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113 related items for PubMed ID: 2450538
1. Pertussis toxin pretreatment abolishes dihydropyridine inhibition of calcium flux in the 235-1 pituitary cell line. Schettini G, Meucci O, Florio T, Grimaldi M, Landolfi E, Magri G, Yasumoto T. Biochem Biophys Res Commun; 1988 Feb 29; 151(1):361-9. PubMed ID: 2450538 [Abstract] [Full Text] [Related]
2. Maitotoxin-induced intracellular calcium rise in PC12 cells: involvement of dihydropyridine-sensitive and omega-conotoxin-sensitive calcium channels and phosphoinositide breakdown. Meucci O, Grimaldi M, Scorziello A, Govoni S, Bergamaschi S, Yasumoto T, Schettini G. J Neurochem; 1992 Aug 29; 59(2):679-88. PubMed ID: 1378490 [Abstract] [Full Text] [Related]
4. Dihydropyridine modulation of voltage-activated calcium channels in PC12 cells: effect of pertussis toxin pretreatment. Schettini G, Meucci O, Grimaldi M, Florio T, Landolfi E, Scorziello A, Ventra C. J Neurochem; 1991 Mar 29; 56(3):805-11. PubMed ID: 1704421 [Abstract] [Full Text] [Related]
6. Pertussis toxin (IAP) enhances maitotoxin (a putative Ca2+ channel agonist)-induced Ca2+ entry into synaptosomes. Ueda H, Tamura S, Fukushima N, Takagi H. Eur J Pharmacol; 1986 Apr 02; 122(3):379-80. PubMed ID: 2423345 [No Abstract] [Full Text] [Related]
7. Inhibition of parathyroid hormone release by maitotoxin, a calcium channel activator. Fitzpatrick LA, Yasumoto T, Aurbach GD. Endocrinology; 1989 Jan 02; 124(1):97-103. PubMed ID: 2535815 [Abstract] [Full Text] [Related]
8. Role of G-proteins in mediating dihydropyridine receptor coupling with voltage sensitive calcium channels. Meucci O, Florio T, Grimaldi M, Landolfi E, Magri G, Schettini G. Pharmacol Res Commun; 1988 Dec 02; 20(12):1083-4. PubMed ID: 2850582 [No Abstract] [Full Text] [Related]
9. Characterization of the MMQ cell, a prolactin-secreting clonal cell line that is responsive to dopamine. Judd AM, Login IS, Kovacs K, Ross PC, Spangelo BL, Jarvis WD, MacLeod RM. Endocrinology; 1988 Nov 02; 123(5):2341-50. PubMed ID: 2844508 [Abstract] [Full Text] [Related]
16. Store-operated Ca(2+) inflow in Reuber hepatoma cells is inhibited by voltage-operated Ca(2+) channel antagonists and, in contrast to freshly isolated hepatocytes, does not require a pertussis toxin-sensitive trimeric GTP-binding protein. Auld A, Chen J, Brereton HM, Wang YJ, Gregory RB, Barritt GJ. Biochim Biophys Acta; 2000 Jun 02; 1497(1):11-26. PubMed ID: 10838155 [Abstract] [Full Text] [Related]
17. Ca2+ channel activating function of maitotoxin, the most potent marine toxin known, in clonal rat pheochromocytoma cells. Takahashi M, Tatsumi M, Ohizumi Y, Yasumoto T. J Biol Chem; 1983 Sep 25; 258(18):10944-9. PubMed ID: 6309824 [Abstract] [Full Text] [Related]
18. Inhibition of voltage-dependent Ca2+ currents and activation of pertussis toxin-sensitive G-proteins via muscarinic receptors in GH3 cells. Offermanns S, Gollasch M, Hescheler J, Spicher K, Schmidt A, Schultz G, Rosenthal W. Mol Endocrinol; 1991 Jul 25; 5(7):995-1002. PubMed ID: 1658634 [Abstract] [Full Text] [Related]
20. Reserpine is a calcium channel antagonist in normal and GH3 rat pituitary cells. Login IS, Judd AM, Cronin MJ, Yasumoto T, MacLeod RM. Am J Physiol; 1985 Jan 25; 248(1 Pt 1):E15-9. PubMed ID: 2578255 [Abstract] [Full Text] [Related] Page: [Next] [New Search]