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2. Mutations in cell division cycle genes CDC36 and CDC39 activate the Saccharomyces cerevisiae mating pheromone response pathway. de Barros Lopes M, Ho JY, Reed SI. Mol Cell Biol; 1990 Jun; 10(6):2966-72. PubMed ID: 2111445 [Abstract] [Full Text] [Related]
3. Mating-defective ste mutations are suppressed by cell division cycle start mutations in Saccharomyces cerevisiae. Shuster JR. Mol Cell Biol; 1982 Sep; 2(9):1052-63. PubMed ID: 6757719 [Abstract] [Full Text] [Related]
4. CDC36 and CDC39 are negative elements in the signal transduction pathway of yeast. Neiman AM, Chang F, Komachi K, Herskowitz I. Cell Regul; 1990 Apr; 1(5):391-401. PubMed ID: 2099190 [Abstract] [Full Text] [Related]
6. Differential transmission of G1 cell cycle arrest and mating signals by Saccharomyces cerevisiae Ste5 mutants in the pheromone pathway. Choi YJ, Kim SH, Park KS, Choi KY. Biochem Cell Biol; 1999 Apr; 77(5):459-68. PubMed ID: 10593609 [Abstract] [Full Text] [Related]
7. "Start" mutants of Saccharomyces cerevisiae are suppressed in carbon catabolite-derepressing medium. Shuster JR. J Bacteriol; 1982 Aug; 151(2):1059-61. PubMed ID: 7047491 [Abstract] [Full Text] [Related]
8. Isolation and transcriptional characterization of three genes which function at start, the controlling event of the Saccharomyces cerevisiae cell division cycle: CDC36, CDC37, and CDC39. Breter HJ, Ferguson J, Peterson TA, Reed SI. Mol Cell Biol; 1983 May; 3(5):881-91. PubMed ID: 6346060 [Abstract] [Full Text] [Related]
14. The yeast MOT2 gene encodes a putative zinc finger protein that serves as a global negative regulator affecting expression of several categories of genes, including mating-pheromone-responsive genes. Irie K, Yamaguchi K, Kawase K, Matsumoto K. Mol Cell Biol; 1994 May; 14(5):3150-7. PubMed ID: 8164670 [Abstract] [Full Text] [Related]
17. Isolation and characterization of Saccharomyces cerevisiae mutants supersensitive to G1 arrest by the mating hormone a-factor. Steden M, Betz R, Duntze W. Mol Gen Genet; 1989 Nov; 219(3):439-44. PubMed ID: 2695825 [Abstract] [Full Text] [Related]