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Journal Abstract Search

214 related items for PubMed ID: 23744350

  • 21. Visualization of BOK pores independent of BAX and BAK reveals a similar mechanism with differing regulation.
    Shalaby R, Diwan A, Flores-Romero H, Hertlein V, Garcia-Saez AJ.
    Cell Death Differ; 2023 Mar; 30(3):731-741. PubMed ID: 36289446
    [Abstract] [Full Text] [Related]

  • 22. Pseudomonas exotoxin A-mediated apoptosis is Bak dependent and preceded by the degradation of Mcl-1.
    Du X, Youle RJ, FitzGerald DJ, Pastan I.
    Mol Cell Biol; 2010 Jul; 30(14):3444-52. PubMed ID: 20457813
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  • 23. Inactivation of prosurvival Bcl-2 proteins activates Bax/Bak through the outer mitochondrial membrane.
    O'Neill KL, Huang K, Zhang J, Chen Y, Luo X.
    Genes Dev; 2016 Apr 15; 30(8):973-88. PubMed ID: 27056669
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  • 24. An interconnected hierarchical model of cell death regulation by the BCL-2 family.
    Chen HC, Kanai M, Inoue-Yamauchi A, Tu HC, Huang Y, Ren D, Kim H, Takeda S, Reyna DE, Chan PM, Ganesan YT, Liao CP, Gavathiotis E, Hsieh JJ, Cheng EH.
    Nat Cell Biol; 2015 Oct 15; 17(10):1270-81. PubMed ID: 26344567
    [Abstract] [Full Text] [Related]

  • 25. Bax and Bak are required for apoptosis induction by sulforaphane, a cruciferous vegetable-derived cancer chemopreventive agent.
    Choi S, Singh SV.
    Cancer Res; 2005 Mar 01; 65(5):2035-43. PubMed ID: 15753404
    [Abstract] [Full Text] [Related]

  • 26. The BH3 alpha-helical mimic BH3-M6 disrupts Bcl-X(L), Bcl-2, and MCL-1 protein-protein interactions with Bax, Bak, Bad, or Bim and induces apoptosis in a Bax- and Bim-dependent manner.
    Kazi A, Sun J, Doi K, Sung SS, Takahashi Y, Yin H, Rodriguez JM, Becerril J, Berndt N, Hamilton AD, Wang HG, Sebti SM.
    J Biol Chem; 2011 Mar 18; 286(11):9382-92. PubMed ID: 21148306
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  • 27. Predominant requirement of Bax for apoptosis in HCT116 cells is determined by Mcl-1's inhibitory effect on Bak.
    Wang C, Youle RJ.
    Oncogene; 2012 Jun 28; 31(26):3177-89. PubMed ID: 22056880
    [Abstract] [Full Text] [Related]

  • 28. Bcl-w forms complexes with Bax and Bak, and elevated ratios of Bax/Bcl-w and Bak/Bcl-w correspond to spermatogonial and spermatocyte apoptosis in the testis.
    Yan W, Samson M, Jégou B, Toppari J.
    Mol Endocrinol; 2000 May 28; 14(5):682-99. PubMed ID: 10809232
    [Abstract] [Full Text] [Related]

  • 29. The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues.
    Lindsten T, Ross AJ, King A, Zong WX, Rathmell JC, Shiels HA, Ulrich E, Waymire KG, Mahar P, Frauwirth K, Chen Y, Wei M, Eng VM, Adelman DM, Simon MC, Ma A, Golden JA, Evan G, Korsmeyer SJ, MacGregor GR, Thompson CB.
    Mol Cell; 2000 Dec 28; 6(6):1389-99. PubMed ID: 11163212
    [Abstract] [Full Text] [Related]

  • 30. Involvement of proapoptotic molecules Bax and Bak in tumor necrosis factor-related apoptosis-inducing ligand (TRAIL)-induced mitochondrial disruption and apoptosis: differential regulation of cytochrome c and Smac/DIABLO release.
    Kandasamy K, Srinivasula SM, Alnemri ES, Thompson CB, Korsmeyer SJ, Bryant JL, Srivastava RK.
    Cancer Res; 2003 Apr 01; 63(7):1712-21. PubMed ID: 12670926
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  • 34. The BCL-2 protein BAK is required for long-chain ceramide generation during apoptosis.
    Siskind LJ, Mullen TD, Romero Rosales K, Clarke CJ, Hernandez-Corbacho MJ, Edinger AL, Obeid LM.
    J Biol Chem; 2010 Apr 16; 285(16):11818-26. PubMed ID: 20172858
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  • 36. A Bax/Bak-independent mechanism of cytochrome c release.
    Mizuta T, Shimizu S, Matsuoka Y, Nakagawa T, Tsujimoto Y.
    J Biol Chem; 2007 Jun 01; 282(22):16623-30. PubMed ID: 17409097
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  • 37. In non-transformed cells Bak activates upon loss of anti-apoptotic Bcl-XL and Mcl-1 but in the absence of active BH3-only proteins.
    Senft D, Weber A, Saathoff F, Berking C, Heppt MV, Kammerbauer C, Rothenfusser S, Kellner S, Kurgyis Z, Besch R, Häcker G.
    Cell Death Dis; 2015 Nov 26; 6(11):e1996. PubMed ID: 26610208
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  • 38. Cell-specific modulation of mitochondrial respiration and metabolism by the pro-apoptotic Bcl-2 family members Bax and Bak.
    Sovilj D, Kelemen CD, Dvorakova S, Zobalova R, Raabova H, Kriska J, Hermanova Z, Knotek T, Anderova M, Klener P, Filimonenko V, Neuzil J, Andera L.
    Apoptosis; 2024 Apr 26; 29(3-4):424-438. PubMed ID: 38001340
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  • 39. Maintenance of the relative proportion of oligodendrocytes to axons even in the absence of BAX and BAK.
    Kawai K, Itoh T, Itoh A, Horiuchi M, Wakayama K, Bannerman P, Garbern JY, Pleasure D, Lindsten T.
    Eur J Neurosci; 2009 Dec 03; 30(11):2030-41. PubMed ID: 20128842
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