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Journal Abstract Search


497 related items for PubMed ID: 24081881

  • 1. Activation of NADPH oxidase 4 in the endoplasmic reticulum promotes cardiomyocyte autophagy and survival during energy stress through the protein kinase RNA-activated-like endoplasmic reticulum kinase/eukaryotic initiation factor 2α/activating transcription factor 4 pathway.
    Sciarretta S, Zhai P, Shao D, Zablocki D, Nagarajan N, Terada LS, Volpe M, Sadoshima J.
    Circ Res; 2013 Nov 08; 113(11):1253-64. PubMed ID: 24081881
    [Abstract] [Full Text] [Related]

  • 2. NOX4 regulates autophagy during energy deprivation.
    Sciarretta S, Volpe M, Sadoshima J.
    Autophagy; 2014 Apr 08; 10(4):699-701. PubMed ID: 24492492
    [Abstract] [Full Text] [Related]

  • 3. Broad suppression of NADPH oxidase activity exacerbates ischemia/reperfusion injury through inadvertent downregulation of hypoxia-inducible factor-1α and upregulation of peroxisome proliferator-activated receptor-α.
    Matsushima S, Kuroda J, Ago T, Zhai P, Ikeda Y, Oka S, Fong GH, Tian R, Sadoshima J.
    Circ Res; 2013 Apr 12; 112(8):1135-49. PubMed ID: 23476056
    [Abstract] [Full Text] [Related]

  • 4. Role of NADPH oxidase in the regulation of autophagy in cardiomyocytes.
    Sciarretta S, Yee D, Ammann P, Nagarajan N, Volpe M, Frati G, Sadoshima J.
    Clin Sci (Lond); 2015 Apr 12; 128(7):387-403. PubMed ID: 25515000
    [Abstract] [Full Text] [Related]

  • 5. Silibinin induces mitochondrial NOX4-mediated endoplasmic reticulum stress response and its subsequent apoptosis.
    Kim SH, Kim KY, Yu SN, Seo YK, Chun SS, Yu HS, Ahn SC.
    BMC Cancer; 2016 Jul 12; 16():452. PubMed ID: 27405931
    [Abstract] [Full Text] [Related]

  • 6. Inhibition of Nogo-B promotes cardiac hypertrophy via endoplasmic reticulum stress.
    Li J, Wu W, Xin Y, Zhao M, Liu X.
    Biomed Pharmacother; 2018 Aug 12; 104():193-203. PubMed ID: 29772440
    [Abstract] [Full Text] [Related]

  • 7. Nox4-derived H2O2 mediates endoplasmic reticulum signaling through local Ras activation.
    Wu RF, Ma Z, Liu Z, Terada LS.
    Mol Cell Biol; 2010 Jul 12; 30(14):3553-68. PubMed ID: 20457808
    [Abstract] [Full Text] [Related]

  • 8. [Endoplasmic reticulum-mediated integrated stress response].
    Tao TQ, Liu XH.
    Sheng Li Ke Xue Jin Zhan; 2013 Aug 12; 44(4):241-6. PubMed ID: 24228513
    [Abstract] [Full Text] [Related]

  • 9. RasGRF Couples Nox4-Dependent Endoplasmic Reticulum Signaling to Ras.
    Wu RF, Liao C, Hatoum H, Fu G, Ochoa CD, Terada LS.
    Arterioscler Thromb Vasc Biol; 2017 Jan 12; 37(1):98-107. PubMed ID: 27856453
    [Abstract] [Full Text] [Related]

  • 10. Increased oxidative stress in the nucleus caused by Nox4 mediates oxidation of HDAC4 and cardiac hypertrophy.
    Matsushima S, Kuroda J, Ago T, Zhai P, Park JY, Xie LH, Tian B, Sadoshima J.
    Circ Res; 2013 Feb 15; 112(4):651-63. PubMed ID: 23271793
    [Abstract] [Full Text] [Related]

