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305 related items for PubMed ID: 24399619

  • 1. Inhibition of the leptin-induced activation of the p38 MAPK pathway contributes to the protective effects of naringin against high glucose-induced injury in H9c2 cardiac cells.
    Chen J, Mo H, Guo R, You Q, Huang R, Wu K.
    Int J Mol Med; 2014 Mar; 33(3):605-12. PubMed ID: 24399619
    [Abstract] [Full Text] [Related]

  • 2. Exogenous hydrogen sulfide protects H9c2 cardiac cells against high glucose-induced injury by inhibiting the activities of the p38 MAPK and ERK1/2 pathways.
    Xu W, Wu W, Chen J, Guo R, Lin J, Liao X, Feng J.
    Int J Mol Med; 2013 Oct; 32(4):917-25. PubMed ID: 23912965
    [Abstract] [Full Text] [Related]

  • 3. Naringin inhibits ROS-activated MAPK pathway in high glucose-induced injuries in H9c2 cardiac cells.
    Chen J, Guo R, Yan H, Tian L, You Q, Li S, Huang R, Wu K.
    Basic Clin Pharmacol Toxicol; 2014 Apr; 114(4):293-304. PubMed ID: 24118820
    [Abstract] [Full Text] [Related]

  • 4. Exogenous H2S protects H9c2 cardiac cells against high glucose-induced injury and inflammation by inhibiting the activation of the NF-κB and IL-1β pathways.
    Xu W, Chen J, Lin J, Liu D, Mo L, Pan W, Feng J, Wu W, Zheng D.
    Int J Mol Med; 2015 Jan; 35(1):177-86. PubMed ID: 25412187
    [Abstract] [Full Text] [Related]

  • 5. Exogenous hydrogen sulfide alleviates high glucose-induced cardiotoxicity via inhibition of leptin signaling in H9c2 cells.
    Zhuang XD, Hu X, Long M, Dong XB, Liu DH, Liao XX.
    Mol Cell Biochem; 2014 Jun; 391(1-2):147-55. PubMed ID: 24687304
    [Abstract] [Full Text] [Related]

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  • 7. The Protective Effect of INH2BP, a Novel PARP Inhibitor 5-Iodo-6-Amino-1,2-Benzopyrone, Against Hydrogen Peroxide-Induced Apoptosis Through ERK and p38 MAPK in H9c2 Cells.
    Back OC, Jang HS, Lee JS, Kim DE, Lee Y, Park ES, Kim IS.
    Pharmacology; 2015 Jun; 96(5-6):259-70. PubMed ID: 26550832
    [Abstract] [Full Text] [Related]

  • 8. Angiotensin-(1-7) protects cardiomyocytes against high glucose-induced injuries through inhibiting reactive oxygen species-activated leptin-p38 mitogen-activated protein kinase/extracellular signal-regulated protein kinase 1/2 pathways, but not the leptin-c-Jun N-terminal kinase pathway in vitro.
    Lei Y, Xu Q, Zeng B, Zhang W, Zhen Y, Zhai Y, Cheng F, Mei W, Zheng D, Feng J, Lan J, Chen J.
    J Diabetes Investig; 2017 Jul; 8(4):434-445. PubMed ID: 27896943
    [Abstract] [Full Text] [Related]

  • 9. Protective effect of angiotensin-(1-7) against hyperglycaemia-induced injury in H9c2 cardiomyoblast cells via the PI3K̸Akt signaling pathway.
    Yang YY, Sun XT, Li ZX, Chen WY, Wang X, Liang ML, Shi H, Yang ZS, Zeng WT.
    Int J Mol Med; 2018 Mar; 41(3):1283-1292. PubMed ID: 29286068
    [Abstract] [Full Text] [Related]

  • 10. Exogenous hydrogen sulfide protects against doxorubicin-induced inflammation and cytotoxicity by inhibiting p38MAPK/NFκB pathway in H9c2 cardiac cells.
    Guo R, Wu K, Chen J, Mo L, Hua X, Zheng D, Chen P, Chen G, Xu W, Feng J.
    Cell Physiol Biochem; 2013 Mar; 32(6):1668-80. PubMed ID: 24356372
    [Abstract] [Full Text] [Related]

