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Journal Abstract Search


113 related items for PubMed ID: 2450538

  • 1. Pertussis toxin pretreatment abolishes dihydropyridine inhibition of calcium flux in the 235-1 pituitary cell line.
    Schettini G, Meucci O, Florio T, Grimaldi M, Landolfi E, Magri G, Yasumoto T.
    Biochem Biophys Res Commun; 1988 Feb 29; 151(1):361-9. PubMed ID: 2450538
    [Abstract] [Full Text] [Related]

  • 2. Maitotoxin-induced intracellular calcium rise in PC12 cells: involvement of dihydropyridine-sensitive and omega-conotoxin-sensitive calcium channels and phosphoinositide breakdown.
    Meucci O, Grimaldi M, Scorziello A, Govoni S, Bergamaschi S, Yasumoto T, Schettini G.
    J Neurochem; 1992 Aug 29; 59(2):679-88. PubMed ID: 1378490
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  • 4. Dihydropyridine modulation of voltage-activated calcium channels in PC12 cells: effect of pertussis toxin pretreatment.
    Schettini G, Meucci O, Grimaldi M, Florio T, Landolfi E, Scorziello A, Ventra C.
    J Neurochem; 1991 Mar 29; 56(3):805-11. PubMed ID: 1704421
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  • 6. Pertussis toxin (IAP) enhances maitotoxin (a putative Ca2+ channel agonist)-induced Ca2+ entry into synaptosomes.
    Ueda H, Tamura S, Fukushima N, Takagi H.
    Eur J Pharmacol; 1986 Apr 02; 122(3):379-80. PubMed ID: 2423345
    [No Abstract] [Full Text] [Related]

  • 7. Inhibition of parathyroid hormone release by maitotoxin, a calcium channel activator.
    Fitzpatrick LA, Yasumoto T, Aurbach GD.
    Endocrinology; 1989 Jan 02; 124(1):97-103. PubMed ID: 2535815
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  • 8. Role of G-proteins in mediating dihydropyridine receptor coupling with voltage sensitive calcium channels.
    Meucci O, Florio T, Grimaldi M, Landolfi E, Magri G, Schettini G.
    Pharmacol Res Commun; 1988 Dec 02; 20(12):1083-4. PubMed ID: 2850582
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  • 9. Characterization of the MMQ cell, a prolactin-secreting clonal cell line that is responsive to dopamine.
    Judd AM, Login IS, Kovacs K, Ross PC, Spangelo BL, Jarvis WD, MacLeod RM.
    Endocrinology; 1988 Nov 02; 123(5):2341-50. PubMed ID: 2844508
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  • 11. Maitotoxin, a Ca2+ channel activator candidate.
    Takahashi M, Ohizumi Y, Yasumoto T.
    J Biol Chem; 1982 Jul 10; 257(13):7287-89. PubMed ID: 6282837
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  • 12. Involvement of pertussis toxin-sensitive G-proteins in the hormonal inhibition of dihydropyridine-sensitive Ca2+ currents in an insulin-secreting cell line (RINm5F).
    Schmidt A, Hescheler J, Offermanns S, Spicher K, Hinsch KD, Klinz FJ, Codina J, Birnbaumer L, Gausepohl H, Frank R.
    J Biol Chem; 1991 Sep 25; 266(27):18025-33. PubMed ID: 1680855
    [Abstract] [Full Text] [Related]

  • 13. Maitotoxin, a calcium channel activator, increases prolactin release from rat pituitary tumor 7315a cells by a mechanism that may involve leukotriene production.
    Koike K, Judd AM, Login IS, Yasumoto T, MacLeod RM.
    Neuroendocrinology; 1986 Sep 25; 43(3):283-90. PubMed ID: 2426623
    [Abstract] [Full Text] [Related]

  • 14. Maitotoxin activates cation channels distinct from the receptor-activated non-selective cation channels of HL-60 cells.
    Musgrave IF, Seifert R, Schultz G.
    Biochem J; 1994 Jul 15; 301 ( Pt 2)(Pt 2):437-41. PubMed ID: 7519011
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  • 16. Store-operated Ca(2+) inflow in Reuber hepatoma cells is inhibited by voltage-operated Ca(2+) channel antagonists and, in contrast to freshly isolated hepatocytes, does not require a pertussis toxin-sensitive trimeric GTP-binding protein.
    Auld A, Chen J, Brereton HM, Wang YJ, Gregory RB, Barritt GJ.
    Biochim Biophys Acta; 2000 Jun 02; 1497(1):11-26. PubMed ID: 10838155
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  • 17. Ca2+ channel activating function of maitotoxin, the most potent marine toxin known, in clonal rat pheochromocytoma cells.
    Takahashi M, Tatsumi M, Ohizumi Y, Yasumoto T.
    J Biol Chem; 1983 Sep 25; 258(18):10944-9. PubMed ID: 6309824
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  • 18. Inhibition of voltage-dependent Ca2+ currents and activation of pertussis toxin-sensitive G-proteins via muscarinic receptors in GH3 cells.
    Offermanns S, Gollasch M, Hescheler J, Spicher K, Schmidt A, Schultz G, Rosenthal W.
    Mol Endocrinol; 1991 Jul 25; 5(7):995-1002. PubMed ID: 1658634
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  • 20. Reserpine is a calcium channel antagonist in normal and GH3 rat pituitary cells.
    Login IS, Judd AM, Cronin MJ, Yasumoto T, MacLeod RM.
    Am J Physiol; 1985 Jan 25; 248(1 Pt 1):E15-9. PubMed ID: 2578255
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