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Journal Abstract Search

361 related items for PubMed ID: 24227784

  • 21. Experimental autoimmune encephalomyelitis (EAE) induced by antigen pulsed dendritic cells in the C57BL/6 mouse: influence of injection route.
    Aghdami N, Gharibdoost F, Moazzeni SM.
    Exp Anim; 2008 Jan; 57(1):45-55. PubMed ID: 18256518
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  • 23. Regulatory B cells (B10 cells) and regulatory T cells have independent roles in controlling experimental autoimmune encephalomyelitis initiation and late-phase immunopathogenesis.
    Matsushita T, Horikawa M, Iwata Y, Tedder TF.
    J Immunol; 2010 Aug 15; 185(4):2240-52. PubMed ID: 20624940
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  • 24. Smek1 deficiency exacerbates experimental autoimmune encephalomyelitis by activating proinflammatory microglia and suppressing the IDO1-AhR pathway.
    Duan RN, Yang CL, Du T, Liu A, Wang AR, Sun WJ, Li X, Li JX, Yan CZ, Liu QJ.
    J Neuroinflammation; 2021 Jun 28; 18(1):145. PubMed ID: 34183017
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  • 27. Experimental Autoimmune Encephalomyelitis.
    Ballerini C.
    Methods Mol Biol; 2021 Jun 28; 2285():375-384. PubMed ID: 33928565
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  • 28. Pathologic role and temporal appearance of newly emerging autoepitopes in relapsing experimental autoimmune encephalomyelitis.
    Vanderlugt CL, Neville KL, Nikcevich KM, Eagar TN, Bluestone JA, Miller SD.
    J Immunol; 2000 Jan 15; 164(2):670-8. PubMed ID: 10623809
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  • 30. Relapsing and remitting experimental allergic encephalomyelitis: a focused response to the encephalitogenic peptide rather than epitope spread.
    Takács K, Chandler P, Altmann DM.
    Eur J Immunol; 1997 Nov 15; 27(11):2927-34. PubMed ID: 9394820
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  • 31. LFA-1 Controls Th1 and Th17 Motility Behavior in the Inflamed Central Nervous System.
    Dusi S, Angiari S, Pietronigro EC, Lopez N, Angelini G, Zenaro E, Della Bianca V, Tosadori G, Paris F, Amoruso A, Carlucci T, Constantin G, Rossi B.
    Front Immunol; 2019 Nov 15; 10():2436. PubMed ID: 31681316
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  • 33. Mannan-conjugated myelin peptides prime non-pathogenic Th1 and Th17 cells and ameliorate experimental autoimmune encephalomyelitis.
    Tseveleki V, Tselios T, Kanistras I, Koutsoni O, Karamita M, Vamvakas SS, Apostolopoulos V, Dotsika E, Matsoukas J, Lassmann H, Probert L.
    Exp Neurol; 2015 May 15; 267():254-67. PubMed ID: 25447934
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  • 34. Early IFN-γ production together with decreased expression of TLR3 and TLR9 characterizes EAE development conditional on the presence of myelin.
    Evangelista MG, Castro SB, Alves CC, Dias AT, Souza VW, Reis LB, Silva LC, Castañon MC, Farias RE, Juliano MA, Ferreira AP.
    Autoimmunity; 2016 Jun 15; 49(4):258-67. PubMed ID: 26911613
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  • 36. CD11c+CD11b+ dendritic cells play an important role in intravenous tolerance and the suppression of experimental autoimmune encephalomyelitis.
    Li H, Zhang GX, Chen Y, Xu H, Fitzgerald DC, Zhao Z, Rostami A.
    J Immunol; 2008 Aug 15; 181(4):2483-93. PubMed ID: 18684939
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  • 38. Infection with Mycobacterium bovis BCG diverts traffic of myelin oligodendroglial glycoprotein autoantigen-specific T cells away from the central nervous system and ameliorates experimental autoimmune encephalomyelitis.
    Sewell DL, Reinke EK, Co DO, Hogan LH, Fritz RB, Sandor M, Fabry Z.
    Clin Diagn Lab Immunol; 2003 Jul 15; 10(4):564-72. PubMed ID: 12853387
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  • 39. Mapping the accumulation of co-infiltrating CNS dendritic cells and encephalitogenic T cells during EAE.
    Clarkson BD, Walker A, Harris M, Rayasam A, Sandor M, Fabry Z.
    J Neuroimmunol; 2014 Dec 15; 277(1-2):39-49. PubMed ID: 25288303
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