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PUBMED FOR HANDHELDS

Journal Abstract Search


312 related items for PubMed ID: 23700264

  • 1. A study of tropomyosin's role in cardiac function and disease using thin-filament reconstituted myocardium.
    Bai F, Wang L, Kawai M.
    J Muscle Res Cell Motil; 2013 Aug; 34(3-4):295-310. PubMed ID: 23700264
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  • 2. Using baculovirus/insect cell expressed recombinant actin to study the molecular pathogenesis of HCM caused by actin mutation A331P.
    Bai F, Caster HM, Rubenstein PA, Dawson JF, Kawai M.
    J Mol Cell Cardiol; 2014 Sep; 74():64-75. PubMed ID: 24793351
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  • 3. The immediate effect of HCM causing actin mutants E99K and A230V on actin-Tm-myosin interaction in thin-filament reconstituted myocardium.
    Bai F, Caster HM, Dawson JF, Kawai M.
    J Mol Cell Cardiol; 2015 Feb; 79():123-32. PubMed ID: 25451174
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  • 6. Dilated and hypertrophic cardiomyopathy mutations in troponin and alpha-tropomyosin have opposing effects on the calcium affinity of cardiac thin filaments.
    Robinson P, Griffiths PJ, Watkins H, Redwood CS.
    Circ Res; 2007 Dec 07; 101(12):1266-73. PubMed ID: 17932326
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  • 7. Dilated cardiomyopathy mutations in thin-filament regulatory proteins reduce contractility, suppress systolic Ca2+, and activate NFAT and Akt signaling.
    Robinson P, Sparrow AJ, Patel S, Malinowska M, Reilly SN, Zhang YH, Casadei B, Watkins H, Redwood C.
    Am J Physiol Heart Circ Physiol; 2020 Aug 01; 319(2):H306-H319. PubMed ID: 32618513
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  • 8. DCM-related tropomyosin mutants E40K/E54K over-inhibit the actomyosin interaction and lead to a decrease in the number of cycling cross-bridges.
    Bai F, Groth HL, Kawai M.
    PLoS One; 2012 Aug 01; 7(10):e47471. PubMed ID: 23077624
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  • 9. Dilated cardiomyopathy mutant tropomyosin mice develop cardiac dysfunction with significantly decreased fractional shortening and myofilament calcium sensitivity.
    Rajan S, Ahmed RP, Jagatheesan G, Petrashevskaya N, Boivin GP, Urboniene D, Arteaga GM, Wolska BM, Solaro RJ, Liggett SB, Wieczorek DF.
    Circ Res; 2007 Jul 20; 101(2):205-14. PubMed ID: 17556658
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  • 11. The cMyBP-C HCM variant L348P enhances thin filament activation through an increased shift in tropomyosin position.
    Mun JY, Kensler RW, Harris SP, Craig R.
    J Mol Cell Cardiol; 2016 Feb 20; 91():141-7. PubMed ID: 26718724
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  • 16. Familial dilated cardiomyopathy mutations uncouple troponin I phosphorylation from changes in myofibrillar Ca²⁺ sensitivity.
    Memo M, Leung MC, Ward DG, dos Remedios C, Morimoto S, Zhang L, Ravenscroft G, McNamara E, Nowak KJ, Marston SB, Messer AE.
    Cardiovasc Res; 2013 Jul 01; 99(1):65-73. PubMed ID: 23539503
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  • 18. The effect of tropomyosin on force and elementary steps of the cross-bridge cycle in reconstituted bovine myocardium.
    Fujita H, Lu X, Suzuki M, Ishiwata S, Kawai M.
    J Physiol; 2004 Apr 15; 556(Pt 2):637-49. PubMed ID: 14742733
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  • 20. Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotype.
    Mirza M, Marston S, Willott R, Ashley C, Mogensen J, McKenna W, Robinson P, Redwood C, Watkins H.
    J Biol Chem; 2005 Aug 05; 280(31):28498-506. PubMed ID: 15923195
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