  • 11. Naringin inhibits high glucose-induced cardiomyocyte apoptosis by attenuating mitochondrial dysfunction and modulating the activation of the p38 signaling pathway.
    Huang H, Wu K, You Q, Huang R, Li S, Wu K.
    Int J Mol Med; 2013 Aug; 32(2):396-402. PubMed ID: 23732220
    [Abstract] [Full Text] [Related]

  • 12. Naringin attenuates high glucose-induced injuries and inflammation by modulating the leptin-JAK2/STAT3 pathway in H9c2 cardiac cells.
    Luo C, Ke X, Xiong S, Sun Y, Xu Q, Zhang W, Lei Y, Ding Y, Zhen Y, Feng J, Cheng F, Chen J.
    Arch Med Sci; 2021 Aug; 17(5):1145-1157. PubMed ID: 34522243
    [Abstract] [Full Text] [Related]

  • 13. ATP-sensitive K⁺ channels contribute to the protective effects of exogenous hydrogen sulfide against high glucose-induced injury in H9c2 cardiac cells.
    Liang W, Chen J, Mo L, Ke X, Zhang W, Zheng D, Pan W, Wu S, Feng J, Song M, Liao X.
    Int J Mol Med; 2016 Mar; 37(3):763-72. PubMed ID: 26820501
    [Abstract] [Full Text] [Related]

  • 14. Curcumin induces the apoptotic intrinsic pathway via upregulation of reactive oxygen species and JNKs in H9c2 cardiac myoblasts.
    Zikaki K, Aggeli IK, Gaitanaki C, Beis I.
    Apoptosis; 2014 Jun; 19(6):958-74. PubMed ID: 24668280
    [Abstract] [Full Text] [Related]

  • 15. Chlorogenic acid analogues from Gynura nepalensis protect H9c2 cardiomyoblasts against H2O2-induced apoptosis.
    Yu BW, Li JL, Guo BB, Fan HM, Zhao WM, Wang HY.
    Acta Pharmacol Sin; 2016 Nov; 37(11):1413-1422. PubMed ID: 27593219
    [Abstract] [Full Text] [Related]

  • 16. Acetylcholine prevents angiotensin II-induced oxidative stress and apoptosis in H9c2 cells.
    Liu JJ, Li DL, Zhou J, Sun L, Zhao M, Kong SS, Wang YH, Yu XJ, Zhou J, Zang WJ.
    Apoptosis; 2011 Jan; 16(1):94-103. PubMed ID: 20963497
    [Abstract] [Full Text] [Related]

  • 17. Exogenous hydrogen sulfide ameliorates high glucose-induced myocardial injury & inflammation via the CIRP-MAPK signaling pathway in H9c2 cardiac cells.
    Zhao HL, Wu BQ, Luo Y, Zhang WY, Hao YL, Liang JJ, Fang F, Liu W, Chen XH.
    Life Sci; 2018 Sep 01; 208():315-324. PubMed ID: 29857073
    [Abstract] [Full Text] [Related]

  • 18. Activation of the p38 MAPK/NF-κB pathway contributes to doxorubicin-induced inflammation and cytotoxicity in H9c2 cardiac cells.
    Guo RM, Xu WM, Lin JC, Mo LQ, Hua XX, Chen PX, Wu K, Zheng DD, Feng JQ.
    Mol Med Rep; 2013 Aug 01; 8(2):603-8. PubMed ID: 23807148
    [Abstract] [Full Text] [Related]

  • 19. Diallyl trisufide (DATS) suppresses high glucose-induced cardiomyocyte apoptosis by inhibiting JNK/NFκB signaling via attenuating ROS generation.
    Kuo WW, Wang WJ, Tsai CY, Way CL, Hsu HH, Chen LM.
    Int J Cardiol; 2013 Sep 20; 168(1):270-80. PubMed ID: 23158927
    [Abstract] [Full Text] [Related]

  • 20. Taurine prevents arsenic-induced cardiac oxidative stress and apoptotic damage: role of NF-kappa B, p38 and JNK MAPK pathway.
    Ghosh J, Das J, Manna P, Sil PC.
    Toxicol Appl Pharmacol; 2009 Oct 01; 240(1):73-87. PubMed ID: 19616567
    [Abstract] [Full Text] [Related]

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