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Journal Abstract Search

211 related items for PubMed ID: 24132183

  • 1. Maternal exposure to dioxin imprints sexual immaturity of the pups through fixing the status of the reduced expression of hypothalamic gonadotropin-releasing hormone.
    Takeda T, Fujii M, Hattori Y, Yamamoto M, Shimazoe T, Ishii Y, Himeno M, Yamada H.
    Mol Pharmacol; 2014 Jan; 85(1):74-82. PubMed ID: 24132183
    [Abstract] [Full Text] [Related]

  • 2. Maternal exposure to dioxin disrupts gonadotropin production in fetal rats and imprints defects in sexual behavior.
    Takeda T, Matsumoto Y, Koga T, Mutoh J, Nishimura Y, Shimazoe T, Ishii Y, Ishida T, Yamada H.
    J Pharmacol Exp Ther; 2009 Jun; 329(3):1091-9. PubMed ID: 19276399
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  • 3. [Molecular Mechanism Whereby Maternal Exposure to Dioxin Suppresses Sexual Maturation of the Offspring after Growing Up].
    Takeda T.
    Yakugaku Zasshi; 2017 Jun; 137(11):1373-1379. PubMed ID: 29093374
    [Abstract] [Full Text] [Related]

  • 4. Dioxin-induced retardation of development through a reduction in the expression of pituitary hormones and possible involvement of an aryl hydrocarbon receptor in this defect: a comparative study using two strains of mice with different sensitivities to dioxin.
    Takeda T, Taura J, Hattori Y, Ishii Y, Yamada H.
    Toxicol Appl Pharmacol; 2014 Aug 01; 278(3):220-9. PubMed ID: 24793433
    [Abstract] [Full Text] [Related]

  • 5. Suppression of fetal testicular cytochrome P450 17 by maternal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin: a mechanism involving an initial effect on gonadotropin synthesis in the pituitary.
    Taketoh J, Mutoh J, Takeda T, Ogishima T, Takeda S, Ishii Y, Ishida T, Yamada H.
    Life Sci; 2007 Mar 13; 80(14):1259-67. PubMed ID: 17291543
    [Abstract] [Full Text] [Related]

  • 6. α-Lipoic acid potentially targets AMP-activated protein kinase and energy production in the fetal brain to ameliorate dioxin-produced attenuation in fetal steroidogenesis.
    Takeda T, Matsuo Y, Nishida K, Fujiki A, Hattori Y, Koga T, Ishii Y, Yamada H.
    J Toxicol Sci; 2017 Mar 13; 42(1):13-23. PubMed ID: 28070105
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  • 8. Dioxin silences gonadotropin expression in perinatal pups by inducing histone deacetylases: a new insight into the mechanism for the imprinting of sexual immaturity by dioxin.
    Takeda T, Fujii M, Taura J, Ishii Y, Yamada H.
    J Biol Chem; 2012 May 25; 287(22):18440-50. PubMed ID: 22493514
    [Abstract] [Full Text] [Related]

  • 9. 2,3,4,7,8-Pentachlorodibenzofuran is far less potent than 2,3,7,8-tetrachlorodibenzo-p-dioxin in disrupting the pituitary-gonad axis of the rat fetus.
    Taura J, Takeda T, Fujii M, Hattori Y, Ishii Y, Kuroki H, Tsukimori K, Uchi H, Furue M, Yamada H.
    Toxicol Appl Pharmacol; 2014 Nov 15; 281(1):48-57. PubMed ID: 25220434
    [Abstract] [Full Text] [Related]

