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Journal Abstract Search


292 related items for PubMed ID: 23892094

  • 1. Treatment with the cysteine precursor l-2-oxothiazolidine-4-carboxylate (OTC) implicates taurine deficiency in severity of dystropathology in mdx mice.
    Terrill JR, Boyatzis A, Grounds MD, Arthur PG.
    Int J Biochem Cell Biol; 2013 Sep; 45(9):2097-108. PubMed ID: 23892094
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  • 3. Pre-clinical evaluation of N-acetylcysteine reveals side effects in the mdx mouse model of Duchenne muscular dystrophy.
    Pinniger GJ, Terrill JR, Assan EB, Grounds MD, Arthur PG.
    J Physiol; 2017 Dec 01; 595(23):7093-7107. PubMed ID: 28887840
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  • 4. L-2-Oxothiazolidine-4-carboxylate reverses glutathione oxidation and delays fatigue of skeletal muscle in vitro.
    Ferreira LF, Gilliam LA, Reid MB.
    J Appl Physiol (1985); 2009 Jul 01; 107(1):211-6. PubMed ID: 19407260
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  • 5. N-Acetylcysteine treatment of dystrophic mdx mice results in protein thiol modifications and inhibition of exercise induced myofibre necrosis.
    Terrill JR, Radley-Crabb HG, Grounds MD, Arthur PG.
    Neuromuscul Disord; 2012 May 01; 22(5):427-34. PubMed ID: 22206641
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  • 7. Visualizing and quantifying oxidized protein thiols in tissue sections: a comparison of dystrophic mdx and normal skeletal mouse muscles.
    Iwasaki T, Terrill J, Shavlakadze T, Grounds MD, Arthur PG.
    Free Radic Biol Med; 2013 Dec 01; 65():1408-1416. PubMed ID: 24095851
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  • 9. Levels of inflammation and oxidative stress, and a role for taurine in dystropathology of the Golden Retriever Muscular Dystrophy dog model for Duchenne Muscular Dystrophy.
    Terrill JR, Duong MN, Turner R, Le Guiner C, Boyatzis A, Kettle AJ, Grounds MD, Arthur PG.
    Redox Biol; 2016 Oct 01; 9():276-286. PubMed ID: 27611888
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  • 10. l-Glutamine administration reduces oxidized glutathione and MAP kinase signaling in dystrophic muscle of mdx mice.
    Mok E, Constantin B, Favreau F, Neveux N, Magaud C, Delwail A, Hankard R.
    Pediatr Res; 2008 Mar 01; 63(3):268-73. PubMed ID: 18287965
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  • 13. Mitochondrial content is preserved throughout disease progression in the mdx mouse model of Duchenne muscular dystrophy, regardless of taurine supplementation.
    Barker RG, Wyckelsma VL, Xu H, Murphy RM.
    Am J Physiol Cell Physiol; 2018 Apr 01; 314(4):C483-C491. PubMed ID: 29351413
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  • 15. Muscle Pathology in Dystrophic Rats and Zebrafish Is Unresponsive to Taurine Treatment, Compared to the mdx Mouse Model for Duchenne Muscular Dystrophy.
    Terrill JR, Huchet C, Le Guiner C, Lafoux A, Caudal D, Tulangekar A, Bryson-Richardson RJ, Sztal TE, Grounds MD, Arthur PG.
    Metabolites; 2023 Feb 04; 13(2):. PubMed ID: 36837851
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  • 16. Comparison of N-acetylcysteine and l-2-oxothiazolidine-4-carboxylate as cysteine deliverers and glutathione precursors in human malignant melanoma transplants in mice.
    Dizdar N, Kullman A, Kågedal B.
    Cancer Chemother Pharmacol; 2000 Feb 04; 45(3):192-8. PubMed ID: 10663636
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  • 17. Oxidative stress and pathology in muscular dystrophies: focus on protein thiol oxidation and dysferlinopathies.
    Terrill JR, Radley-Crabb HG, Iwasaki T, Lemckert FA, Arthur PG, Grounds MD.
    FEBS J; 2013 Sep 04; 280(17):4149-64. PubMed ID: 23332128
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  • 18. The role of reactive oxygen species in the hearts of dystrophin-deficient mdx mice.
    Williams IA, Allen DG.
    Am J Physiol Heart Circ Physiol; 2007 Sep 04; 293(3):H1969-77. PubMed ID: 17573457
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  • 19. Effect of N-acetylcysteine plus deferoxamine on oxidative stress and inflammation in dystrophic muscle cells.
    Moraes LH, Bollineli RC, Mizobuti DS, Silveira Ldos R, Marques MJ, Minatel E.
    Redox Rep; 2015 May 04; 20(3):109-15. PubMed ID: 25361473
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  • 20. The utilization of N-acetylcysteine and 2-oxothiazolidine-4-carboxylate by rat hepatocytes is limited by their rate of uptake and conversion to cysteine.
    Banks MF, Stipanuk MH.
    J Nutr; 1994 Mar 04; 124(3):378-87. PubMed ID: 8120657
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