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Journal Abstract Search

240 related items for PubMed ID: 24154627

  • 21. An Early Neutrophil Recruitment into the Infectious Site Is Critical for Bacterial Lipoprotein Tolerance-Afforded Protection against Microbial Sepsis.
    Chen W, Zhao S, Ita M, Li Y, Ji J, Jiang Y, Redmond HP, Wang JH, Liu J.
    J Immunol; 2020 Jan 15; 204(2):408-417. PubMed ID: 31801813
    [Abstract] [Full Text] [Related]

  • 22. Toll-like receptor 4 is essential to preserving cardiac function and survival in low-grade polymicrobial sepsis.
    Zhang M, Zou L, Feng Y, Chen YJ, Zhou Q, Ichinose F, Chao W.
    Anesthesiology; 2014 Dec 15; 121(6):1270-80. PubMed ID: 24937074
    [Abstract] [Full Text] [Related]

  • 23. Importance of the Complement Alternative Pathway in Serum Chemotactic Activity During Sepsis.
    Xu G, Feng Y, Li D, Zhou Q, Chao W, Zou L.
    Shock; 2018 Oct 15; 50(4):435-441. PubMed ID: 29040213
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  • 24. Toll-like receptor 3 plays a central role in cardiac dysfunction during polymicrobial sepsis.
    Gao M, Ha T, Zhang X, Liu L, Wang X, Kelley J, Singh K, Kao R, Gao X, Williams D, Li C.
    Crit Care Med; 2012 Aug 15; 40(8):2390-9. PubMed ID: 22635047
    [Abstract] [Full Text] [Related]

  • 25. Complement expression in retinal pigment epithelial cells is modulated by activated macrophages.
    Luo C, Zhao J, Madden A, Chen M, Xu H.
    Exp Eye Res; 2013 Jul 15; 112():93-101. PubMed ID: 23644095
    [Abstract] [Full Text] [Related]

  • 26. IL-12, but not IL-18, is critical to neutrophil activation and resistance to polymicrobial sepsis induced by cecal ligation and puncture.
    Moreno SE, Alves-Filho JC, Alfaya TM, da Silva JS, Ferreira SH, Liew FY.
    J Immunol; 2006 Sep 01; 177(5):3218-24. PubMed ID: 16920961
    [Abstract] [Full Text] [Related]

  • 27. Lipopolysaccharide clearance, bacterial clearance, and systemic inflammatory responses are regulated by cell type-specific functions of TLR4 during sepsis.
    Deng M, Scott MJ, Loughran P, Gibson G, Sodhi C, Watkins S, Hackam D, Billiar TR.
    J Immunol; 2013 May 15; 190(10):5152-60. PubMed ID: 23562812
    [Abstract] [Full Text] [Related]

  • 28. Crucial role of TNF receptors 1 and 2 in the control of polymicrobial sepsis.
    Secher T, Vasseur V, Poisson DM, Mitchell JA, Cunha FQ, Alves-Filho JC, Ryffel B.
    J Immunol; 2009 Jun 15; 182(12):7855-64. PubMed ID: 19494310
    [Abstract] [Full Text] [Related]

  • 29. Cutting edge: myeloid differentiation factor 88 deficiency improves resistance against sepsis caused by polymicrobial infection.
    Weighardt H, Kaiser-Moore S, Vabulas RM, Kirschning CJ, Wagner H, Holzmann B.
    J Immunol; 2002 Sep 15; 169(6):2823-7. PubMed ID: 12218091
    [Abstract] [Full Text] [Related]

  • 30. G-protein-coupled receptor kinase-5 mediates inflammation but does not regulate cellular infiltration or bacterial load in a polymicrobial sepsis model in mice.
    Packiriswamy N, Lee T, Raghavendra PB, Durairaj H, Wang H, Parameswaran N.
    J Innate Immun; 2013 Sep 15; 5(4):401-13. PubMed ID: 23485819
    [Abstract] [Full Text] [Related]

  • 31. TLR3-Dependent Activation of TLR2 Endogenous Ligands via the MyD88 Signaling Pathway Augments the Innate Immune Response.
    Teixeira HS, Zhao J, Kazmierski E, Kinane DF, Benakanakere MR.
    Cells; 2020 Aug 17; 9(8):. PubMed ID: 32824595
    [Abstract] [Full Text] [Related]

  • 32. NADPH oxidase-dependent reactive oxygen species mediate amplified TLR4 signaling and sepsis-induced mortality in Nrf2-deficient mice.
    Kong X, Thimmulappa R, Kombairaju P, Biswal S.
    J Immunol; 2010 Jul 01; 185(1):569-77. PubMed ID: 20511556
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  • 34. CRTH2 is a critical regulator of neutrophil migration and resistance to polymicrobial sepsis.
    Ishii M, Asano K, Namkoong H, Tasaka S, Mizoguchi K, Asami T, Kamata H, Kimizuka Y, Fujiwara H, Funatsu Y, Kagawa S, Miyata J, Ishii K, Nakamura M, Hirai H, Nagata K, Kunkel SL, Hasegawa N, Betsuyaku T.
    J Immunol; 2012 Jun 01; 188(11):5655-64. PubMed ID: 22544936
    [Abstract] [Full Text] [Related]

  • 35. Improved host defense against septic peritonitis in mice lacking MyD88 and TRIF is linked to a normal interferon response.
    Reim D, Rossmann-Bloeck T, Jusek G, Prazeres da Costa O, Holzmann B.
    J Leukoc Biol; 2011 Sep 01; 90(3):613-20. PubMed ID: 21628330
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  • 37. Macrophage-activating lipopeptide-2 exerts protective effects in a murine sepsis model.
    Zeckey C, Tschernig T, Hildebrand F, Frink M, Frömke C, Dorsch M, Krettek C, Barkhausen T.
    Shock; 2010 Jun 01; 33(6):614-9. PubMed ID: 19940812
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