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PUBMED FOR HANDHELDS

Journal Abstract Search


978 related items for PubMed ID: 23623170

  • 1. Response to antiplatelet therapy is independent of endogenous thrombin generation potential.
    Gremmel T, Panzer S, Steiner S, Seidinger D, Koppensteiner R, Pabinger I, Kopp CW, Ay C.
    Thromb Res; 2013 Jul; 132(1):e24-30. PubMed ID: 23623170
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  • 3. The influencing factors for clopidogrel-mediated platelet inhibition are assay-dependent.
    Gremmel T, Steiner S, Seidinger D, Koppensteiner R, Panzer S, Kopp CW.
    Thromb Res; 2011 Oct; 128(4):352-7. PubMed ID: 21621250
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  • 4. Comparison of Aggregometry with Flow Cytometry for the Assessment of Agonists´-Induced Platelet Reactivity in Patients on Dual Antiplatelet Therapy.
    Gremmel T, Koppensteiner R, Panzer S.
    PLoS One; 2015 Oct; 10(6):e0129666. PubMed ID: 26058047
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  • 6. A high maintenance dose increases the inhibitory response to clopidogrel in patients with high on-treatment residual platelet reactivity.
    Gremmel T, Steiner S, Seidinger D, Koppensteiner R, Panzer S, Kopp CW.
    Int J Cardiol; 2012 Oct 04; 160(2):109-13. PubMed ID: 21546102
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  • 9. The formation of monocyte-platelet aggregates is independent of on-treatment residual agonists'-inducible platelet reactivity.
    Gremmel T, Kopp CW, Seidinger D, Giurgea GA, Koppensteiner R, Steiner S, Panzer S.
    Atherosclerosis; 2009 Dec 04; 207(2):608-13. PubMed ID: 19616779
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  • 15. A comparison of the antiplatelet effects of prasugrel and high-dose clopidogrel as assessed by VASP-phosphorylation and light transmission aggregometry.
    Jakubowski JA, Payne CD, Li YG, Farid NA, Brandt JT, Small DS, Salazar DE, Winters KJ.
    Thromb Haemost; 2008 Jan 04; 99(1):215-22. PubMed ID: 18217157
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  • 17. Impact of anemia on platelet response to clopidogrel in patients undergoing percutaneous coronary stenting.
    Toma C, Zahr F, Moguilanski D, Grate S, Semaan RW, Lemieux N, Lee JS, Cortese-Hassett A, Mulukutla S, Rao SV, Marroquin OC.
    Am J Cardiol; 2012 Apr 15; 109(8):1148-53. PubMed ID: 22277895
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  • 19. Multiple electrode aggregometry and vasodilator stimulated phosphoprotein-phosphorylation assay in clinical routine for prediction of postprocedural major adverse cardiovascular events.
    Freynhofer MK, Brozovic I, Bruno V, Farhan S, Vogel B, Jakl G, Willheim M, Hübl W, Wojta J, Huber K.
    Thromb Haemost; 2011 Aug 15; 106(2):230-9. PubMed ID: 21614416
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