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PUBMED FOR HANDHELDS

Journal Abstract Search


187 related items for PubMed ID: 23760206

  • 1. The opportunistic pathogen Pseudomonas aeruginosa activates the DNA double-strand break signaling and repair pathway in infected cells.
    Elsen S, Collin-Faure V, Gidrol X, Lemercier C.
    Cell Mol Life Sci; 2013 Nov; 70(22):4385-97. PubMed ID: 23760206
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  • 2. Carcinogenic bacterial pathogen Helicobacter pylori triggers DNA double-strand breaks and a DNA damage response in its host cells.
    Toller IM, Neelsen KJ, Steger M, Hartung ML, Hottiger MO, Stucki M, Kalali B, Gerhard M, Sartori AA, Lopes M, Müller A.
    Proc Natl Acad Sci U S A; 2011 Sep 06; 108(36):14944-9. PubMed ID: 21896770
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  • 3. Wild-type p53-induced phosphatase 1 dephosphorylates histone variant gamma-H2AX and suppresses DNA double strand break repair.
    Moon SH, Lin L, Zhang X, Nguyen TA, Darlington Y, Waldman AS, Lu X, Donehower LA.
    J Biol Chem; 2010 Apr 23; 285(17):12935-47. PubMed ID: 20118229
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  • 7. A minority of foci or pan-nuclear apoptotic staining of gammaH2AX in the S phase after UV damage contain DNA double-strand breaks.
    de Feraudy S, Revet I, Bezrookove V, Feeney L, Cleaver JE.
    Proc Natl Acad Sci U S A; 2010 Apr 13; 107(15):6870-5. PubMed ID: 20351298
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  • 8. Dephosphorylation of γ-H2AX by WIP1: an important homeostatic regulatory event in DNA repair and cell cycle control.
    Moon SH, Nguyen TA, Darlington Y, Lu X, Donehower LA.
    Cell Cycle; 2010 Jun 01; 9(11):2092-6. PubMed ID: 20495376
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  • 9. 53BP1-dependent robust localized KAP-1 phosphorylation is essential for heterochromatic DNA double-strand break repair.
    Noon AT, Shibata A, Rief N, Löbrich M, Stewart GS, Jeggo PA, Goodarzi AA.
    Nat Cell Biol; 2010 Feb 01; 12(2):177-84. PubMed ID: 20081839
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  • 12. Nucleotide excision repair-dependent DNA double-strand break formation and ATM signaling activation in mammalian quiescent cells.
    Wakasugi M, Sasaki T, Matsumoto M, Nagaoka M, Inoue K, Inobe M, Horibata K, Tanaka K, Matsunaga T.
    J Biol Chem; 2014 Oct 10; 289(41):28730-7. PubMed ID: 25164823
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  • 13. ATM Localization and Heterochromatin Repair Depend on Direct Interaction of the 53BP1-BRCT2 Domain with γH2AX.
    Baldock RA, Day M, Wilkinson OJ, Cloney R, Jeggo PA, Oliver AW, Watts FZ, Pearl LH.
    Cell Rep; 2015 Dec 15; 13(10):2081-9. PubMed ID: 26628370
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  • 16. Ataxia telangiectasia mutated activation by transcription- and topoisomerase I-induced DNA double-strand breaks.
    Sordet O, Redon CE, Guirouilh-Barbat J, Smith S, Solier S, Douarre C, Conti C, Nakamura AJ, Das BB, Nicolas E, Kohn KW, Bonner WM, Pommier Y.
    EMBO Rep; 2009 Aug 15; 10(8):887-93. PubMed ID: 19557000
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  • 17. Cytolethal distending toxin (CDT) is a radiomimetic agent and induces persistent levels of DNA double-strand breaks in human fibroblasts.
    Fahrer J, Huelsenbeck J, Jaurich H, Dörsam B, Frisan T, Eich M, Roos WP, Kaina B, Fritz G.
    DNA Repair (Amst); 2014 Jun 15; 18():31-43. PubMed ID: 24680221
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  • 18. A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci.
    Riballo E, Kühne M, Rief N, Doherty A, Smith GC, Recio MJ, Reis C, Dahm K, Fricke A, Krempler A, Parker AR, Jackson SP, Gennery A, Jeggo PA, Löbrich M.
    Mol Cell; 2004 Dec 03; 16(5):715-24. PubMed ID: 15574327
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