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79 related items for PubMed ID: 23933532

  • 1. The cathepsin B inhibitor, z-FA-CMK is toxic and readily induced cell death in human T lymphocytes.
    Liow KY, Chow SC.
    Toxicol Appl Pharmacol; 2013 Nov 01; 272(3):559-67. PubMed ID: 23933532
    [Abstract] [Full Text] [Related]

  • 2. The aminopeptidase inhibitor, z-L-CMK, is toxic and induces cell death in Jurkat T cells through oxidative stress.
    Yeo EH, Goh WL, Chow SC.
    Toxicol Mech Methods; 2018 Mar 01; 28(3):157-166. PubMed ID: 28849708
    [Abstract] [Full Text] [Related]

  • 3. The cathepsin B inhibitor z-FA-CMK induces cell death in leukemic T cells via oxidative stress.
    Liow KY, Chow SC.
    Naunyn Schmiedebergs Arch Pharmacol; 2018 Jan 01; 391(1):71-82. PubMed ID: 29085973
    [Abstract] [Full Text] [Related]

  • 4. Suppression of Human T Cell Proliferation Mediated by the Cathepsin B Inhibitor, z-FA-FMK Is Due to Oxidative Stress.
    Rajah T, Chow SC.
    PLoS One; 2015 Jan 01; 10(4):e0123711. PubMed ID: 25915766
    [Abstract] [Full Text] [Related]

  • 5. Functional characterization of Jurkat T cells rescued from CD95/Fas-induced apoptosis through the inhibition of caspases.
    Ko SC, Johnson VL, Chow SC.
    Biochem Biophys Res Commun; 2000 Apr 21; 270(3):1009-15. PubMed ID: 10772942
    [Abstract] [Full Text] [Related]

  • 6. Z-FA-fmk inhibits effector caspases but not initiator caspases 8 and 10, and demonstrates that novel anticancer retinoid-related molecules induce apoptosis via the intrinsic pathway.
    Lopez-Hernandez FJ, Ortiz MA, Bayon Y, Piedrafita FJ.
    Mol Cancer Ther; 2003 Mar 21; 2(3):255-63. PubMed ID: 12657720
    [Abstract] [Full Text] [Related]

  • 7. Effector caspases are dispensable for the early nuclear morphological changes during chemical-induced apoptosis.
    Johnson VL, Ko SC, Holmstrom TH, Eriksson JE, Chow SC.
    J Cell Sci; 2000 Sep 21; 113 ( Pt 17)():2941-53. PubMed ID: 10934034
    [Abstract] [Full Text] [Related]

  • 8. The broad-spectrum caspase inhibitor Boc-Asp-CMK induces cell death in human leukaemia cells.
    Frydrych I, Mlejnek P, Dolezel P, Zoumpourlis V, Krumpochova P.
    Toxicol In Vitro; 2008 Aug 21; 22(5):1356-60. PubMed ID: 18434077
    [Abstract] [Full Text] [Related]

  • 9. The inhibition of human T cell proliferation by the caspase inhibitor z-VAD-FMK is mediated through oxidative stress.
    Rajah T, Chow SC.
    Toxicol Appl Pharmacol; 2014 Jul 15; 278(2):100-6. PubMed ID: 24768707
    [Abstract] [Full Text] [Related]

  • 10. Doxorubicin treatment activates a Z-VAD-sensitive caspase, which causes deltapsim loss, caspase-9 activity, and apoptosis in Jurkat cells.
    Gamen S, Anel A, Pérez-Galán P, Lasierra P, Johnson D, Piñeiro A, Naval J.
    Exp Cell Res; 2000 Jul 10; 258(1):223-35. PubMed ID: 10912804
    [Abstract] [Full Text] [Related]

  • 11. Caspase-independent cell death induced by anti-CD2 or staurosporine in activated human peripheral T lymphocytes.
    Déas O, Dumont C, MacFarlane M, Rouleau M, Hebib C, Harper F, Hirsch F, Charpentier B, Cohen GM, Senik A.
    J Immunol; 1998 Oct 01; 161(7):3375-83. PubMed ID: 9759854
    [Abstract] [Full Text] [Related]

  • 12. The cathepsin B inhibitor, z-FA-FMK, inhibits human T cell proliferation in vitro and modulates host response to pneumococcal infection in vivo.
    Lawrence CP, Kadioglu A, Yang AL, Coward WR, Chow SC.
    J Immunol; 2006 Sep 15; 177(6):3827-36. PubMed ID: 16951345
    [Abstract] [Full Text] [Related]

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  • 14. Brefeldin A-mediated apoptosis requires the activation of caspases and is inhibited by Bcl-2.
    Guo H, Tittle TV, Allen H, Maziarz RT.
    Exp Cell Res; 1998 Nov 25; 245(1):57-68. PubMed ID: 9828101
    [Abstract] [Full Text] [Related]

  • 15. Caspase inhibition selectively reduces the apoptotic component of oxygen-glucose deprivation-induced cortical neuronal cell death.
    Gottron FJ, Ying HS, Choi DW.
    Mol Cell Neurosci; 1997 Nov 25; 9(3):159-69. PubMed ID: 9245499
    [Abstract] [Full Text] [Related]

  • 16. Evidence that inhibition of cathepsin-B contributes to the neuroprotective properties of caspase inhibitor Tyr-Val-Ala-Asp-chloromethyl ketone.
    Gray J, Haran MM, Schneider K, Vesce S, Ray AM, Owen D, White IR, Cutler P, Davis JB.
    J Biol Chem; 2001 Aug 31; 276(35):32750-5. PubMed ID: 11427531
    [Abstract] [Full Text] [Related]

  • 17. Inhibition of caspase or FADD function blocks proliferation but not MAP kinase-activation and interleukin-2-production during primary stimulation of T cells.
    Mack A, Häcker G.
    Eur J Immunol; 2002 Jul 31; 32(7):1986-92. PubMed ID: 12115619
    [Abstract] [Full Text] [Related]

  • 18. Macrophages but not smooth muscle cells undergo benzyloxycarbonyl-Val-Ala-DL-Asp(O-Methyl)-fluoromethylketone-induced nonapoptotic cell death depending on receptor-interacting protein 1 expression: implications for the stabilization of macrophage-rich atherosclerotic plaques.
    Martinet W, De Meyer GR, Timmermans JP, Herman AG, Kockx MM.
    J Pharmacol Exp Ther; 2006 Jun 31; 317(3):1356-64. PubMed ID: 16537794
    [Abstract] [Full Text] [Related]

  • 19. Detection of caspase activation in situ by fluorochrome-labeled caspase inhibitors.
    Amstad PA, Yu G, Johnson GL, Lee BW, Dhawan S, Phelps DJ.
    Biotechniques; 2001 Sep 31; 31(3):608-10, 612, 614, passim. PubMed ID: 11570504
    [Abstract] [Full Text] [Related]

  • 20. Oligomerized tumor necrosis factor-related apoptosis inducing ligand strongly induces cell death in myeloma cells, but also activates proinflammatory signaling pathways.
    Berg D, Stühmer T, Siegmund D, Müller N, Giner T, Dittrich-Breiholz O, Kracht M, Bargou R, Wajant H.
    FEBS J; 2009 Dec 31; 276(23):6912-27. PubMed ID: 19895579
    [Abstract] [Full Text] [Related]

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