammation and oxidative stress in the myocardium of ovariectomized rats.
    Zhu X, Tang Z, Cong B, Du J, Wang C, Wang L, Ni X, Lu J.
    Menopause; 2013 Oct; 20(10):1084-91. PubMed ID: 23571523
    [Abstract] [Full Text] [Related]

  • 2. MiR-22/Sp-1 Links Estrogens With the Up-Regulation of Cystathionine γ-Lyase in Myocardium, Which Contributes to Estrogenic Cardioprotection Against Oxidative Stress.
    Wang L, Tang ZP, Zhao W, Cong BH, Lu JQ, Tang XL, Li XH, Zhu XY, Ni X.
    Endocrinology; 2015 Jun; 156(6):2124-37. PubMed ID: 25825815
    [Abstract] [Full Text] [Related]

  • 3. Exercise Increases Cystathionine-γ-lyase Expression and Decreases the Status of Oxidative Stress in Myocardium of Ovariectomized Rats.
    Tang Z, Wang Y, Zhu X, Ni X, Lu J.
    Int Heart J; 2016 Jun; 57(1):96-103. PubMed ID: 26673437
    [Abstract] [Full Text] [Related]

  • 4. Interaction between hydrogen sulfide and nitric oxide on cardiac protection in rats with metabolic syndrome.
    Rong-na L, Xiang-jun Z, Yu-han C, Ling-qiao L, Gang H.
    Zhongguo Yi Xue Ke Xue Yuan Xue Bao; 2011 Feb; 33(1):25-32. PubMed ID: 21375934
    [Abstract] [Full Text] [Related]

  • 5. The cystathionine γ-lyase/hydrogen sulfide system maintains cellular glutathione status.
    Lee ZW, Low YL, Huang S, Wang T, Deng LW.
    Biochem J; 2014 Jun 15; 460(3):425-35. PubMed ID: 24707893
    [Abstract] [Full Text] [Related]

  • 6. Dysregulation of cystathionine γ-lyase (CSE)/hydrogen sulfide pathway contributes to ox-LDL-induced inflammation in macrophage.
    Wang XH, Wang F, You SJ, Cao YJ, Cao LD, Han Q, Liu CF, Hu LF.
    Cell Signal; 2013 Nov 15; 25(11):2255-62. PubMed ID: 23872072
    [Abstract] [Full Text] [Related]

  • 7. Diallyl trisulfide protects against high glucose-induced cardiac apoptosis by stimulating the production of cystathionine gamma-lyase-derived hydrogen sulfide.
    Tsai CY, Wen SY, Shibu MA, Yang YC, Peng H, Wang B, Wei YM, Chang HY, Lee CY, Huang CY, Kuo WW.
    Int J Cardiol; 2015 Sep 15; 195():300-10. PubMed ID: 26056963
    [Abstract] [Full Text] [Related]

  • 8. Cystathionine γ-lyase promotes estrogen-stimulated uterine artery blood flow via glutathione homeostasis.
    Bok R, Guerra DD, Lorca RA, Wennersten SA, Harris PS, Rauniyar AK, Stabler SP, MacLean KN, Roede JR, Brown LD, Hurt KJ.
    Redox Biol; 2021 Apr 15; 40():101827. PubMed ID: 33485059
    [Abstract] [Full Text] [Related]

  • 9. The cystathionine γ-lyase/hydrogen sulfide pathway mediates the trimetazidine-induced protection of H9c2 cells against hypoxia/reoxygenation-induced apoptosis and oxidative stress.
    Zheng W, Liu C.
    Anatol J Cardiol; 2019 Sep 15; 22(3):102-111. PubMed ID: 31475956
    [Abstract] [Full Text] [Related]

  • 10. Metabolic changes of H2S in smokers and patients of COPD which might involve in inflammation, oxidative stress and steroid sensitivity.
    Sun Y, Wang K, Li MX, He W, Chang JR, Liao CC, Lin F, Qi YF, Wang R, Chen YH.
    Sci Rep; 2015 Oct 12; 5():14971. PubMed ID: 26455818
    [Abstract] [Full Text] [Related]

