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Journal Abstract Search

153 related items for PubMed ID: 23828637

  • 41. Assessment of lesion pathology in a new animal model of MS by multiparametric MRI and DTI.
    Boretius S, Escher A, Dallenga T, Wrzos C, Tammer R, Brück W, Nessler S, Frahm J, Stadelmann C.
    Neuroimage; 2012 Feb 01; 59(3):2678-88. PubMed ID: 21914485
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  • 42. Association of TAG-1 with Caspr2 is essential for the molecular organization of juxtaparanodal regions of myelinated fibers.
    Traka M, Goutebroze L, Denisenko N, Bessa M, Nifli A, Havaki S, Iwakura Y, Fukamauchi F, Watanabe K, Soliven B, Girault JA, Karagogeos D.
    J Cell Biol; 2003 Sep 15; 162(6):1161-72. PubMed ID: 12975355
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  • 43. Axonal domain disorganization in Caspr1 and Caspr2 mutant myelinated axons affects neuromuscular junction integrity, leading to muscle atrophy.
    Saifetiarova J, Liu X, Taylor AM, Li J, Bhat MA.
    J Neurosci Res; 2017 Jul 15; 95(7):1373-1390. PubMed ID: 28370195
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  • 44. Relationship between the clinical scoring and demyelination in central nervous system with total antioxidant capacity of plasma during experimental autoimmune encephalomyelitis development in mice.
    Zargari M, Allameh A, Sanati MH, Tiraihi T, Lavasani S, Emadyan O.
    Neurosci Lett; 2007 Jan 22; 412(1):24-8. PubMed ID: 17157437
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  • 48. Type I interferon receptor signalling is induced during demyelination while its function for myelin damage and repair is redundant.
    Schmidt H, Raasch J, Merkler D, Klinker F, Krauss S, Brück W, Prinz M.
    Exp Neurol; 2009 Apr 22; 216(2):306-11. PubMed ID: 19121307
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  • 52. In toxic demyelination oligodendroglial cell death occurs early and is FAS independent.
    Hesse A, Wagner M, Held J, Brück W, Salinas-Riester G, Hao Z, Waisman A, Kuhlmann T.
    Neurobiol Dis; 2010 Feb 22; 37(2):362-9. PubMed ID: 19853662
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  • 53. Distribution of oligodendrocyte loss and mitochondrial toxicity in the cuprizone-induced experimental demyelination model.
    Acs P, Selak MA, Komoly S, Kalman B.
    J Neuroimmunol; 2013 Sep 15; 262(1-2):128-31. PubMed ID: 23890807
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  • 55. Gangliosides contribute to stability of paranodal junctions and ion channel clusters in myelinated nerve fibers.
    Susuki K, Baba H, Tohyama K, Kanai K, Kuwabara S, Hirata K, Furukawa K, Furukawa K, Rasband MN, Yuki N.
    Glia; 2007 May 15; 55(7):746-57. PubMed ID: 17352383
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  • 56. Lipopolysaccharide delays demyelination and promotes oligodendrocyte precursor proliferation in the central nervous system.
    Skripuletz T, Miller E, Grote L, Gudi V, Pul R, Voss E, Skuljec J, Moharregh-Khiabani D, Trebst C, Stangel M.
    Brain Behav Immun; 2011 Nov 15; 25(8):1592-606. PubMed ID: 21635946
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  • 57. Remyelination protects axons from demyelination-associated axon degeneration.
    Irvine KA, Blakemore WF.
    Brain; 2008 Jun 15; 131(Pt 6):1464-77. PubMed ID: 18490361
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  • 58. Matrix metalloproteinases and their tissue inhibitors in cuprizone-induced demyelination and remyelination of brain white and gray matter.
    Skuljec J, Gudi V, Ulrich R, Frichert K, Yildiz O, Pul R, Voss EV, Wissel K, Baumgärtner W, Stangel M.
    J Neuropathol Exp Neurol; 2011 Sep 15; 70(9):758-69. PubMed ID: 21865884
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  • 60. The chemokine receptor CXCR2 is differentially regulated on glial cells in vivo but is not required for successful remyelination after cuprizone-induced demyelination.
    Lindner M, Trebst C, Heine S, Skripuletz T, Koutsoudaki PN, Stangel M.
    Glia; 2008 Aug 01; 56(10):1104-13. PubMed ID: 18442092
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