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Journal Abstract Search

399 related items for PubMed ID: 23807240

  • 1. Mood and memory deficits in a model of Gulf War illness are linked with reduced neurogenesis, partial neuron loss, and mild inflammation in the hippocampus.
    Parihar VK, Hattiangady B, Shuai B, Shetty AK.
    Neuropsychopharmacology; 2013 Nov; 38(12):2348-62. PubMed ID: 23807240
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  • 2. Curcumin treatment leads to better cognitive and mood function in a model of Gulf War Illness with enhanced neurogenesis, and alleviation of inflammation and mitochondrial dysfunction in the hippocampus.
    Kodali M, Hattiangady B, Shetty GA, Bates A, Shuai B, Shetty AK.
    Brain Behav Immun; 2018 Mar; 69():499-514. PubMed ID: 29454881
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  • 3. Stress and combined exposure to low doses of pyridostigmine bromide, DEET, and permethrin produce neurochemical and neuropathological alterations in cerebral cortex, hippocampus, and cerebellum.
    Abdel-Rahman A, Abou-Donia S, El-Masry E, Shetty A, Abou-Donia M.
    J Toxicol Environ Health A; 2004 Jan 23; 67(2):163-92. PubMed ID: 14675905
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  • 4. FDA-approved cannabidiol [Epidiolex®] alleviates Gulf War Illness-linked cognitive and mood dysfunction, hyperalgesia, neuroinflammatory signaling, and declined neurogenesis.
    Kodali M, Madhu LN, Kolla VSV, Attaluri S, Huard C, Somayaji Y, Shuai B, Jordan C, Rao X, Shetty S, Shetty AK.
    Mil Med Res; 2024 Aug 22; 11(1):61. PubMed ID: 39169440
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  • 8. Gulf War agent exposure causes impairment of long-term memory formation and neuropathological changes in a mouse model of Gulf War Illness.
    Zakirova Z, Tweed M, Crynen G, Reed J, Abdullah L, Nissanka N, Mullan M, Mullan MJ, Mathura V, Crawford F, Ait-Ghezala G.
    PLoS One; 2015 Aug 22; 10(3):e0119579. PubMed ID: 25785457
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  • 10. Long-term changes in neuroimaging markers, cognitive function and psychiatric symptoms in an experimental model of Gulf War Illness.
    Wu X, Shetty AK, Reddy DS.
    Life Sci; 2021 Nov 15; 285():119971. PubMed ID: 34560085
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  • 13. Neurogenesis and chronic neurobehavioral outcomes are partially improved by vagus nerve stimulation in a mouse model of Gulf War illness.
    Iannucci J, Nizamutdinov D, Shapiro LA.
    Neurotoxicology; 2022 May 15; 90():205-215. PubMed ID: 35421512
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  • 16. Fingolimod mitigates memory loss in a mouse model of Gulf War Illness amid decreasing the activation of microglia, protein kinase R, and NFκB.
    Carreras I, Jung Y, Lopez-Benitez J, Tognoni CM, Dedeoglu A.
    Neurotoxicology; 2023 May 15; 96():197-206. PubMed ID: 37160207
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  • 17. Monosodium luminol reinstates redox homeostasis, improves cognition, mood and neurogenesis, and alleviates neuro- and systemic inflammation in a model of Gulf War Illness.
    Shetty AK, Attaluri S, Kodali M, Shuai B, Shetty GA, Upadhya D, Hattiangady B, Madhu LN, Upadhya R, Bates A, Rao X.
    Redox Biol; 2020 Jan 15; 28():101389. PubMed ID: 31778892
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  • 18. Corticosterone and pyridostigmine/DEET exposure attenuate peripheral cytokine expression: Supporting a dominant role for neuroinflammation in a mouse model of Gulf War Illness.
    Michalovicz LT, Locker AR, Kelly KA, Miller JV, Barnes Z, Fletcher MA, Miller DB, Klimas NG, Morris M, Lasley SM, O'Callaghan JP.
    Neurotoxicology; 2019 Jan 15; 70():26-32. PubMed ID: 30339781
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  • 19. Pyridostigmine bromide modulates topical irritant-induced cytokine release from human epidermal keratinocytes and isolated perfused porcine skin.
    Monteiro-Riviere NA, Baynes RE, Riviere JE.
    Toxicology; 2003 Feb 01; 183(1-3):15-28. PubMed ID: 12504339
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  • 20. Moderate, intermittent voluntary exercise in a model of Gulf War Illness improves cognitive and mood function with alleviation of activated microglia and astrocytes, and enhanced neurogenesis in the hippocampus.
    Kodali M, Mishra V, Hattiangady B, Attaluri S, Gonzalez JJ, Shuai B, Shetty AK.
    Brain Behav Immun; 2021 Oct 01; 97():135-149. PubMed ID: 34245811
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