  • 11. Anti-atherogenic effect of hydrogen sulfide by over-expression of cystathionine gamma-lyase (CSE) gene.
    Cheung SH, Kwok WK, To KF, Lau JY.
    PLoS One; 2014 Oct 12; 9(11):e113038. PubMed ID: 25397776
    [Abstract] [Full Text] [Related]

  • 12. Statins upregulate cystathionine γ-lyase transcription and H2S generation via activating Akt signaling in macrophage.
    Xu Y, Du HP, Li J, Xu R, Wang YL, You SJ, Liu H, Wang F, Cao YJ, Liu CF, Hu LF.
    Pharmacol Res; 2014 Sep 12; 87():18-25. PubMed ID: 24951966
    [Abstract] [Full Text] [Related]

  • 13. Upregulation of cystathionine-γ-lyase/hydrogen sulfide pathway underlies the celecoxib counteraction of cyclosporine-induced hypertension and renal insult in rats.
    Helmy MM, Helmy MW, El-Mas MM.
    Prostaglandins Other Lipid Mediat; 2019 Apr 12; 141():1-10. PubMed ID: 30658157
    [Abstract] [Full Text] [Related]

  • 14. Silymarin preconditioning protected insulin resistant rats from liver ischemia-reperfusion injury: role of endogenous H2S.
    Younis NN, Shaheen MA, Mahmoud MF.
    J Surg Res; 2016 Aug 12; 204(2):398-409. PubMed ID: 27565076
    [Abstract] [Full Text] [Related]

  • 15. Involvement of hydrogen sulfide and homocysteine transsulfuration pathway in the progression of kidney fibrosis after ureteral obstruction.
    Jung KJ, Jang HS, Kim JI, Han SJ, Park JW, Park KM.
    Biochim Biophys Acta; 2013 Dec 12; 1832(12):1989-97. PubMed ID: 23846016
    [Abstract] [Full Text] [Related]

  • 16. Decreased endogenous production of hydrogen sulfide accelerates atherosclerosis.
    Mani S, Li H, Untereiner A, Wu L, Yang G, Austin RC, Dickhout JG, Lhoták Š, Meng QH, Wang R.
    Circulation; 2013 Jun 25; 127(25):2523-34. PubMed ID: 23704252
    [Abstract] [Full Text] [Related]

  • 17. Increased activity of osteocyte autophagy in ovariectomized rats and its correlation with oxidative stress status and bone loss.
    Yang Y, Zheng X, Li B, Jiang S, Jiang L.
    Biochem Biophys Res Commun; 2014 Aug 15; 451(1):86-92. PubMed ID: 25063028
    [Abstract] [Full Text] [Related]

  • 18. [Change in plasma H2S level and therapeutic effect of H2S supplementation in tubulointerstitial fibrosis among rats with unilateral ureteral obstruction].
    Zhao DA, Liu J, Huang Q, Han ZM.
    Zhongguo Dang Dai Er Ke Za Zhi; 2013 Oct 15; 15(10):903-8. PubMed ID: 24131846
    [Abstract] [Full Text] [Related]

  • 19. Hydrogen sulfide-producing cystathionine γ-lyase is critical in the progression of kidney fibrosis.
    Han SJ, Noh MR, Jung JM, Ishii I, Yoo J, Kim JI, Park KM.
    Free Radic Biol Med; 2017 Nov 15; 112():423-432. PubMed ID: 28842346
    [Abstract] [Full Text] [Related]

  • 20. Hydrogen sulfide regulates lung tissue-oxidized glutathione and total antioxidant capacity in hypoxic pulmonary hypertensive rats.
    Wei HL, Zhang CY, Jin HF, Tang CS, Du JB.
    Acta Pharmacol Sin; 2008 Jun 15; 29(6):670-9. PubMed ID: 18501113
    [Abstract] [Full Text] [Related]

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