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Journal Abstract Search

438 related items for PubMed ID: 23910792

  • 1. Schizophrenia-like neurophysiological abnormalities in 22q11.2 deletion syndrome and their association to COMT and PRODH genotypes.
    Zarchi O, Carmel M, Avni C, Attias J, Frisch A, Michaelovsky E, Patya M, Green T, Weinberger R, Weizman A, Gothelf D.
    J Psychiatr Res; 2013 Nov; 47(11):1623-9. PubMed ID: 23910792
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  • 2. Association of COMT and PRODH gene variants with intelligence quotient (IQ) and executive functions in 22q11.2DS subjects.
    Carmel M, Zarchi O, Michaelovsky E, Frisch A, Patya M, Green T, Gothelf D, Weizman A.
    J Psychiatr Res; 2014 Sep; 56():28-35. PubMed ID: 24853458
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  • 3. Genetic variation in COMT and PRODH is associated with brain anatomy in patients with schizophrenia.
    Zinkstok J, Schmitz N, van Amelsvoort T, Moeton M, Baas F, Linszen D.
    Genes Brain Behav; 2008 Feb; 7(1):61-9. PubMed ID: 17504246
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  • 4. Candidate genes and the behavioral phenotype in 22q11.2 deletion syndrome.
    Prasad SE, Howley S, Murphy KC.
    Dev Disabil Res Rev; 2008 Feb; 14(1):26-34. PubMed ID: 18636634
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  • 5. PRODH rs450046 and proline x COMT Val¹⁵⁸ Met interaction effects on intelligence and startle in adults with 22q11 deletion syndrome.
    de Koning MB, van Duin ED, Boot E, Bloemen OJ, Bakker JA, Abel KM, van Amelsvoort TA.
    Psychopharmacology (Berl); 2015 Sep; 232(17):3111-22. PubMed ID: 26068888
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  • 7. Catechol-o-methyltransferase (COMT) and proline dehydrogenase (PRODH) mRNAs in the dorsolateral prefrontal cortex in schizophrenia, bipolar disorder, and major depression.
    Tunbridge E, Burnet PW, Sodhi MS, Harrison PJ.
    Synapse; 2004 Feb; 51(2):112-8. PubMed ID: 14618678
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  • 10. Startle reactivity and prepulse inhibition of the acoustic startle response are modulated by catechol-O-methyl-transferase Val(158) Met polymorphism in adults with 22q11 deletion syndrome.
    de Koning MB, Boot E, Bloemen OJ, van Duin ED, Abel KM, de Haan L, Linszen DH, van Amelsvoort TA.
    J Psychopharmacol; 2012 Dec; 26(12):1548-60. PubMed ID: 22952320
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  • 11. Biological effects of COMT haplotypes and psychosis risk in 22q11.2 deletion syndrome.
    Gothelf D, Law AJ, Frisch A, Chen J, Zarchi O, Michaelovsky E, Ren-Patterson R, Lipska BK, Carmel M, Kolachana B, Weizman A, Weinberger DR.
    Biol Psychiatry; 2014 Mar 01; 75(5):406-13. PubMed ID: 23992923
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  • 12. Association of catechol-O-methyltransferase Val(108/158) Met genetic polymorphism with schizophrenia, P50 sensory gating, and negative symptoms in a Chinese population.
    Mao Q, Tan YL, Luo XG, Tian L, Wang ZR, Tan SP, Chen S, Yang GG, An HM, Yang FD, Zhang XY.
    Psychiatry Res; 2016 Aug 30; 242():271-276. PubMed ID: 27315458
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  • 13. Further evidence for high rates of schizophrenia in 22q11.2 deletion syndrome.
    Monks S, Niarchou M, Davies AR, Walters JT, Williams N, Owen MJ, van den Bree MB, Murphy KC.
    Schizophr Res; 2014 Mar 30; 153(1-3):231-6. PubMed ID: 24534796
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  • 15. Sensory gating deficit is associated with catechol-O-methyltransferase polymorphisms in bipolar disorder.
    Ancín I, Cabranes JA, Vázquez-Álvarez B, Santos JL, Sánchez-Morla E, García-Jiménez MÁ, Fernández C, Barabash A.
    World J Biol Psychiatry; 2011 Aug 30; 12(5):376-84. PubMed ID: 21595525
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  • 19. No evidence for an effect of COMT Val158Met genotype on executive function in patients with 22q11 deletion syndrome.
    Glaser B, Debbane M, Hinard C, Morris MA, Dahoun SP, Antonarakis SE, Eliez S.
    Am J Psychiatry; 2006 Mar 30; 163(3):537-9. PubMed ID: 16513880
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