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Journal Abstract Search

351 related items for PubMed ID: 23963538

  • 1. Activation of polyamine catabolic enzymes involved in diverse responses against epibrassinolide-induced apoptosis in LNCaP and DU145 prostate cancer cell lines.
    Obakan P, Arisan ED, Calcabrini A, Agostinelli E, Bolkent S, Palavan-Unsal N.
    Amino Acids; 2014 Mar; 46(3):553-64. PubMed ID: 23963538
    [Abstract] [Full Text] [Related]

  • 2. Epibrassinolide-induced apoptosis regardless of p53 expression via activating polyamine catabolic machinery, a common target for androgen sensitive and insensitive prostate cancer cells.
    Obakan P, Arisan ED, Coker-Gurkan A, Palavan-Unsal N.
    Prostate; 2014 Dec; 74(16):1622-33. PubMed ID: 25214240
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  • 3. CDK inhibitors induce mitochondria-mediated apoptosis through the activation of polyamine catabolic pathway in LNCaP, DU145 and PC3 prostate cancer cells.
    Arisan ED, Obakan P, Coker-Gurkan A, Calcabrini A, Agostinelli E, Unsal NP.
    Curr Pharm Des; 2014 Dec; 20(2):180-8. PubMed ID: 23701543
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  • 4. Metabolic and antiproliferative consequences of activated polyamine catabolism in LNCaP prostate carcinoma cells.
    Kee K, Vujcic S, Merali S, Diegelman P, Kisiel N, Powell CT, Kramer DL, Porter CW.
    J Biol Chem; 2004 Jun 25; 279(26):27050-8. PubMed ID: 15096507
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  • 7. Ornithine Decarboxylase Is Sufficient for Prostate Tumorigenesis via Androgen Receptor Signaling.
    Shukla-Dave A, Castillo-Martin M, Chen M, Lobo J, Gladoun N, Collazo-Lorduy A, Khan FM, Ponomarev V, Yi Z, Zhang W, Pandolfi PP, Hricak H, Cordon-Cardo C.
    Am J Pathol; 2016 Dec 25; 186(12):3131-3145. PubMed ID: 27770613
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  • 9. The polyamine oxidase inhibitor MDL-72,527 selectively induces apoptosis of transformed hematopoietic cells through lysosomotropic effects.
    Dai H, Kramer DL, Yang C, Murti KG, Porter CW, Cleveland JL.
    Cancer Res; 1999 Oct 01; 59(19):4944-54. PubMed ID: 10519408
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  • 10. Potentiation of apple procyanidin-triggered apoptosis by the polyamine oxidase inactivator MDL 72527 in human colon cancer-derived metastatic cells.
    Gossé F, Roussi S, Guyot S, Schoenfelder A, Mann A, Bergerat JP, Seiler N, Raul F.
    Int J Oncol; 2006 Aug 01; 29(2):423-8. PubMed ID: 16820885
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  • 13. Spermine oxidase SMO(PAOh1), Not N1-acetylpolyamine oxidase PAO, is the primary source of cytotoxic H2O2 in polyamine analogue-treated human breast cancer cell lines.
    Pledgie A, Huang Y, Hacker A, Zhang Z, Woster PM, Davidson NE, Casero RA.
    J Biol Chem; 2005 Dec 02; 280(48):39843-51. PubMed ID: 16207710
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  • 14. Curcumin mediates polyamine metabolism and sensitizes gastrointestinal cancer cells to antitumor polyamine-targeted therapies.
    Murray-Stewart T, Dunworth M, Lui Y, Giardiello FM, Woster PM, Casero RA.
    PLoS One; 2018 Dec 02; 13(8):e0202677. PubMed ID: 30138353
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  • 17. Polyamine metabolism and cancer.
    Thomas T, Thomas TJ.
    J Cell Mol Med; 2003 Dec 02; 7(2):113-26. PubMed ID: 12927050
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  • 18. The role of polyamine catabolism in polyamine analogue-induced programmed cell death.
    Ha HC, Woster PM, Yager JD, Casero RA.
    Proc Natl Acad Sci U S A; 1997 Oct 14; 94(21):11557-62. PubMed ID: 9326648
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  • 20. Purvalanol A is a strong apoptotic inducer via activating polyamine catabolic pathway in MCF-7 estrogen receptor positive breast cancer cells.
    Obakan P, Arısan ED, Özfiliz P, Çoker-Gürkan A, Palavan-Ünsal N.
    Mol Biol Rep; 2014 Jan 14; 41(1):145-54. PubMed ID: 24190492
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