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312 related items for PubMed ID: 23700264
1. A study of tropomyosin's role in cardiac function and disease using thin-filament reconstituted myocardium. Bai F, Wang L, Kawai M. J Muscle Res Cell Motil; 2013 Aug; 34(3-4):295-310. PubMed ID: 23700264 [Abstract] [Full Text] [Related]
2. Using baculovirus/insect cell expressed recombinant actin to study the molecular pathogenesis of HCM caused by actin mutation A331P. Bai F, Caster HM, Rubenstein PA, Dawson JF, Kawai M. J Mol Cell Cardiol; 2014 Sep; 74():64-75. PubMed ID: 24793351 [Abstract] [Full Text] [Related]
3. The immediate effect of HCM causing actin mutants E99K and A230V on actin-Tm-myosin interaction in thin-filament reconstituted myocardium. Bai F, Caster HM, Dawson JF, Kawai M. J Mol Cell Cardiol; 2015 Feb; 79():123-32. PubMed ID: 25451174 [Abstract] [Full Text] [Related]
6. Dilated and hypertrophic cardiomyopathy mutations in troponin and alpha-tropomyosin have opposing effects on the calcium affinity of cardiac thin filaments. Robinson P, Griffiths PJ, Watkins H, Redwood CS. Circ Res; 2007 Dec 07; 101(12):1266-73. PubMed ID: 17932326 [Abstract] [Full Text] [Related]
7. Dilated cardiomyopathy mutations in thin-filament regulatory proteins reduce contractility, suppress systolic Ca2+, and activate NFAT and Akt signaling. Robinson P, Sparrow AJ, Patel S, Malinowska M, Reilly SN, Zhang YH, Casadei B, Watkins H, Redwood C. Am J Physiol Heart Circ Physiol; 2020 Aug 01; 319(2):H306-H319. PubMed ID: 32618513 [Abstract] [Full Text] [Related]
8. DCM-related tropomyosin mutants E40K/E54K over-inhibit the actomyosin interaction and lead to a decrease in the number of cycling cross-bridges. Bai F, Groth HL, Kawai M. PLoS One; 2012 Aug 01; 7(10):e47471. PubMed ID: 23077624 [Abstract] [Full Text] [Related]
18. The effect of tropomyosin on force and elementary steps of the cross-bridge cycle in reconstituted bovine myocardium. Fujita H, Lu X, Suzuki M, Ishiwata S, Kawai M. J Physiol; 2004 Apr 15; 556(Pt 2):637-49. PubMed ID: 14742733 [Abstract] [Full Text] [Related]