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Journal Abstract Search

959 related items for PubMed ID: 24148808

  • 1. Endocannabinoids decrease neuropathic pain-related behavior in mice through the activation of one or both peripheral CB₁ and CB₂ receptors.
    Desroches J, Charron S, Bouchard JF, Beaulieu P.
    Neuropharmacology; 2014 Feb; 77():441-52. PubMed ID: 24148808
    [Abstract] [Full Text] [Related]

  • 2. Alterations in endocannabinoid tone following chemotherapy-induced peripheral neuropathy: effects of endocannabinoid deactivation inhibitors targeting fatty-acid amide hydrolase and monoacylglycerol lipase in comparison to reference analgesics following cisplatin treatment.
    Guindon J, Lai Y, Takacs SM, Bradshaw HB, Hohmann AG.
    Pharmacol Res; 2013 Jan; 67(1):94-109. PubMed ID: 23127915
    [Abstract] [Full Text] [Related]

  • 3. Peripheral antinociceptive effects of inhibitors of monoacylglycerol lipase in a rat model of inflammatory pain.
    Guindon J, Guijarro A, Piomelli D, Hohmann AG.
    Br J Pharmacol; 2011 Aug; 163(7):1464-78. PubMed ID: 21198549
    [Abstract] [Full Text] [Related]

  • 4. The antinociceptive effects of intraplantar injections of 2-arachidonoyl glycerol are mediated by cannabinoid CB2 receptors.
    Guindon J, Desroches J, Beaulieu P.
    Br J Pharmacol; 2007 Mar; 150(6):693-701. PubMed ID: 17179944
    [Abstract] [Full Text] [Related]

  • 5. JZL184 is anti-hyperalgesic in a murine model of cisplatin-induced peripheral neuropathy.
    Khasabova IA, Yao X, Paz J, Lewandowski CT, Lindberg AE, Coicou L, Burlakova N, Simone DA, Seybold VS.
    Pharmacol Res; 2014 Dec; 90():67-75. PubMed ID: 25304184
    [Abstract] [Full Text] [Related]

  • 6. Peripheral deficiency and antiallodynic effects of 2-arachidonoyl glycerol in a mouse model of paclitaxel-induced neuropathic pain.
    Thomas A, Okine BN, Finn DP, Masocha W.
    Biomed Pharmacother; 2020 Sep; 129():110456. PubMed ID: 32603895
    [Abstract] [Full Text] [Related]

  • 7. Modulation of the anti-nociceptive effects of 2-arachidonoyl glycerol by peripherally administered FAAH and MGL inhibitors in a neuropathic pain model.
    Desroches J, Guindon J, Lambert C, Beaulieu P.
    Br J Pharmacol; 2008 Nov; 155(6):913-24. PubMed ID: 18695638
    [Abstract] [Full Text] [Related]

  • 8. Inhibitors of monoacylglycerol lipase, fatty-acid amide hydrolase and endocannabinoid transport differentially suppress capsaicin-induced behavioral sensitization through peripheral endocannabinoid mechanisms.
    Spradley JM, Guindon J, Hohmann AG.
    Pharmacol Res; 2010 Sep; 62(3):249-58. PubMed ID: 20416378
    [Abstract] [Full Text] [Related]

  • 9. Spinal anandamide produces analgesia in neuropathic rats: possible CB(1)- and TRPV1-mediated mechanisms.
    Starowicz K, Makuch W, Osikowicz M, Piscitelli F, Petrosino S, Di Marzo V, Przewlocka B.
    Neuropharmacology; 2012 Mar; 62(4):1746-55. PubMed ID: 22178705
    [Abstract] [Full Text] [Related]

  • 10. Inhibition of FAAH reduces nitroglycerin-induced migraine-like pain and trigeminal neuronal hyperactivity in mice.
    Nozaki C, Markert A, Zimmer A.
    Eur Neuropsychopharmacol; 2015 Aug; 25(8):1388-96. PubMed ID: 25910421
    [Abstract] [Full Text] [Related]

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  • 12. The endocannabinoid hydrolysis inhibitor SA-57: Intrinsic antinociceptive effects, augmented morphine-induced antinociception, and attenuated heroin seeking behavior in mice.
    Wilkerson JL, Ghosh S, Mustafa M, Abdullah RA, Niphakis MJ, Cabrera R, Maldonado RL, Cravatt BF, Lichtman AH.
    Neuropharmacology; 2017 Mar 01; 114():156-167. PubMed ID: 27890602
    [Abstract] [Full Text] [Related]

  • 13. Blockade of endocannabinoid-degrading enzymes attenuates neuropathic pain.
    Kinsey SG, Long JZ, O'Neal ST, Abdullah RA, Poklis JL, Boger DL, Cravatt BF, Lichtman AH.
    J Pharmacol Exp Ther; 2009 Sep 01; 330(3):902-10. PubMed ID: 19502530
    [Abstract] [Full Text] [Related]

  • 14. The multiplicity of spinal AA-5-HT anti-nociceptive action in a rat model of neuropathic pain.
    Malek N, Kostrzewa M, Makuch W, Pajak A, Kucharczyk M, Piscitelli F, Przewlocka B, Di Marzo V, Starowicz K.
    Pharmacol Res; 2016 Sep 01; 111():251-263. PubMed ID: 27326920
    [Abstract] [Full Text] [Related]

  • 15. Cannabinoid CB1 Discrimination: Effects of Endocannabinoids and Catabolic Enzyme Inhibitors.
    Leonard MZ, Alapafuja SO, Ji L, Shukla VG, Liu Y, Nikas SP, Makriyannis A, Bergman J, Kangas BD.
    J Pharmacol Exp Ther; 2017 Dec 01; 363(3):314-323. PubMed ID: 28947487
    [Abstract] [Full Text] [Related]

  • 16. Brain-Permeant and -Impermeant Inhibitors of Fatty Acid Amide Hydrolase Synergize with the Opioid Analgesic Morphine to Suppress Chemotherapy-Induced Neuropathic Nociception Without Enhancing Effects of Morphine on Gastrointestinal Transit.
    Slivicki RA, Saberi SA, Iyer V, Vemuri VK, Makriyannis A, Hohmann AG.
    J Pharmacol Exp Ther; 2018 Dec 01; 367(3):551-563. PubMed ID: 30275151
    [Abstract] [Full Text] [Related]

  • 17. Cannabinoid type-1 receptor reduces pain and neurotoxicity produced by chemotherapy.
    Khasabova IA, Khasabov S, Paz J, Harding-Rose C, Simone DA, Seybold VS.
    J Neurosci; 2012 May 16; 32(20):7091-101. PubMed ID: 22593077
    [Abstract] [Full Text] [Related]

  • 18. Attenuation of persistent pain-related behavior by fatty acid amide hydrolase (FAAH) inhibitors in a rat model of HIV sensory neuropathy.
    Nasirinezhad F, Jergova S, Pearson JP, Sagen J.
    Neuropharmacology; 2015 Aug 16; 95():100-9. PubMed ID: 25486617
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