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Journal Abstract Search

270 related items for PubMed ID: 23840758

  • 1. The overexpression of Twinkle helicase ameliorates the progression of cardiac fibrosis and heart failure in pressure overload model in mice.
    Tanaka A, Ide T, Fujino T, Onitsuka K, Ikeda M, Takehara T, Hata Y, Ylikallio E, Tyynismaa H, Suomalainen A, Sunagawa K.
    PLoS One; 2013; 8(6):e67642. PubMed ID: 23840758
    [Abstract] [Full Text] [Related]

  • 2. Overexpression of TFAM or twinkle increases mtDNA copy number and facilitates cardioprotection associated with limited mitochondrial oxidative stress.
    Ikeda M, Ide T, Fujino T, Arai S, Saku K, Kakino T, Tyynismaa H, Yamasaki T, Yamada K, Kang D, Suomalainen A, Sunagawa K.
    PLoS One; 2015; 10(3):e0119687. PubMed ID: 25822152
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  • 3. Overexpression of mitochondrial transcription factor a ameliorates mitochondrial deficiencies and cardiac failure after myocardial infarction.
    Ikeuchi M, Matsusaka H, Kang D, Matsushima S, Ide T, Kubota T, Fujiwara T, Hamasaki N, Takeshita A, Sunagawa K, Tsutsui H.
    Circulation; 2005 Aug 02; 112(5):683-90. PubMed ID: 16043643
    [Abstract] [Full Text] [Related]

  • 4. Twinkle overexpression prevents cardiac rupture after myocardial infarction by alleviating impaired mitochondrial biogenesis.
    Inoue T, Ikeda M, Ide T, Fujino T, Matsuo Y, Arai S, Saku K, Sunagawa K.
    Am J Physiol Heart Circ Physiol; 2016 Sep 01; 311(3):H509-19. PubMed ID: 27342873
    [Abstract] [Full Text] [Related]

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  • 6. Genetic enhancement of ventricular contractility protects against pressure-overload-induced cardiac dysfunction.
    Du XJ, Fang L, Gao XM, Kiriazis H, Feng X, Hotchkin E, Finch AM, Chaulet H, Graham RM.
    J Mol Cell Cardiol; 2004 Nov 01; 37(5):979-87. PubMed ID: 15522275
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  • 7. Eicosapentaenoic acid suppresses adverse effects of C-reactive protein overexpression on pressure overload-induced cardiac remodeling.
    Nagai T, Anzai T, Mano Y, Kaneko H, Anzai A, Sugano Y, Maekawa Y, Takahashi T, Yoshikawa T, Fukuda K.
    Heart Vessels; 2013 May 01; 28(3):404-11. PubMed ID: 22875408
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  • 8. A novel mtDNA repair fusion protein attenuates maladaptive remodeling and preserves cardiac function in heart failure.
    Bradley JM, Li Z, Organ CL, Polhemus DJ, Otsuka H, Islam KN, Bhushan S, Gorodnya OM, Ruchko MV, Gillespie MN, Wilson GL, Lefer DJ.
    Am J Physiol Heart Circ Physiol; 2018 Feb 01; 314(2):H311-H321. PubMed ID: 29101177
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  • 9. Inhibition of protein phosphatase 1 by inhibitor-2 exacerbates progression of cardiac failure in a model with pressure overload.
    Grote-Wessels S, Baba HA, Boknik P, El-Armouche A, Fabritz L, Gillmann HJ, Kucerova D, Matus M, Müller FU, Neumann J, Schmitz M, Stümpel F, Theilmeier G, Wohlschlaeger J, Schmitz W, Kirchhefer U.
    Cardiovasc Res; 2008 Aug 01; 79(3):464-71. PubMed ID: 18453636
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  • 10. Dapagliflozin Attenuates Cardiac Remodeling in Mice Model of Cardiac Pressure Overload.
    Shi L, Zhu D, Wang S, Jiang A, Li F.
    Am J Hypertens; 2019 Apr 22; 32(5):452-459. PubMed ID: 30689697
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  • 12. Overexpression of mitochondrial peroxiredoxin-3 prevents left ventricular remodeling and failure after myocardial infarction in mice.
    Matsushima S, Ide T, Yamato M, Matsusaka H, Hattori F, Ikeuchi M, Kubota T, Sunagawa K, Hasegawa Y, Kurihara T, Oikawa S, Kinugawa S, Tsutsui H.
    Circulation; 2006 Apr 11; 113(14):1779-86. PubMed ID: 16585391
    [Abstract] [Full Text] [Related]

  • 13. Protein tyrosine phosphatase 1B inactivation limits aging-associated heart failure in mice.
    Besnier M, Coquerel D, Favre J, Dumesnil A, Guerrot D, Remy-Jouet I, Mulder P, Djerada Z, Tamion F, Richard V, Ouvrard-Pascaud A.
    Am J Physiol Heart Circ Physiol; 2018 Jun 01; 314(6):H1279-H1288. PubMed ID: 29569957
    [Abstract] [Full Text] [Related]

  • 14. Pressure overload-induced LV hypertrophy and dysfunction in mice are exacerbated by congenital NOS3 deficiency.
    Ichinose F, Bloch KD, Wu JC, Hataishi R, Aretz HT, Picard MH, Scherrer-Crosbie M.
    Am J Physiol Heart Circ Physiol; 2004 Mar 01; 286(3):H1070-5. PubMed ID: 14644766
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  • 15. C-reactive protein overexpression exacerbates pressure overload-induced cardiac remodeling through enhanced inflammatory response.
    Nagai T, Anzai T, Kaneko H, Mano Y, Anzai A, Maekawa Y, Takahashi T, Meguro T, Yoshikawa T, Fukuda K.
    Hypertension; 2011 Feb 01; 57(2):208-15. PubMed ID: 21220701
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  • 17. The valosin-containing protein is a novel repressor of cardiomyocyte hypertrophy induced by pressure overload.
    Zhou N, Ma B, Stoll S, Hays TT, Qiu H.
    Aging Cell; 2017 Oct 01; 16(5):1168-1179. PubMed ID: 28799247
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  • 19. Genetic and pharmacological inhibition of galectin-3 prevents cardiac remodeling by interfering with myocardial fibrogenesis.
    Yu L, Ruifrok WP, Meissner M, Bos EM, van Goor H, Sanjabi B, van der Harst P, Pitt B, Goldstein IJ, Koerts JA, van Veldhuisen DJ, Bank RA, van Gilst WH, Silljé HH, de Boer RA.
    Circ Heart Fail; 2013 Jan 01; 6(1):107-17. PubMed ID: 23230309
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