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268 related items for PubMed ID: 24056861

  • 21. E-cadherin and Src associate with extradesmosomal Dsg3 and modulate desmosome assembly and adhesion.
    Rötzer V, Hartlieb E, Vielmuth F, Gliem M, Spindler V, Waschke J.
    Cell Mol Life Sci; 2015 Dec; 72(24):4885-97. PubMed ID: 26115704
    [Abstract] [Full Text] [Related]

  • 22. Desmosome signaling. Inhibition of p38MAPK prevents pemphigus vulgaris IgG-induced cytoskeleton reorganization.
    Berkowitz P, Hu P, Liu Z, Diaz LA, Enghild JJ, Chua MP, Rubenstein DS.
    J Biol Chem; 2005 Jun 24; 280(25):23778-84. PubMed ID: 15840580
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  • 23. Desmosome assembly and disassembly are membrane raft-dependent.
    Stahley SN, Saito M, Faundez V, Koval M, Mattheyses AL, Kowalczyk AP.
    PLoS One; 2014 Jun 24; 9(1):e87809. PubMed ID: 24498201
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  • 24. Role of ADAM10 and ADAM17 in the Regulation of Keratinocyte Adhesion in Pemphigus Vulgaris.
    Kugelmann D, Anders M, Sigmund AM, Egu DT, Eichkorn RA, Yazdi AS, Sárdy M, Hertl M, Didona D, Hashimoto T, Waschke J.
    Front Immunol; 2022 Jun 24; 13():884248. PubMed ID: 35844545
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  • 25. p38MAPK signaling and desmoglein-3 internalization are linked events in pemphigus acantholysis.
    Jolly PS, Berkowitz P, Bektas M, Lee HE, Chua M, Diaz LA, Rubenstein DS.
    J Biol Chem; 2010 Mar 19; 285(12):8936-41. PubMed ID: 20093368
    [Abstract] [Full Text] [Related]

  • 26. The extent of desmoglein 3 depletion in pemphigus vulgaris is dependent on Ca(2+)-induced differentiation: a role in suprabasal epidermal skin splitting?
    Spindler V, Endlich A, Hartlieb E, Vielmuth F, Schmidt E, Waschke J.
    Am J Pathol; 2011 Oct 19; 179(4):1905-16. PubMed ID: 21864491
    [Abstract] [Full Text] [Related]

  • 27. IgG-induced clustering of desmogleins 1 and 3 in skin of patients with pemphigus fits with the desmoglein nonassembly depletion hypothesis.
    Oktarina DA, van der Wier G, Diercks GF, Jonkman MF, Pas HH.
    Br J Dermatol; 2011 Sep 19; 165(3):552-62. PubMed ID: 21692763
    [Abstract] [Full Text] [Related]

  • 28. Lack of plakophilin 1 increases keratinocyte migration and reduces desmosome stability.
    South AP, Wan H, Stone MG, Dopping-Hepenstal PJ, Purkis PE, Marshall JF, Leigh IM, Eady RA, Hart IR, McGrath JA.
    J Cell Sci; 2003 Aug 15; 116(Pt 16):3303-14. PubMed ID: 12840072
    [Abstract] [Full Text] [Related]

  • 29. Anti-desmoglein 3 (Dsg3) monoclonal antibodies deplete desmosomes of Dsg3 and differ in their Dsg3-depleting activities related to pathogenicity.
    Yamamoto Y, Aoyama Y, Shu E, Tsunoda K, Amagai M, Kitajima Y.
    J Biol Chem; 2007 Jun 15; 282(24):17866-76. PubMed ID: 17428808
    [Abstract] [Full Text] [Related]

  • 30. A new ex vivo human oral mucosa model reveals that p38MAPK inhibition is not effective in preventing autoantibody-induced mucosal blistering in pemphigus.
    Egu DT, Sigmund AM, Schmidt E, Spindler V, Walter E, Waschke J.
    Br J Dermatol; 2020 Apr 15; 182(4):987-994. PubMed ID: 31218663
    [Abstract] [Full Text] [Related]

  • 31. Pathogenic relevance of IgG and IgM antibodies against desmoglein 3 in blister formation in pemphigus vulgaris.
    Tsunoda K, Ota T, Saito M, Hata T, Shimizu A, Ishiko A, Yamada T, Nakagawa T, Kowalczyk AP, Amagai M.
    Am J Pathol; 2011 Aug 15; 179(2):795-806. PubMed ID: 21718682
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  • 32. Dsg2 Upregulation as a Rescue Mechanism in Pemphigus.
    Sigmund AM, Steinert LS, Egu DT, Bayerbach FC, Waschke J, Vielmuth F.
    Front Immunol; 2020 Aug 15; 11():581370. PubMed ID: 33193387
    [Abstract] [Full Text] [Related]

  • 33. Pemphigus vulgaris antigen, a desmoglein type of cadherin, is localized within keratinocyte desmosomes.
    Kárpáti S, Amagai M, Prussick R, Cehrs K, Stanley JR.
    J Cell Biol; 1993 Jul 15; 122(2):409-15. PubMed ID: 8320263
    [Abstract] [Full Text] [Related]

  • 34. Role of Dsg1- and Dsg3-Mediated Signaling in Pemphigus Autoantibody-Induced Loss of Keratinocyte Cohesion.
    Walter E, Vielmuth F, Wanuske MT, Seifert M, Pollmann R, Eming R, Waschke J.
    Front Immunol; 2019 Jul 15; 10():1128. PubMed ID: 31178865
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  • 35. p38 MAPK activation is downstream of the loss of intercellular adhesion in pemphigus vulgaris.
    Mao X, Sano Y, Park JM, Payne AS.
    J Biol Chem; 2011 Jan 14; 286(2):1283-91. PubMed ID: 21078676
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  • 36. Plakoglobin-dependent disruption of the desmosomal plaque in pemphigus vulgaris.
    de Bruin A, Caldelari R, Williamson L, Suter MM, Hunziker T, Wyder M, Müller EJ.
    Exp Dermatol; 2007 Jun 14; 16(6):468-75. PubMed ID: 17518986
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  • 37. Pemphigus vulgaris autoimmune globulin induces Src-dependent tyrosine-phosphorylation of plakophilin 3 and its detachment from desmoglein 3.
    Cirillo N, AlShwaimi E, McCullough M, Prime SS.
    Autoimmunity; 2014 Mar 14; 47(2):134-40. PubMed ID: 24328683
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  • 38. Plakophilin 1 but not plakophilin 3 regulates desmoglein clustering.
    Fuchs M, Foresti M, Radeva MY, Kugelmann D, Keil R, Hatzfeld M, Spindler V, Waschke J, Vielmuth F.
    Cell Mol Life Sci; 2019 Sep 14; 76(17):3465-3476. PubMed ID: 30949721
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  • 39. Pemphigus: a Comprehensive Review on Pathogenesis, Clinical Presentation and Novel Therapeutic Approaches.
    Pollmann R, Schmidt T, Eming R, Hertl M.
    Clin Rev Allergy Immunol; 2018 Feb 14; 54(1):1-25. PubMed ID: 29313220
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  • 40. Role of Src and Cortactin in Pemphigus Skin Blistering.
    Kugelmann D, Rötzer V, Walter E, Egu DT, Fuchs MT, Vielmuth F, Vargas-Robles H, Schnoor M, Hertl M, Eming R, Rottner K, Schmidt A, Spindler V, Waschke J.
    Front Immunol; 2019 Feb 14; 10():626. PubMed ID: 31024527
    [Abstract] [Full Text] [Related]


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