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Journal Abstract Search

250 related items for PubMed ID: 23896637

  • 1. The negative cell cycle regulators, p27(Kip1), p18(Ink4c), and GSK-3, play critical role in maintaining quiescence of adult human pancreatic β-cells and restrict their ability to proliferate.
    Stein J, Milewski WM, Dey A.
    Islets; 2013; 5(4):156-69. PubMed ID: 23896637
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  • 2. The expression of p18INK4 and p27kip1 cyclin-dependent kinase inhibitors is regulated differently during human B cell differentiation.
    Schrantz N, Beney GE, Auffredou MT, Bourgeade MF, Leca G, Vazquez A.
    J Immunol; 2000 Oct 15; 165(8):4346-52. PubMed ID: 11035070
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  • 5. GSK-3 inactivation or depletion promotes β-cell replication via down regulation of the CDK inhibitor, p27 (Kip1).
    Stein J, Milewski WM, Hara M, Steiner DF, Dey A.
    Islets; 2011 Oct 15; 3(1):21-34. PubMed ID: 21278490
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  • 7. Simultaneous downregulation of CDK inhibitors p18(Ink4c) and p27(Kip1) is required for MEN2A-RET-mediated mitogenesis.
    Joshi PP, Kulkarni MV, Yu BK, Smith KR, Norton DL, van Veelen W, Höppener JW, Franklin DS.
    Oncogene; 2007 Jan 25; 26(4):554-70. PubMed ID: 16953232
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  • 9. Involvement of p21(WAF1/Cip1), p27(Kip1), and p18(INK4c) in troglitazone-induced cell-cycle arrest in human hepatoma cell lines.
    Koga H, Sakisaka S, Harada M, Takagi T, Hanada S, Taniguchi E, Kawaguchi T, Sasatomi K, Kimura R, Hashimoto O, Ueno T, Yano H, Kojiro M, Sata M.
    Hepatology; 2001 May 25; 33(5):1087-97. PubMed ID: 11343236
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  • 12. p18Ink4c, but not p27Kip1, collaborates with Men1 to suppress neuroendocrine organ tumors.
    Bai F, Pei XH, Nishikawa T, Smith MD, Xiong Y.
    Mol Cell Biol; 2007 Feb 25; 27(4):1495-504. PubMed ID: 17145768
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  • 13. Cell cycle exit during terminal erythroid differentiation is associated with accumulation of p27(Kip1) and inactivation of cdk2 kinase.
    Hsieh FF, Barnett LA, Green WF, Freedman K, Matushansky I, Skoultchi AI, Kelley LL.
    Blood; 2000 Oct 15; 96(8):2746-54. PubMed ID: 11023508
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  • 15. CDK inhibitors p18(INK4c) and p27(Kip1) mediate two separate pathways to collaboratively suppress pituitary tumorigenesis.
    Franklin DS, Godfrey VL, Lee H, Kovalev GI, Schoonhoven R, Chen-Kiang S, Su L, Xiong Y.
    Genes Dev; 1998 Sep 15; 12(18):2899-911. PubMed ID: 9744866
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  • 16. Expression of p16Ink4a compensates for p18Ink4c loss in cyclin-dependent kinase 4/6-dependent tumors and tissues.
    Ramsey MR, Krishnamurthy J, Pei XH, Torrice C, Lin W, Carrasco DR, Ligon KL, Xiong Y, Sharpless NE.
    Cancer Res; 2007 May 15; 67(10):4732-41. PubMed ID: 17510401
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  • 17. Cyclin D1 overexpression induces progestin resistance in T-47D breast cancer cells despite p27(Kip1) association with cyclin E-Cdk2.
    Musgrove EA, Hunter LJ, Lee CS, Swarbrick A, Hui R, Sutherland RL.
    J Biol Chem; 2001 Dec 14; 276(50):47675-83. PubMed ID: 11590147
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  • 19. Transforming growth factor beta(1) selectively inhibits the cyclic AMP-dependent proliferation of primary thyroid epithelial cells by preventing the association of cyclin D3-cdk4 with nuclear p27(kip1).
    Depoortere F, Pirson I, Bartek J, Dumont JE, Roger PP.
    Mol Biol Cell; 2000 Mar 14; 11(3):1061-76. PubMed ID: 10712520
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  • 20. Molecular pathway for (-)-epigallocatechin-3-gallate-induced cell cycle arrest and apoptosis of human prostate carcinoma cells.
    Gupta S, Hussain T, Mukhtar H.
    Arch Biochem Biophys; 2003 Feb 01; 410(1):177-85. PubMed ID: 12559991
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