  • 10. The aryl hydrocarbon receptor is indispensable for dioxin-induced defects in sexually-dimorphic behaviors due to the reduction in fetal steroidogenesis of the pituitary-gonadal axis in rats.
    Hattori Y, Takeda T, Nakamura A, Nishida K, Shioji Y, Fukumitsu H, Yamada H, Ishii Y.
    Biochem Pharmacol; 2018 Aug 15; 154():213-221. PubMed ID: 29753751
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  • 11. 2,3,7,8-tetrachlorodibenzo-p-dioxin potentially attenuates the gene expression of pituitary gonadotropin β-subunits in a fetal age-specific fashion: a comparative study using cultured pituitaries.
    Takeda T, Yamamoto M, Himeno M, Takechi S, Yamaguchi T, Ishida T, Ishii Y, Yamada H.
    J Toxicol Sci; 2011 Apr 15; 36(2):221-9. PubMed ID: 21467749
    [Abstract] [Full Text] [Related]

  • 12. Effects of intrauterine 2,3,7,8-tetrachlorodibenzo-p-dioxin on the development and function of the gonadotrophin releasing hormone neuronal system in the male rat.
    Clements RJ, Lawrence RC, Blank JL.
    Reprod Toxicol; 2009 Jul 15; 28(1):38-45. PubMed ID: 19490993
    [Abstract] [Full Text] [Related]

  • 13. Gestational dioxin exposure suppresses prolactin-stimulated nursing in lactating dam rats to impair development of postnatal offspring.
    Takeda T, Fujii M, Izumoto W, Hattori Y, Matsushita T, Yamada H, Ishii Y.
    Biochem Pharmacol; 2020 Aug 15; 178():114106. PubMed ID: 32569627
    [Abstract] [Full Text] [Related]

  • 14. Maternal exposure to dioxin reduces hypothalamic but not pituitary metabolome in fetal rats: a possible mechanism for a fetus-specific reduction in steroidogenesis.
    Matsumoto Y, Ishida T, Takeda T, Koga T, Fujii M, Ishii Y, Fujimura Y, Miura D, Wariishi H, Yamada H.
    J Toxicol Sci; 2010 Jun 15; 35(3):365-73. PubMed ID: 20519845
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  • 16. Prenatal testosterone and luteinizing hormone levels in male rats exposed during pregnancy to 2,3,7,8-tetrachlorodibenzo-p-dioxin and diethylstilbestrol.
    Haavisto T, Nurmela K, Pohjanvirta R, Huuskonen H, El-Gehani F, Paranko J.
    Mol Cell Endocrinol; 2001 Jun 10; 178(1-2):169-79. PubMed ID: 11403907
    [Abstract] [Full Text] [Related]

  • 17. [The gender-specific effect of maternal exposure to dioxin on fetal steroidogenesis in the adrenal gland].
    Takeda T, Hattori Y, Fujii M, Taura J, Ishi Y, Yamada H.
    Fukuoka Igaku Zasshi; 2013 Apr 10; 104(4):143-51. PubMed ID: 23858793
    [Abstract] [Full Text] [Related]

  • 18. Dioxin-induced fetal growth retardation: the role of a preceding attenuation in the circulating level of glucocorticoid.
    Hattori Y, Takeda T, Fujii M, Taura J, Ishii Y, Yamada H.
    Endocrine; 2014 Nov 10; 47(2):572-80. PubMed ID: 24723259
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  • 19. [The effect of maternal exposure to dioxin on fetal steroidogenesis in the steroidogenic organs].
    Takeda T, Taura J, Fujii M, Koga T, Ishii Y, Yamada H.
    Fukuoka Igaku Zasshi; 2011 Apr 10; 102(4):159-66. PubMed ID: 21706896
    [Abstract] [Full Text] [Related]

  • 20. Toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in the developing male Wistar(Han) rat. I: No decrease in epididymal sperm count after a single acute dose.
    Bell DR, Clode S, Fan MQ, Fernandes A, Foster PM, Jiang T, Loizou G, MacNicoll A, Miller BG, Rose M, Tran L, White S.
    Toxicol Sci; 2007 Sep 10; 99(1):214-23. PubMed ID: 17545212
    [Abstract] [Full Text] [Related]

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