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PUBMED FOR HANDHELDS

Journal Abstract Search


213 related items for PubMed ID: 23946262

  • 1. Identification of differentially expressed proteins by treatment with PUGNAc in 3T3-L1 adipocytes through analysis of ATP-binding proteome.
    Lee JE, Park JH, Moon PG, Baek MC.
    Proteomics; 2013 Oct; 13(20):2998-3012. PubMed ID: 23946262
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  • 2. PUGNAc induces protein ubiquitination in C2C12 myotube cells.
    Park JH, Lee JE, Moon PG, Baek MC.
    Cell Biochem Funct; 2015 Dec; 33(8):525-33. PubMed ID: 26531776
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  • 3. O-GlcNAc modification on IRS-1 and Akt2 by PUGNAc inhibits their phosphorylation and induces insulin resistance in rat primary adipocytes.
    Park SY, Ryu J, Lee W.
    Exp Mol Med; 2005 Jun 30; 37(3):220-9. PubMed ID: 16000877
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  • 4. Proteomics and PUGNAcity will overcome questioning of insulin resistance induction by nonselective inhibition of O-GlcNAcase.
    Dehennaut V, Lefebvre T.
    Proteomics; 2013 Oct 30; 13(20):2944-6. PubMed ID: 23983178
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  • 5. Elevated nucleocytoplasmic glycosylation by O-GlcNAc results in insulin resistance associated with defects in Akt activation in 3T3-L1 adipocytes.
    Vosseller K, Wells L, Lane MD, Hart GW.
    Proc Natl Acad Sci U S A; 2002 Apr 16; 99(8):5313-8. PubMed ID: 11959983
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  • 9. Inhibition of O-GlcNAcase using a potent and cell-permeable inhibitor does not induce insulin resistance in 3T3-L1 adipocytes.
    Macauley MS, He Y, Gloster TM, Stubbs KA, Davies GJ, Vocadlo DJ.
    Chem Biol; 2010 Sep 24; 17(9):937-48. PubMed ID: 20851343
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  • 10. PUGNAc treatment leads to an unusual accumulation of free oligosaccharides in CHO cells.
    Mehdy A, Morelle W, Rosnoblet C, Legrand D, Lefebvre T, Duvet S, Foulquier F.
    J Biochem; 2012 Apr 24; 151(4):439-46. PubMed ID: 22337894
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  • 11. Role of the O-GlcNAc modification on insulin resistance and endoplasmic reticulum stress in 3T3-L1 cells.
    Sermikli BP, Aydogdu G, Yilmaz E.
    Mol Biol Rep; 2020 Aug 24; 47(8):5927-5942. PubMed ID: 32691273
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  • 13. Elevation of global O-GlcNAc levels in 3T3-L1 adipocytes by selective inhibition of O-GlcNAcase does not induce insulin resistance.
    Macauley MS, Bubb AK, Martinez-Fleites C, Davies GJ, Vocadlo DJ.
    J Biol Chem; 2008 Dec 12; 283(50):34687-95. PubMed ID: 18842583
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  • 17. Analysis of PUGNAc and NAG-thiazoline as transition state analogues for human O-GlcNAcase: mechanistic and structural insights into inhibitor selectivity and transition state poise.
    Whitworth GE, Macauley MS, Stubbs KA, Dennis RJ, Taylor EJ, Davies GJ, Greig IR, Vocadlo DJ.
    J Am Chem Soc; 2007 Jan 24; 129(3):635-44. PubMed ID: 17227027
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  • 19. Implications of the O-GlcNAc modification in the regulation of nuclear apoptosis in T cells.
    Johnson B, Opimba M, Bernier J.
    Biochim Biophys Acta; 2014 Jan 24; 1840(1):191-8. PubMed ID: 24035784
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  • 20. Streptozotocin-induced beta-cell death is independent of its inhibition of O-GlcNAcase in pancreatic Min6 cells.
    Gao Y, Parker GJ, Hart GW.
    Arch Biochem Biophys; 2000 Nov 15; 383(2):296-302. PubMed ID: 11185566
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