  • 11. Critical role of X-box binding protein 1 in NADPH oxidase 4-triggered cardiac hypertrophy is mediated by receptor interacting protein kinase 1.
    Chen L, Zhao M, Li J, Wang Y, Bao Q, Wu S, Deng X, Tang X, Wu W, Liu X.
    Cell Cycle; 2017 Feb 16; 16(4):348-359. PubMed ID: 27929749
    [Abstract] [Full Text] [Related]

  • 12. Critical role of Nox4-based NADPH oxidase in glucose-induced oxidative stress in the kidney: implications in type 2 diabetic nephropathy.
    Sedeek M, Callera G, Montezano A, Gutsol A, Heitz F, Szyndralewiez C, Page P, Kennedy CR, Burns KD, Touyz RM, Hébert RL.
    Am J Physiol Renal Physiol; 2010 Dec 16; 299(6):F1348-58. PubMed ID: 20630933
    [Abstract] [Full Text] [Related]

  • 13. Nox2 and Nox4 mediate tumour necrosis factor-α-induced ventricular remodelling in mice.
    Moe KT, Yin NO, Naylynn TM, Khairunnisa K, Wutyi MA, Gu Y, Atan MS, Wong MC, Koh TH, Wong P.
    J Cell Mol Med; 2011 Dec 16; 15(12):2601-13. PubMed ID: 21251215
    [Abstract] [Full Text] [Related]

  • 14. NOX2-derived reactive oxygen species are crucial for CD29-induced pro-survival signalling in cardiomyocytes.
    Rosc-Schlüter BI, Häuselmann SP, Lorenz V, Mochizuki M, Facciotti F, Pfister O, Kuster GM.
    Cardiovasc Res; 2012 Mar 01; 93(3):454-62. PubMed ID: 22198504
    [Abstract] [Full Text] [Related]

  • 15. Yin and Yang of NADPH Oxidases in Myocardial Ischemia-Reperfusion.
    Matsushima S, Sadoshima J.
    Antioxidants (Basel); 2022 May 27; 11(6):. PubMed ID: 35739967
    [Abstract] [Full Text] [Related]

  • 16. Nox4 and nox2 NADPH oxidases mediate distinct cellular redox signaling responses to agonist stimulation.
    Anilkumar N, Weber R, Zhang M, Brewer A, Shah AM.
    Arterioscler Thromb Vasc Biol; 2008 Jul 27; 28(7):1347-54. PubMed ID: 18467643
    [Abstract] [Full Text] [Related]

  • 17. Elimination of NADPH oxidase activity promotes reductive stress and sensitizes the heart to ischemic injury.
    Yu Q, Lee CF, Wang W, Karamanlidis G, Kuroda J, Matsushima S, Sadoshima J, Tian R.
    J Am Heart Assoc; 2014 Jan 27; 3(1):e000555. PubMed ID: 24470522
    [Abstract] [Full Text] [Related]

  • 18. Thioredoxin-interacting protein mediates high glucose-induced reactive oxygen species generation by mitochondria and the NADPH oxidase, Nox4, in mesangial cells.
    Shah A, Xia L, Goldberg H, Lee KW, Quaggin SE, Fantus IG.
    J Biol Chem; 2013 Mar 08; 288(10):6835-48. PubMed ID: 23329835
    [Abstract] [Full Text] [Related]

  • 19. Rheb is a critical regulator of autophagy during myocardial ischemia: pathophysiological implications in obesity and metabolic syndrome.
    Sciarretta S, Zhai P, Shao D, Maejima Y, Robbins J, Volpe M, Condorelli G, Sadoshima J.
    Circulation; 2012 Mar 06; 125(9):1134-46. PubMed ID: 22294621
    [Abstract] [Full Text] [Related]

  • 20. Vascular Nox (NADPH Oxidase) Compartmentalization, Protein Hyperoxidation, and Endoplasmic Reticulum Stress Response in Hypertension.
    Camargo LL, Harvey AP, Rios FJ, Tsiropoulou S, Da Silva RNO, Cao Z, Graham D, McMaster C, Burchmore RJ, Hartley RC, Bulleid N, Montezano AC, Touyz RM.
    Hypertension; 2018 Jul 06; 72(1):235-246. PubMed ID: 29844144
    [Abstract] [Full Text] [Related]


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