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Journal Abstract Search

114 related items for PubMed ID: 24295730

  • 1. Ontogenetic manganese exposure with perinatal 6-OHDA lesioning alters behavioral responses of rats to dopamine D₁ and D2 agonist treatments.
    Szkilnik R, Brus R, Malinowska-Borowska J, Nowak D, Waliczek M, Kostrzewa RM, Nowak P.
    Environ Toxicol Pharmacol; 2014 Jan; 37(1):28-36. PubMed ID: 24295730
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  • 2. Ontogenetic exposure of rats to pre- and post-natal manganese enhances behavioral impairments produced by perinatal 6-hydroxydopamine.
    Nowak P, Bojanek K, Szkilnik R, Jośko J, Boroń D, Adwent M, Gorczyca P, Kostrzewa RM, Brus R.
    Neurotox Res; 2011 May; 19(4):536-43. PubMed ID: 20369314
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  • 4. Ontogenetic quinpirole treatments fail to prime for D2 agonist-enhancement of locomotor activity in 6-hydroxydopamine-lesioned rats.
    Brus R, Kostrzewa RM, Nowak P, Perry KW, Kostrzewa JP.
    Neurotox Res; 2003 May; 5(5):329-38. PubMed ID: 14715452
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  • 6. Repeated D1 dopamine receptor agonist administration prevents the development of both D1 and D2 striatal receptor supersensitivity following denervation.
    Hu XT, White FJ.
    Synapse; 1992 Mar; 10(3):206-16. PubMed ID: 1532677
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  • 7. Effect of repeated L-DOPA, bromocriptine, or lisuride administration on preproenkephalin-A and preproenkephalin-B mRNA levels in the striatum of the 6-hydroxydopamine-lesioned rat.
    Henry B, Crossman AR, Brotchie JM.
    Exp Neurol; 1999 Feb; 155(2):204-20. PubMed ID: 10072296
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  • 9. Perinatal manganese exposure and hydroxyl radical formation in rat brain.
    Bałasz M, Szkilnik R, Brus R, Malinowska-Borowska J, Kasperczyk S, Nowak D, Kostrzewa RM, Nowak P.
    Neurotox Res; 2015 Jan; 27(1):1-14. PubMed ID: 25323423
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  • 10. Histaminergic activity in a rodent model of Parkinson's disease.
    Nowak P, Noras L, Jochem J, Szkilnik R, Brus H, Körossy E, Drab J, Kostrzewa RM, Brus R.
    Neurotox Res; 2009 Apr; 15(3):246-51. PubMed ID: 19384597
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  • 12. D1 priming enhances both D1- and D2-mediated rotational behavior and striatal Fos expression in 6-hydroxydopamine lesioned rats.
    Pollack AE, Thomas LI.
    Pharmacol Biochem Behav; 2010 Jan; 94(3):346-51. PubMed ID: 19800912
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  • 13. Evidence for involvement of brain dopamine and other mechanisms in the behavioral action of the N-methyl-D-aspartic acid antagonist MK-801 in control and 6-hydroxydopamine-lesioned rats.
    Criswell HE, Johnson KB, Mueller RA, Breese GR.
    J Pharmacol Exp Ther; 1993 May; 265(2):1001-10. PubMed ID: 8098756
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  • 18. Differential interaction of competitive NMDA and AMPA antagonists with selective dopamine D-1 and D-2 agonists in a rat model of Parkinson's disease.
    Löschmann PA, Wüllner U, Heneka MT, Schulz JB, Kunow M, Wachtel H, Klockgether T.
    Synapse; 1997 Aug; 26(4):381-91. PubMed ID: 9215597
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  • 19. Neonatal 6-OHDA lesions differentially affect striatal D1 and D2 receptors.
    Neal BS, Joyce JN.
    Synapse; 1992 May; 11(1):35-46. PubMed ID: 1351321
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  • 20. Abnormal involuntary movement (AIM) expression following D2 dopamine agonist challenge is determined by the nature of prior dopamine receptor stimulation (priming) in 6-hydroxydopamine lesioned rats.
    Drake JD, Kibuuka LN, Dimitrov KD, Pollack AE.
    Pharmacol Biochem Behav; 2013 Apr; 105():26-33. PubMed ID: 23369985
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